Lateral habenula glutamatergic neurons projecting to the dorsal raphe nucleus promote aggressive arousal in mice.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
21 07 2022
Historique:
received: 14 05 2021
accepted: 29 06 2022
entrez: 21 7 2022
pubmed: 22 7 2022
medline: 26 7 2022
Statut: epublish

Résumé

The dorsal raphe nucleus (DRN) is known to control aggressive behavior in mice. Here, we found that glutamatergic projections from the lateral habenula (LHb) to the DRN were activated in male mice that experienced pre-exposure to a rival male mouse ("social instigation") resulting in heightened intermale aggression. Both chemogenetic and optogenetic suppression of the LHb-DRN projection blocked heightened aggression after social instigation in male mice. In contrast, inhibition of this pathway did not affect basal levels of aggressive behavior, suggesting that the activity of the LHb-DRN projection is not necessary for the expression of species-typical aggressive behavior, but required for the increase of aggressive behavior resulting from social instigation. Anatomical analysis showed that LHb neurons synapse on non-serotonergic DRN neurons that project to the ventral tegmental area (VTA), and optogenetic activation of the DRN-VTA projection increased aggressive behaviors. Our results demonstrate that the LHb glutamatergic inputs to the DRN promote aggressive arousal induced by social instigation, which contributes to aggressive behavior by activating VTA-projecting non-serotonergic DRN neurons as one of its potential targets.

Identifiants

pubmed: 35864121
doi: 10.1038/s41467-022-31728-z
pii: 10.1038/s41467-022-31728-z
pmc: PMC9304121
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

4039

Subventions

Organisme : NIMH NIH HHS
ID : R01 MH120514
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH120637
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH104559
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH127820
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH114882
Pays : United States

Informations de copyright

© 2022. The Author(s).

