COVID-19 outcomes in giant cell arteritis and polymyalgia rheumatica versus rheumatoid arthritis: A national, multicenter, cohort study.


Journal

Journal of autoimmunity
ISSN: 1095-9157
Titre abrégé: J Autoimmun
Pays: England
ID NLM: 8812164

Informations de publication

Date de publication:
10 2022
Historique:
received: 10 05 2022
revised: 07 07 2022
accepted: 08 07 2022
pubmed: 5 8 2022
medline: 23 11 2022
entrez: 4 8 2022
Statut: ppublish

Résumé

To determine whether giant cell arteritis and polymyalgia rheumatica (GCA/PMR) represent independent risk factors for worse outcomes in COVID-19. Observational, national, French, multicenter cohort (NCT04353609) comprising patients aged ≥18 years with confirmed diagnoses of either GCA, PMR or rheumatoid arthritis (RA) having presented COVID-19; those under rituximab were excluded. Primary endpoint was COVID-19 severity in GCA/PMR patients as compared to RA. We also aimed to describe the evolution of GCA/PMR patients following COVID-19. Multinomial logistic regression models were performed, with and without adjustment on pre-specified confounding factors (i.e., age, sex, body mass index, arterial hypertension, diabetes and cardiovascular disease). Unadjusted and adjusted multinomial odds-ratio (OR/aOR) and their 95% confidence intervals (CIs) were calculated as effect size using RA as reference group. Between April 15, 2020, and August 20, 2021, 674 patients [45 (6.6%) GCA, 47 (7.0%) PMR, 582 (86.4%) RA; 62.8 years, 73.2% female] were included. Compared to RA patients, those with GCA/PMR were older and more frequently presented hypertension, diabetes and cardiovascular disease. Severe COVID-19 and death occurred in 24 (26.1%) and 16 (17.8%) patients with GCA/PMR, respectively. Unadjusted analyses revealed higher odds of severe COVID-19 [OR = 3.32 (95% CI 1.89-5.83; p < 0.001)] and death [OR = 3.20 (95%CI 1.67-6.13; p < 0.001)] for GCA/PMR compared to RA. After model adjustment, these odds were attenuated. Patients with GCA/PMR were more likely to have severe COVID-19 and higher mortality compared to those with RA. This worse prognosis is mostly due to well known risk factors for the general population rather than vasculitis per se.

Identifiants

pubmed: 35926375
pii: S0896-8411(22)00076-2
doi: 10.1016/j.jaut.2022.102868
pmc: PMC9296684
pii:
doi:

Types de publication

Observational Study Multicenter Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102868

Informations de copyright

Copyright © 2022 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors have declared that no conflict of interest exists.

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Auteurs

Matheus Vieira (M)

Sorbonne Universités AP-HP, Groupe Hospitalier Pitié-Salpêtrière, Département de Médecine Interne et Immunologie Clinique, F-75013, Paris, France; Centre National de Références Maladies Autoimmunes Systémiques Rares, Centre National de Références Maladies Autoinflammatoires et Amylose Inflammatoire, France; Inflammation-Immunopathology-Biotherapy Department (DMU 3iD), France; INSERM 959, Groupe Hospitalier, AP-HP, Paris, France.

Cloé Comarmond (C)

Hôpital Lariboisière, Département de Médecine Interne et Immunologie, Clinique, Université, Paris, Cité, France.

Julien Labreuche (J)

Univ. Lille, CHU Lille, ULR 2694 - METRICS: Évaluation des technologies de Santé et des Pratiques Médicales, F-59000, Lille, France; CHU Lille, Department of Biostatistics, F-59000, Lille, France.

Adrien Mirouse (A)

Sorbonne Universités AP-HP, Groupe Hospitalier Pitié-Salpêtrière, Département de Médecine Interne et Immunologie Clinique, F-75013, Paris, France; Centre National de Références Maladies Autoimmunes Systémiques Rares, Centre National de Références Maladies Autoinflammatoires et Amylose Inflammatoire, France; Inflammation-Immunopathology-Biotherapy Department (DMU 3iD), France; INSERM 959, Groupe Hospitalier, AP-HP, Paris, France.

David Saadoun (D)

Sorbonne Universités AP-HP, Groupe Hospitalier Pitié-Salpêtrière, Département de Médecine Interne et Immunologie Clinique, F-75013, Paris, France; Centre National de Références Maladies Autoimmunes Systémiques Rares, Centre National de Références Maladies Autoinflammatoires et Amylose Inflammatoire, France; Inflammation-Immunopathology-Biotherapy Department (DMU 3iD), France; INSERM 959, Groupe Hospitalier, AP-HP, Paris, France.

Christophe Richez (C)

Service de Rhumatologie, Centre de Référence des Maladies Autoimmunes Systémiques Rares de L'Est et Du Sud-Ouest de France, CHU de Bordeaux and UMR-CNRS 5164, Université de Bordeaux, Bordeaux, France.

René-Marc Flipo (RM)

Service de Rhumatologie, Université Lille, CHU Lille, Lille, France.

Eric Hachulla (E)

University Lille, INSERM U995, CHU Lille, Service de Médecine Interne et Immunologie Clinique, Centre de Référence des Maladies Autoimmunes Systémiques rares Du Nord et Nord-Ouest de France (CeRAINO), U1286 - INFINITE - Institute for Translational Research in Inflammation, 59000, Lille, France.

Elodie Drumez (E)

Univ. Lille, CHU Lille, ULR 2694 - METRICS: Évaluation des technologies de Santé et des Pratiques Médicales, F-59000, Lille, France; CHU Lille, Department of Biostatistics, F-59000, Lille, France.

Patrice Cacoub (P)

Sorbonne Universités AP-HP, Groupe Hospitalier Pitié-Salpêtrière, Département de Médecine Interne et Immunologie Clinique, F-75013, Paris, France; Centre National de Références Maladies Autoimmunes Systémiques Rares, Centre National de Références Maladies Autoinflammatoires et Amylose Inflammatoire, France; Inflammation-Immunopathology-Biotherapy Department (DMU 3iD), France; INSERM 959, Groupe Hospitalier, AP-HP, Paris, France. Electronic address: patrice.cacoub@aphp.fr.

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