Malaria Transmission Intensity Likely Modifies RTS, S/AS01 Efficacy Due to a Rebound Effect in Ghana, Malawi, and Gabon.


Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
01 11 2022
Historique:
received: 20 05 2022
accepted: 26 07 2022
pubmed: 29 7 2022
medline: 4 11 2022
entrez: 28 7 2022
Statut: ppublish

Résumé

RTS,S/AS01 is the first malaria vaccine to be approved and recommended for widespread implementation by the World Health Organization (WHO). Trials reported lower vaccine efficacies in higher-incidence sites, potentially due to a "rebound" in malaria cases in vaccinated children. When naturally acquired protection in the control group rises and vaccine protection in the vaccinated wanes concurrently, malaria incidence can become greater in the vaccinated than in the control group, resulting in negative vaccine efficacies. Using data from the 2009-2014 phase III trial (NCT00866619) in Lilongwe, Malawi; Kintampo, Ghana; and Lambaréné, Gabon, we evaluate this hypothesis by estimating malaria incidence in each vaccine group over time and in varying transmission settings. After estimating transmission intensities using ecological variables, we fit models with 3-way interactions between vaccination, time, and transmission intensity. Over time, incidence decreased in the control group and increased in the vaccine group. Three-dose efficacy in the lowest-transmission-intensity group (0.25 cases per person-year [CPPY]) decreased from 88.2% to 15.0% over 4.5 years, compared with 81.6% to -27.7% in the highest-transmission-intensity group (3 CPPY). These findings suggest that interventions, including the fourth RTS,S dose, that protect vaccinated individuals during the potential rebound period should be implemented for high-transmission settings.

Sections du résumé

BACKGROUND
RTS,S/AS01 is the first malaria vaccine to be approved and recommended for widespread implementation by the World Health Organization (WHO). Trials reported lower vaccine efficacies in higher-incidence sites, potentially due to a "rebound" in malaria cases in vaccinated children. When naturally acquired protection in the control group rises and vaccine protection in the vaccinated wanes concurrently, malaria incidence can become greater in the vaccinated than in the control group, resulting in negative vaccine efficacies.
METHODS
Using data from the 2009-2014 phase III trial (NCT00866619) in Lilongwe, Malawi; Kintampo, Ghana; and Lambaréné, Gabon, we evaluate this hypothesis by estimating malaria incidence in each vaccine group over time and in varying transmission settings. After estimating transmission intensities using ecological variables, we fit models with 3-way interactions between vaccination, time, and transmission intensity.
RESULTS
Over time, incidence decreased in the control group and increased in the vaccine group. Three-dose efficacy in the lowest-transmission-intensity group (0.25 cases per person-year [CPPY]) decreased from 88.2% to 15.0% over 4.5 years, compared with 81.6% to -27.7% in the highest-transmission-intensity group (3 CPPY).
CONCLUSIONS
These findings suggest that interventions, including the fourth RTS,S dose, that protect vaccinated individuals during the potential rebound period should be implemented for high-transmission settings.

Identifiants

pubmed: 35899811
pii: 6650987
doi: 10.1093/infdis/jiac322
pmc: PMC10205900
doi:

Substances chimiques

Malaria Vaccines 0

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1646-1656

Subventions

Organisme : NIAID NIH HHS
ID : K24 AI134990
Pays : United States
Organisme : NIAID NIH HHS
ID : R01 AI137410
Pays : United States
Organisme : NIH HHS
ID : K24AI134990)
Pays : United States

Informations de copyright

© The Author(s) 2022. Published by Oxford University Press on behalf of Infectious Diseases Society of America. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Déclaration de conflit d'intérêts

Potential conflicts of interest. All authors: No reported conflicts. All authors have submitted the ICMJE Form for Disclosure of Potential Conflicts of Interest. Conflicts that the editors consider relevant to the content of the manuscript have been disclosed.

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Auteurs

Griffin J Bell (GJ)

Department of Epidemiology, Gillings School of Global Public Health, University of North Carolina, Chapel Hill, North Carolina, USA.

Varun Goel (V)

Carolina Population Center, University of North Carolina, Chapel Hill, North Carolina, USA.

Paulin Essone (P)

Centre de Recherches Médicales de Lambaréné, Lambaréné, Gabon.

David Dosoo (D)

Kintampo Health Research Centre, Kintampo, Ghana.

Bright Adu (B)

Noguchi Memorial Institute for Medical Research, University of Ghana, Legon, Ghana.

Benedicta Ayiedu Mensah (BA)

Noguchi Memorial Institute for Medical Research, University of Ghana, Legon, Ghana.

Stephaney Gyaase (S)

Kintampo Health Research Centre, Kintampo, Ghana.

Kenneth Wiru (K)

Kintampo Health Research Centre, Kintampo, Ghana.

Fabrice Mougeni (F)

Centre de Recherches Médicales de Lambaréné, Lambaréné, Gabon.

Musah Osei (M)

Kintampo Health Research Centre, Kintampo, Ghana.

Pamela Minsoko (P)

Centre de Recherches Médicales de Lambaréné, Lambaréné, Gabon.

Cyrus Sinai (C)

Carolina Population Center, University of North Carolina, Chapel Hill, North Carolina, USA.

Karamoko Niaré (K)

Department of Pathology and Laboratory Medicine, Brown University, Providence, Rhode Island, USA.

Jonathan J Juliano (JJ)

Division of Infectious Diseases, School of Medicine, University of North Carolina, Chapel Hill, North Carolina, USA.

Michael Hudgens (M)

Department of Biostatistics, Gillings School of Global Public Health, University of North Carolina, Chapel Hill, North Carolina, USA.

Anita Ghansah (A)

Noguchi Memorial Institute for Medical Research, University of Ghana, Legon, Ghana.

Portia Kamthunzi (P)

University of North Carolina Project, Lilongwe, Malawi.

Tisungane Mvalo (T)

University of North Carolina Project, Lilongwe, Malawi.

Selidji Todagbe Agnandji (ST)

Centre de Recherches Médicales de Lambaréné, Lambaréné, Gabon.
Institute of Tropical Medicine, University of Tübingen, Tübingen, Germany.

Jeffrey A Bailey (JA)

Department of Pathology and Laboratory Medicine, Brown University, Providence, Rhode Island, USA.

Kwaku Poku Asante (KP)

Kintampo Health Research Centre, Kintampo, Ghana.

Michael Emch (M)

Department of Epidemiology, Gillings School of Global Public Health, University of North Carolina, Chapel Hill, North Carolina, USA.
Carolina Population Center, University of North Carolina, Chapel Hill, North Carolina, USA.

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