Références

Archer, J. & Huntingford, F. Game theory models and escalation of animal fights. in The Dynamics of Aggression: Biological and Social Processes in Dyads and Groups (eds. Potegal, M. & Kuntson, J. F.) 3–33 (Lawrence Erlbaum Associates, 1994).
Heiligenberg, W. The effect of external stimuli on the attack readiness of a cichlid fish. Z. Vgl. Physiol. 49, 459–464 (1965).
doi: 10.1007/BF00298113
Potegal, M. Time course of aggressive arousal in female hamsters and male rats. Behav. Neural Biol. 58, 120–124 (1992).
pubmed: 1456930 doi: 10.1016/0163-1047(92)90339-6
Fish, E. W., Faccidomo, S. & Miczek, K. A. Aggression heightened by alcohol or social instigation in mice: reduction by the 5-HT(1B) receptor agonist CP-94,253. Psychopharmacology 146, 391–399 (1999).
pubmed: 10550489 doi: 10.1007/PL00005484
Potegal, M. Aggressive arousal: the amygdala connection. in The Dynamics of Aggression: Biological and Social Processes in Dyads and Groups (eds. Potegal, M. & Knutson, J. F.) 73–132 (Lawrence Erlbaum Associates, 1994).
Miczek, K. A., de Boer, S. F. & Haller, J. Excessive aggression as model of violence: a critical evaluation of current preclinical methods. Psychopharmacology 226, 445–458 (2013).
pubmed: 23430160 pmcid: 3595336 doi: 10.1007/s00213-013-3008-x
Lagerspetz, K. & Hautojärvi, S. The effect of prior aggressive or sexual arousal on subsequent aggressive or sexual reactions in male mice. Scand. J. Psychol. 8, 1–6 (1967).
pubmed: 6068329 doi: 10.1111/j.1467-9450.1967.tb01365.x
Nordman, J. C. et al. Potentiation of divergent medial amygdala pathways drives experience-dependent aggression escalation. J. Neurosci. 40, 4858–4880 (2020).
pubmed: 32424020 pmcid: 7326350 doi: 10.1523/JNEUROSCI.0370-20.2020
de Boer, S. F. & Koolhaas, J. M. 5-HT1A and 5-HT1B receptor agonists and aggression: a pharmacological challenge of the serotonin deficiency hypothesis. Eur. J. Pharmacol. 526, 125–139 (2005).
pubmed: 16310183 doi: 10.1016/j.ejphar.2005.09.065
Miczek, K. A. et al. Escalated aggressive behavior: new pharmacotherapeutic approaches and opportunities. Ann. N. Y. Acad. Sci. 1036, 336–355 (2004).
pubmed: 15817748 doi: 10.1196/annals.1330.021
Olivier, B. Serotonin and aggression. Ann. N. Y. Acad. Sci. 1036, 382–392 (2004).
pubmed: 15817750 doi: 10.1196/annals.1330.022
Berman, M., Tracy, J. & Coccaro, E. F. The serotonin hypothesis of aggression revisited. Clin. Psychol. Rev. 17, 651–665 (1997).
pubmed: 9336689 doi: 10.1016/S0272-7358(97)00039-1
Takahashi, A. & Miczek, K. A. Neurogenetics of aggressive behavior: studies in rodents. Curr. Top. Behav. Neurosci. 17, 3–44 (2014).
pubmed: 24318936 pmcid: 4092042 doi: 10.1007/7854_2013_263
Takahashi, A. et al. Glutamate input in the dorsal raphe nucleus as a determinant of escalated aggression in male mice. J. Neurosci. 35, 6452–6463 (2015).
pubmed: 25904796 pmcid: 6605224 doi: 10.1523/JNEUROSCI.2450-14.2015
Muroi, Y. & Ishii, T. Glutamatergic signals in the dorsal raphe nucleus regulate maternal aggression and care in an opposing manner in mice. Neuroscience 400, 33–47 (2019).
pubmed: 30605702 doi: 10.1016/j.neuroscience.2018.12.034
Pollak Dorocic, I. et al. A whole-brain atlas of inputs to serotonergic neurons of the dorsal and median raphe nuclei. Neuron 83, 663–678 (2014).
pubmed: 25102561 doi: 10.1016/j.neuron.2014.07.002
Ogawa, S. K., Cohen, J. Y., Hwang, D., Uchida, N. & Watabe-Uchida, M. Organization of monosynaptic inputs to the serotonin and dopamine neuromodulatory systems. Cell Rep. 8, 1105–1118 (2014).
pubmed: 25108805 pmcid: 4142108 doi: 10.1016/j.celrep.2014.06.042
Weissbourd, B. et al. Presynaptic partners of dorsal raphe serotonergic and GABAergic neurons. Neuron 83, 645–662 (2014).
pubmed: 25102560 pmcid: 4779447 doi: 10.1016/j.neuron.2014.06.024
Zhou, L. et al. Organization of functional long-range circuits controlling the activity of serotonergic neurons in the dorsal raphe nucleus. Cell Rep. 20, 1991–1993 (2017).
pubmed: 28834759 doi: 10.1016/j.celrep.2017.08.032
Hale, M. W. & Lowry, C. A. Functional topography of midbrain and pontine serotonergic systems: implications for synaptic regulation of serotonergic circuits. Psychopharmacology 213, 243–264 (2011).
pubmed: 21088958 doi: 10.1007/s00213-010-2089-z
Tanaka, K. F. et al. Expanding the repertoire of optogenetically targeted cells with an enhanced gene expression system. Cell Rep. 2, 397–406 (2012).
pubmed: 22854021 doi: 10.1016/j.celrep.2012.06.011
Zingg, B. et al. AAV-mediated anterograde transsynaptic tagging: mapping corticocollicular input-defined neural pathways for defense behaviors. Neuron 93, 33–47 (2017).
pubmed: 27989459 doi: 10.1016/j.neuron.2016.11.045
de Almeida, R. M. M., Ferrari, P. F., Parmigiani, S. & Miczek, K. A. Escalated aggressive behavior: dopamine, serotonin and GABA. Eur. J. Pharmacol. 526, 51–64 (2005).
pubmed: 16325649 doi: 10.1016/j.ejphar.2005.10.004
Golden, S. A., Jin, M. & Shaham, Y. Animal models of (or for) aggression reward, addiction, and relapse: behavior and circuits. J. Neurosci. 39, 3996–4008 (2019).
