Preclinical Development of [211At]meta- astatobenzylguanidine ([211At]MABG) as an Alpha Particle Radiopharmaceutical Therapy for Neuroblastoma.


Journal

Clinical cancer research : an official journal of the American Association for Cancer Research
ISSN: 1557-3265
Titre abrégé: Clin Cancer Res
Pays: United States
ID NLM: 9502500

Informations de publication

Date de publication:
15 09 2022
Historique:
received: 09 03 2022
revised: 09 06 2022
accepted: 19 07 2022
pubmed: 22 7 2022
medline: 17 9 2022
entrez: 21 7 2022
Statut: ppublish

Résumé

[131I]meta-iodobenzylguanidine ([131I]MIBG) is a targeted radiotherapeutic administered systemically to deliver beta particle radiation in neuroblastoma. However, relapses in the bone marrow are common. [211At]meta-astatobenzylguanidine ([211At] MABG) is an alpha particle emitter with higher biological effectiveness and short path length which effectively sterilizes microscopic residual disease. Here we investigated the safety and antitumor activity [211At]MABG in preclinical models of neuroblastoma. We defined the maximum tolerated dose (MTD), biodistribution, and toxicity of [211At]MABG in immunodeficient mice in comparison with [131I]MIBG. We compared the antitumor efficacy of [211At]MABG with [131I]MIBG in three murine xenograft models. Finally, we explored the efficacy of [211At]MABG after tail vein xenografting designed to model disseminated neuroblastoma. The MTD of [211At]MABG was 66.7 MBq/kg (1.8 mCi/kg) in CB17SC scid-/- mice and 51.8 MBq/kg (1.4 mCi/kg) in NOD.Cg-Prkdcscid Il2rgtm1Wjl/SzJ (NSG) mice. Biodistribution of [211At]MABG was similar to [131I]MIBG. Long-term toxicity studies on mice administered with doses up to 41.5 MBq/kg (1.12 mCi/kg) showed the radiotherapeutic to be well tolerated. Both 66.7 MBq/kg (1.8 mCi/kg) single dose and fractionated dosing 16.6 MBq/kg/fraction (0.45 mCi/kg) × 4 over 11 days induced marked tumor regression in two of the three models studied. Survival was significantly prolonged for mice treated with 12.9 MBq/kg/fraction (0.35 mCi/kg) × 4 doses over 11 days [211At]MABG in the disseminated disease (IMR-05NET/GFP/LUC) model (P = 0.003) suggesting eradication of microscopic disease. [211At]MABG has significant survival advantage in disseminated models of neuroblastoma. An alpha particle emitting radiopharmaceutical may be effective against microscopic disseminated disease, warranting clinical development.

Identifiants

pubmed: 35861867
pii: 707126
doi: 10.1158/1078-0432.CCR-22-0400
pmc: PMC9475242
mid: NIHMS1826704
doi:

Substances chimiques

Astatine-211 0
Guanidines 0
Iodine Radioisotopes 0
Iodine-131 0
Radiopharmaceuticals 0
3-Iodobenzylguanidine 35MRW7B4AD
Astatine XI595HAL7H

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

4146-4157

Subventions

Organisme : NCI NIH HHS
ID : R01 CA219006
Pays : United States
Organisme : NCI NIH HHS
ID : R35 CA220500
Pays : United States

Informations de copyright

©2022 The Authors; Published by the American Association for Cancer Research.

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Auteurs

Vandana Batra (V)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.
Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania.

Minu Samanta (M)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Mehran Makvandi (M)

Department of Radiology, University of Pennsylvania, Philadelphia, Pennsylvania.

David Groff (D)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Paul Martorano (P)

Department of Radiology, University of Pennsylvania, Philadelphia, Pennsylvania.

Jimmy Elias (J)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Pietro Ranieri (P)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Matthew Tsang (M)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Catherine Hou (C)

Department of Radiology, University of Pennsylvania, Philadelphia, Pennsylvania.

Yimei Li (Y)

Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania.
Department of Biostatistics, Epidemiology, and Informatics, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania.

Bruce Pawel (B)

Department of Pathology and Laboratory Medicine, Children's Hospital Los Angeles and Keck School of Medicine, University of Southern California, Los Angeles, California.

Daniel Martinez (D)

Division of Anatomic Pathology, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

Ganesan Vaidyanathan (G)

Department of Radiology, Duke University Medical Center, Durham, North Carolina.

Sean Carlin (S)

Department of Radiology, University of Pennsylvania, Philadelphia, Pennsylvania.

Daniel A Pryma (DA)

Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania.
Department of Radiology, University of Pennsylvania, Philadelphia, Pennsylvania.

John M Maris (JM)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.
Perelman School of Medicine at the University of Pennsylvania, Philadelphia, Pennsylvania.

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Classifications MeSH