Dual antigen-targeted off-the-shelf NK cells show durable response and prevent antigen escape in lymphoma and leukemia.


Journal

Blood
ISSN: 1528-0020
Titre abrégé: Blood
Pays: United States
ID NLM: 7603509

Informations de publication

Date de publication:
08 12 2022
Historique:
accepted: 11 07 2022
received: 19 12 2021
pubmed: 3 8 2022
medline: 15 12 2022
entrez: 2 8 2022
Statut: ppublish

Résumé

Substantial numbers of B cell leukemia and lymphoma patients relapse due to antigen loss or heterogeneity after anti-CD19 chimeric antigen receptor (CAR) T cell therapy. To overcome antigen escape and address antigen heterogeneity, we engineered induced pluripotent stem cell-derived NK cells to express both an NK cell-optimized anti-CD19 CAR for direct targeting and a high affinity, non-cleavable CD16 to augment antibody-dependent cellular cytotoxicity. In addition, we introduced a membrane-bound IL-15/IL-15R fusion protein to promote in vivo persistence. These engineered cells, termed iDuo NK cells, displayed robust CAR-mediated cytotoxic activity that could be further enhanced with therapeutic antibodies targeting B cell malignancies. In multiple in vitro and xenogeneic adoptive transfer models, iDuo NK cells exhibited robust anti-lymphoma activity. Furthermore, iDuo NK cells effectively eliminated both CD19+ and CD19- lymphoma cells and displayed a unique propensity for targeting malignant cells over healthy cells that expressed CD19, features not achievable with anti-CAR19 T cells. iDuo NK cells combined with therapeutic antibodies represent a promising approach to prevent relapse due to antigen loss and tumor heterogeneity in patients with B cell malignancies.

Identifiants

pubmed: 35917442
pii: S0006-4971(22)00973-9
doi: 10.1182/blood.2021015184
pmc: PMC9918847
doi:

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2451-2462

Subventions

Organisme : NCI NIH HHS
ID : P01 CA065493
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA203348
Pays : United States
Organisme : NCI NIH HHS
ID : R35 CA197292
Pays : United States
Organisme : NCI NIH HHS
ID : P01 CA111412
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL155150
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2022 by The American Society of Hematology.

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Auteurs

Frank Cichocki (F)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Jodie P Goodridge (JP)

Fate Therapeutics, San Diego, CA.

Ryan Bjordahl (R)

Fate Therapeutics, San Diego, CA.

Sajid Mahmood (S)

Fate Therapeutics, San Diego, CA.

Zachary B Davis (ZB)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Svetlana Gaidarova (S)

Fate Therapeutics, San Diego, CA.

Ramzey Abujarour (R)

Fate Therapeutics, San Diego, CA.

Brian Groff (B)

Fate Therapeutics, San Diego, CA.

Alec Witty (A)

Fate Therapeutics, San Diego, CA.

Hongbo Wang (H)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Katie Tuininga (K)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Behiye Kodal (B)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Martin Felices (M)

Department of Medicine, University of Minnesota, Minneapolis, MN.

Greg Bonello (G)

Fate Therapeutics, San Diego, CA.

Janel Huffman (J)

Fate Therapeutics, San Diego, CA.

Thomas Dailey (T)

Fate Therapeutics, San Diego, CA.

Tom T Lee (TT)

Fate Therapeutics, San Diego, CA.

Bruce Walcheck (B)

Department of Veterinary and Biomedical Sciences, University of Minnesota, St. Paul, MN.

Bahram Valamehr (B)

Fate Therapeutics, San Diego, CA.

Jeffrey S Miller (JS)

Department of Medicine, University of Minnesota, Minneapolis, MN.

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Classifications MeSH