pubmed: 30833504 pmcid: 6529864 doi: 10.1523/JNEUROSCI.0151-19.2019
Yu, Q. et al. Dopamine and serotonin signaling during two sensitive developmental periods differentially impact adult aggressive and affective behaviors in mice. Mol. Psychiatry 19, 688–698 (2014).
pubmed: 24589889 pmcid: 4311886 doi: 10.1038/mp.2014.10
Proulx, C. D., Hikosaka, O. & Malinow, R. Reward processing by the lateral habenula in normal and depressive behaviors. Nat. Neurosci. 17, 1146–1152 (2014).
pubmed: 25157511 pmcid: 4305435 doi: 10.1038/nn.3779
Chou, M. Y. et al. Social conflict resolution regulated by two dorsal habenular subregions in zebrafish. Science 352, 87–90 (2016).
pubmed: 27034372 doi: 10.1126/science.aac9508
Golden, S. A. et al. Basal forebrain projections to the lateral habenula modulate aggression reward. Nature 534, 688–692 (2016).
pubmed: 27357796 pmcid: 4930107 doi: 10.1038/nature18601
Flanigan, M. E. et al. Orexin signaling in GABAergic lateral habenula neurons modulates aggressive behavior in male mice. Nat. Neurosci. 23, 638–650 (2020).
pubmed: 32284606 pmcid: 7195257 doi: 10.1038/s41593-020-0617-7
Gan, G. et al. Habenula-prefrontal resting-state connectivity in reactive aggressive men – a pilot study. Neuropharmacology 156, 107396 (2019).
pubmed: 30366001 doi: 10.1016/j.neuropharm.2018.10.025
Cerniauskas, I. et al. Chronic stress induces activity, synaptic, and transcriptional remodeling of the lateral habenula associated with deficits in motivated behaviors. Neuron 104, 899–915.e8 (2019).
pubmed: 31672263 pmcid: 6895430 doi: 10.1016/j.neuron.2019.09.005
Levinstein, M. R., Coffey, K. R., Marx, R. G., Lesiak, A. J. & Neumaier, J. F. Stress induces divergent gene expression among lateral habenula efferent pathways. Neurobiol. Stress 13, 100268 (2020).
pubmed: 33344721 pmcid: 7739173 doi: 10.1016/j.ynstr.2020.100268
Qi, J. et al. A glutamatergic reward input from the dorsal raphe to ventral tegmental area dopamine neurons. Nat. Commun. 5, 5390 (2014).
pubmed: 25388237 doi: 10.1038/ncomms6390
McDevitt, R. A. et al. Serotonergic versus nonserotonergic dorsal raphe projection neurons: differential participation in reward circuitry. Cell Rep. 8, 1857–1869 (2014).
pubmed: 25242321 pmcid: 4181379 doi: 10.1016/j.celrep.2014.08.037
Nordman, J. & Li, Z. The dorsal raphe regulates the duration of attack through the medial orbitofrontal cortex and medial amygdala. eNeuro 7, ENEURO.0331-20.2020 (2020).
pubmed: 33055195 pmcid: 7665904 doi: 10.1523/ENEURO.0331-20.2020
Balázsfi, D. et al. Differential roles of the two raphe nuclei in amiable social behavior and aggression – An optogenetic study. Front. Behav. Neurosci. 12, 163 (2018).
pubmed: 30116182 pmcid: 6082963 doi: 10.3389/fnbeh.2018.00163
Takahashi, A. et al. Neuromodulatory effect of interleukin 1β in the dorsal raphe nucleus on individual differences in aggression. Mol. Psychiatry 27, 2563–2579 (2021).
Denenberg, V. H., Gaulin-Kremer, E., Gandelman, R. & Zarrow, M. X. The development of standard stimulus animals for mouse (Mus musculus) aggression testing by means of olfactory bulbectomy. Anim. Behav. 21, 590–598 (1973).
pubmed: 4795937 doi: 10.1016/S0003-3472(73)80021-1
Grant, E. C. & Mackintosh, J. H. A comparison of the social postures of some common laboratory rodents. Behaviour 21, 246–259 (1963).
doi: 10.1163/156853963X00185
Miczek, K. A. & O’Donnell, J. M. Intruder-evoked aggression in isolated and nonisolated mice: effects of psychomotor stimulants and L-dopa. Psychopharmacology 57, 47–55 (1978).
pubmed: 26933 doi: 10.1007/BF00426957
Franklin, K. & Paxinos, G. The Mouse Brain in Stereotaxic Coordinates, Ed 3 (Academic, San Diego, 2008).
Atasoy, D., Betley, J. N., Su, H. H. & Sternson, S. M. Deconstruction of a neural circuit for hunger. Nature 488, 172–177 (2012).
pubmed: 22801496 pmcid: 3416931 doi: 10.1038/nature11270
Mahn, M., Prigge, M., Ron, S., Levy, R. & Yizhar, O. Biophysical constraints of optogenetic inhibition at presynaptic terminals. Nat. Neurosci. 19, 554–556 (2016).
pubmed: 26950004 pmcid: 4926958 doi: 10.1038/nn.4266
Chow, B. Y. et al. High-performance genetically targetable optical neural silencing by light-driven proton pumps. Nature 463, 98–102 (2010).
pubmed: 20054397 pmcid: 2939492 doi: 10.1038/nature08652
Tsunematsu, T. et al. Long-lasting silencing of orexin/hypocretin neurons using archaerhodopsin induces slow-wave sleep in mice. Behav. Brain Res. 255, 64–74 (2013).
pubmed: 23707248 doi: 10.1016/j.bbr.2013.05.021
Bellavance, M. A. et al. Parallel inhibitory and excitatory trigemino-facial feedback circuitry for reflexive vibrissa movement. Neuron 95, 673–682.e4 (2017).
pubmed: 28735746 pmcid: 5845798 doi: 10.1016/j.neuron.2017.06.045
Takahashi, A., Shimamoto, A., Boyson, C. O., DeBold, J. F. & Miczek, K. A. GABA(B) receptor modulation of serotonin neurons in the dorsal raphé nucleus and escalation of aggression in mice. J. Neurosci. 30, 11771–11780 (2010).
pubmed: 20810897 pmcid: 2943331 doi: 10.1523/JNEUROSCI.1814-10.2010

