Clinical trajectories and biomarkers for weight variability in early Parkinson's disease.


Journal

NPJ Parkinson's disease
ISSN: 2373-8057
Titre abrégé: NPJ Parkinsons Dis
Pays: United States
ID NLM: 101675390

Informations de publication

Date de publication:
02 Aug 2022
Historique:
received: 03 02 2022
accepted: 13 07 2022
entrez: 2 8 2022
pubmed: 3 8 2022
medline: 3 8 2022
Statut: epublish

Résumé

Unexplained weight changes that occur in Parkinson's disease (PD), are often neglected and remain a poorly understood non-motor feature in patients with PD. A specific 'Park-weight' phenotype with low body weight has been described, and our aim was to evaluate the clinical and prognostic trajectories and biomarkers of weight variability in PD. We evaluated body weight-related biomarkers in 405 de novo PD patients and 187 healthy controls (HC) over a 5-year follow-up period from the PPMI database. Body-weight variability was defined as intra-individual variability in body weight between visits. PD patients were categorized as weight losers, gainers, or patients with stable weight. The differential progression of motor and non-motor clinical variables between groups was explored using linear mixed-effects models. Finally, we estimated longitudinal changes in weight as a function of baseline and longitudinal striatal presynaptic dopaminergic transporter imaging. PD patients presented a greater weight variability compared to HC (p = 0.003). Patients who developed weight loss had lower CSF amyloid-beta 1-42 (p = 0.009) at baseline. In addition, patients with weight loss showed a faster cognitive decline (p = 0.001), whereas patients with weight gain showed a slower motor progression (p = 0.001), compared to patients with stable weight. Baseline right striatal denervation was a predictor of weight variability in both PD patients and HC (p < 0.001). Similarly, weight variability in PD patients was associated with the progression of right striatal denervation (p < 0.001). Weight variability and specifically weight loss are more frequent in PD compared to HC, and are associated with specific motor, non-motor and cognitive progression patterns. A greater CSF amyloid burden was present at baseline in patients with subsequent weight loss. Presynaptic dopaminergic imaging in the right striatum may serve as a predictor of future weight changes in PD and HC.

Identifiants

pubmed: 35918350
doi: 10.1038/s41531-022-00362-3
pii: 10.1038/s41531-022-00362-3
pmc: PMC9345874
doi:

Types de publication

Journal Article

Langues

eng

Pagination

95

Informations de copyright

© 2022. The Author(s).

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Auteurs

Daniele Urso (D)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK. daniele.urso@kcl.ac.uk.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK. daniele.urso@kcl.ac.uk.
Center for Neurodegenerative Diseases and the Aging Brain, Department of Clinical Research in Neurology, University of Bari 'Aldo Moro', "Pia Fondazione Cardinale G. Panico", Tricase, Lecce, Italy. daniele.urso@kcl.ac.uk.

Daniel J van Wamelen (DJ)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK.
Radboud University Medical Center, Donders Institute for Brain, Cognition and Behaviour, Department of Neurology, Nijmegen, the Netherlands.

Lucia Batzu (L)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK.

Valentina Leta (V)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK.

Juliet Staunton (J)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK.

José A Pineda-Pardo (JA)

HM CINAC. Centro Integral de Neurociencias AC. HM Hospitales. Fundación de Investigación HM Hospitales. HM Hospitales, Madrid, Spain.
Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas Instituto Carlos III, Madrid, Spain.

Giancarlo Logroscino (G)

Center for Neurodegenerative Diseases and the Aging Brain, Department of Clinical Research in Neurology, University of Bari 'Aldo Moro', "Pia Fondazione Cardinale G. Panico", Tricase, Lecce, Italy.

Jagdish Sharma (J)

Geriatric Medicine (Movement Disorders), Lincoln County Hospital, Lincoln, United Kingdom, University of Lincoln, Lincoln, UK.

K Ray Chaudhuri (K)

King's College London, Department of Neurosciences, Institute of Psychiatry, Psychology & Neuroscience, London, UK.
Parkinson's Foundation Centre of Excellence, King's College Hospital, London, UK.

Classifications MeSH