Association of Arterial Spin Labeling Parameters With Cognitive Decline, Vascular Events, and Mortality in a Memory-Clinic Sample.

Cerebrovascular circulation aged brain cerebral blood flow coefficient of variation memory-clinic perfusion spin labels

Journal

The American journal of geriatric psychiatry : official journal of the American Association for Geriatric Psychiatry
ISSN: 1545-7214
Titre abrégé: Am J Geriatr Psychiatry
Pays: England
ID NLM: 9309609

Informations de publication

Date de publication:
12 2022
Historique:
received: 27 10 2021
revised: 25 05 2022
accepted: 12 06 2022
pubmed: 24 7 2022
medline: 8 11 2022
entrez: 23 7 2022
Statut: ppublish

Résumé

Cognitive decline in older adults has been attributed to reduced cerebral blood flow (CBF). Recently, the spatial coefficient of variation (sCoV) of ASL has been proposed as a proxy marker of cerebrovascular insufficiency. We investigated the association between baseline ASL parameters with cognitive decline, incident cerebrovascular disease, and risk of vascular events and mortality. About 368 memory-clinic patients underwent three-annual neuropsychological assessments and brain MRI scans at baseline and follow-up. MRIs were graded for white matter hyperintensities (WMH), lacunes, cerebral microbleeds (CMBs), cortical infarcts, and intracranial stenosis. Baseline gray (GM) and white matter (WM) CBF and GM-sCoV were obtained with ExploreASL from 2D-EPI pseudo-continuous ASL images. Cognitive assessment was done using a validated neuropsychological battery. Data on incident vascular events (heart disease, stroke, transient ischemic attack) and mortality were obtained. Higher baseline GM-sCoV was associated with decline in the memory domain over 3 years of follow-up. Furthermore, higher GM-sCoV was associated with a decline in the memory domain only in participants without dementia. Higher baseline GM-sCoV was associated with progression of WMH and incident CMBs. During a mean follow-up of 3 years, 29 (7.8%) participants developed vascular events and 18 (4.8%) died. Participants with higher baseline mean GM-sCoV were at increased risk of vascular events. Higher baseline GM-sCoV of ASL was associated with a decline in memory and risk of cerebrovascular disease and vascular events, suggesting that cerebrovascular insufficiency may contribute to accelerated cognitive decline and worse clinical outcomes in memory clinic participants.

Sections du résumé

BACKGROUND
Cognitive decline in older adults has been attributed to reduced cerebral blood flow (CBF). Recently, the spatial coefficient of variation (sCoV) of ASL has been proposed as a proxy marker of cerebrovascular insufficiency. We investigated the association between baseline ASL parameters with cognitive decline, incident cerebrovascular disease, and risk of vascular events and mortality.
DESIGN, SETTING, AND PARTICIPANTS
About 368 memory-clinic patients underwent three-annual neuropsychological assessments and brain MRI scans at baseline and follow-up. MRIs were graded for white matter hyperintensities (WMH), lacunes, cerebral microbleeds (CMBs), cortical infarcts, and intracranial stenosis. Baseline gray (GM) and white matter (WM) CBF and GM-sCoV were obtained with ExploreASL from 2D-EPI pseudo-continuous ASL images. Cognitive assessment was done using a validated neuropsychological battery. Data on incident vascular events (heart disease, stroke, transient ischemic attack) and mortality were obtained.
RESULTS
Higher baseline GM-sCoV was associated with decline in the memory domain over 3 years of follow-up. Furthermore, higher GM-sCoV was associated with a decline in the memory domain only in participants without dementia. Higher baseline GM-sCoV was associated with progression of WMH and incident CMBs. During a mean follow-up of 3 years, 29 (7.8%) participants developed vascular events and 18 (4.8%) died. Participants with higher baseline mean GM-sCoV were at increased risk of vascular events.
CONCLUSIONS
Higher baseline GM-sCoV of ASL was associated with a decline in memory and risk of cerebrovascular disease and vascular events, suggesting that cerebrovascular insufficiency may contribute to accelerated cognitive decline and worse clinical outcomes in memory clinic participants.

Identifiants

pubmed: 35871110
pii: S1064-7481(22)00443-2
doi: 10.1016/j.jagp.2022.06.007
pii:
doi:

Substances chimiques

Spin Labels 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1298-1309

Informations de copyright

Copyright © 2022 American Association for Geriatric Psychiatry. Published by Elsevier Inc. All rights reserved.

Auteurs

Bibek Gyanwali (B)

Memory Aging & Cognition Centre, National University Health System (BG, ORK, CC, SH), Singapore.

Henk Jmm Mutsaerts (HJ)

Department of Radiology and Nuclear Medicine, Amsterdam University Medical Center, Amsterdam Neuroscience (HJMMM), Amsterdam, the Netherlands.

Chuen Seng Tan (CS)

Saw Swee Hock School of Public Health, National University of Singapore and National University Health System (CST, SH), Singapore.

Omar Rajab Kaweilh (OR)

Memory Aging & Cognition Centre, National University Health System (BG, ORK, CC, SH), Singapore.

Jan Petr (J)

Helmholtz-Zentrum Dresden-Rossendorf, Institute of Radiopharmaceutical Cancer Research (JP), Dresden, Germany.

Christopher Chen (C)

Memory Aging & Cognition Centre, National University Health System (BG, ORK, CC, SH), Singapore; Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore (CC, SH), Singapore.

Saima Hilal (S)

Memory Aging & Cognition Centre, National University Health System (BG, ORK, CC, SH), Singapore; Saw Swee Hock School of Public Health, National University of Singapore and National University Health System (CST, SH), Singapore; Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore (CC, SH), Singapore. Electronic address: saimahilal@nus.edu.sg.

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