RANTES and CD40L under Conditions of Long-Term Physical Exercise: A Potential Link to Adaptive Immunity.


Journal

International journal of environmental research and public health
ISSN: 1660-4601
Titre abrégé: Int J Environ Res Public Health
Pays: Switzerland
ID NLM: 101238455

Informations de publication

Date de publication:
16 07 2022
Historique:
received: 17 05 2022
revised: 01 07 2022
accepted: 14 07 2022
entrez: 27 7 2022
pubmed: 28 7 2022
medline: 29 7 2022
Statut: epublish

Résumé

Regular physical exercise was found to be associated with an improved immune response in previous studies. RANTES and CD40L play a pivotal role in host defense, and individuals lacking adequate expression are prone to virus and opportunistic infections. A total of 98 participants were enrolled in this study. The probands were asked to perform moderate physical activity, and bicycle stress tests were performed at the baseline and after 8 months of training to evaluate individual performance. RANTES and CD40L were found to be increased by long-term physical exercise. In particular, probands with a performance gain of ≥3% displayed a pronounced elevation of both markers, paired with a decrease in circulating IL6 levels and an improved lipid profile. In summary, we were able to highlight rising levels of serum RANTES and CD40L under the conditions of physical exercise. Taking their role in host defense into account, a conjunction of physical activity and the adaptive immune system could therefore be assumed. Furthermore, low inflammatory profiles in probands with a significant performance gain suggest a modulation through exercise rather than a generalized pro-inflammatory status.

Identifiants

pubmed: 35886510
pii: ijerph19148658
doi: 10.3390/ijerph19148658
pmc: PMC9316936
pii:
doi:

Substances chimiques

Biomarkers 0
Chemokine CCL5 0
CD40 Ligand 147205-72-9

Banques de données

ClinicalTrials.gov
['NCT02097199']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

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Auteurs

Max Lenz (M)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.
Ludwig Boltzmann Cluster for Cardiovascular Research, 1090 Vienna, Austria.

Robert Schönbauer (R)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

Stefan Stojkovic (S)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

Jonghui Lee (J)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

Constantin Gatterer (C)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

Michael Lichtenauer (M)

Clinic of Internal Medicine II, Department of Cardiology, Paracelsus Medical University of Salzburg, 5020 Salzburg, Austria.

Vera Paar (V)

Clinic of Internal Medicine II, Department of Cardiology, Paracelsus Medical University of Salzburg, 5020 Salzburg, Austria.

Michael Emich (M)

Austrian Federal Ministry of Defence, Austrian Armed Forces, 1090 Vienna, Austria.

Monika Fritzer-Szekeres (M)

Chemical Laboratory Analysis, Department of Medical, Medical University of Vienna, 1090 Vienna, Austria.

Jeanette Strametz-Juranek (J)

Rehabilitation Centre Bad Tatzmannsdorf, 7431 Bad Tatzmannsdorf, Austria.

Senta Graf (S)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

Michael Sponder (M)

Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, 1090 Vienna, Austria.

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Classifications MeSH