Auteurs

Aki Takahashi (A)

Laboratory of Behavioral Neurobiology, Faculty of Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8577, Japan. aktakaha@human.tsukuba.ac.jp.
Laboratory of Behavioral Neuroendocrinology, Faculty of Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8577, Japan. aktakaha@human.tsukuba.ac.jp.
Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA. aktakaha@human.tsukuba.ac.jp.

Romain Durand-de Cuttoli (R)

Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

Meghan E Flanigan (ME)

Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.
Bowles Center for Alcohol Studies, University of North Carolina School of Medicine, Chapel Hill, 27599, NC, USA.

Emi Hasegawa (E)

Department of Molecular Behavioral Physiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.
International Institute for Integrative Sleep Medicine (WPI-IIIS), University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Tomomi Tsunematsu (T)

Super-network Brain Physiology, Graduate School of Life Sciences, Tohoku University, Sendai, Miyagi, 980-8577, Japan.
Advanced Interdisciplinary Research Division, Frontier Research Institute for Interdisciplinary Sciences, Tohoku University, Sendai, Miyagi, 980-8578, Japan.
Precursory Research for Embryonic Science and Technology, Japan Science and Technology Agency, Kawaguchi, Saitama, 332-0012, Japan.

Hossein Aleyasin (H)

Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

Yoan Cherasse (Y)

International Institute for Integrative Sleep Medicine (WPI-IIIS), University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Ken Miya (K)

Department of Molecular Neurobiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.
Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Takuya Okada (T)

Department of Molecular Neurobiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Kazuko Keino-Masu (K)

Department of Molecular Neurobiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Koshiro Mitsui (K)

Laboratory of Behavioral Neurobiology, Faculty of Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8577, Japan.
Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Long Li (L)

Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

Vishwendra Patel (V)

Department of Pharmacological Sciences and Department of Psychiatry, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

Robert D Blitzer (RD)

Department of Pharmacological Sciences and Department of Psychiatry, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

Michael Lazarus (M)

International Institute for Integrative Sleep Medicine (WPI-IIIS), University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Kenji F Tanaka (KF)

Department of Neuropsychiatry, Keio University School of Medicine, Shinjuku, Tokyo, 160-8582, Japan.

Akihiro Yamanaka (A)

Department of Neuroscience II, Research Institute of Environmental Medicine, Nagoya University, Nagoya, Aichi, 464-8601, Japan.

Takeshi Sakurai (T)

Department of Molecular Behavioral Physiology, Faculty of Medicine, University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.
International Institute for Integrative Sleep Medicine (WPI-IIIS), University of Tsukuba, Tsukuba, Ibaraki, 305-8575, Japan.

Sonoko Ogawa (S)

Laboratory of Behavioral Neuroendocrinology, Faculty of Human Sciences, University of Tsukuba, Tsukuba, Ibaraki, 305-8577, Japan.

Scott J Russo (SJ)

Nash Family Department of Neuroscience and Brain & Body Research Center, Icahn School of Medicine at Mount Sinai, New York, NY, 10029, USA.

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