Use of a proline-specific endopeptidase to reintroduce gluten in patients with non-coeliac gluten sensitivity: A randomized trial.

Gastrointestinal symptoms Gluten re-introduction Gluten-free diet Non-coeliac gluten sensitivity Prolyl endoprotease

Journal

Clinical nutrition (Edinburgh, Scotland)
ISSN: 1532-1983
Titre abrégé: Clin Nutr
Pays: England
ID NLM: 8309603

Informations de publication

Date de publication:
09 2022
Historique:
received: 22 03 2022
revised: 15 07 2022
accepted: 15 07 2022
pubmed: 17 8 2022
medline: 9 9 2022
entrez: 16 8 2022
Statut: ppublish

Résumé

A gluten-free diet (GFD) is the main therapy for non-coeliac gluten sensitivity (NCGS). However, the availability of novel enzymes with the ability to digest gluten could represent a therapeutic opportunity for NCGS patients to avoid a GFD. To evaluate the controlled reintroduction of gluten with or without the endopeptidase P1016 in NCGS patients. This is a randomized, double-blind, placebo-controlled monocentric study, Registered under ClinicalTrials.gov Identifier no. NCT01864993. Gluten was reintroduced incrementally over a 3-week period under nutritional control. NCGS patients were randomized into two groups and administered P1016 or placebo during gluten reintroduction. We evaluated symptoms (visual analogue scale, VAS), quality of life (SF-36) and mental health symptoms (SCL-90) on a weekly basis. We enrolled a total 23 patients who were allocated to a placebo group (n = 11, age 38.4 ± 2.9) or an intervention group (n = 12, age 39.5 ± 3.1). No effect of P1016 on symptoms was found. During gluten reintroduction, patients reported a significant increase in abdominal pain and a worsening of stool consistency. Furthermore, no differences were found between the groups regarding SCL-90 and SF-36 scores. Our results demonstrate a lack of effect of P1016 in the management of NCGS patients and the possible reintroduction of gluten.

Sections du résumé

BACKGROUND
A gluten-free diet (GFD) is the main therapy for non-coeliac gluten sensitivity (NCGS). However, the availability of novel enzymes with the ability to digest gluten could represent a therapeutic opportunity for NCGS patients to avoid a GFD.
AIMS
To evaluate the controlled reintroduction of gluten with or without the endopeptidase P1016 in NCGS patients.
METHODS
This is a randomized, double-blind, placebo-controlled monocentric study, Registered under ClinicalTrials.gov Identifier no. NCT01864993. Gluten was reintroduced incrementally over a 3-week period under nutritional control. NCGS patients were randomized into two groups and administered P1016 or placebo during gluten reintroduction. We evaluated symptoms (visual analogue scale, VAS), quality of life (SF-36) and mental health symptoms (SCL-90) on a weekly basis.
RESULTS
We enrolled a total 23 patients who were allocated to a placebo group (n = 11, age 38.4 ± 2.9) or an intervention group (n = 12, age 39.5 ± 3.1). No effect of P1016 on symptoms was found. During gluten reintroduction, patients reported a significant increase in abdominal pain and a worsening of stool consistency. Furthermore, no differences were found between the groups regarding SCL-90 and SF-36 scores.
CONCLUSIONS
Our results demonstrate a lack of effect of P1016 in the management of NCGS patients and the possible reintroduction of gluten.

Identifiants

pubmed: 35973395
pii: S0261-5614(22)00273-4
doi: 10.1016/j.clnu.2022.07.029
pii:
doi:

Substances chimiques

Glutens 8002-80-0
Proline 9DLQ4CIU6V
Prolyl Oligopeptidases EC 3.4.21.26

Banques de données

ClinicalTrials.gov
['NCT01864993']

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2025-2030

Informations de copyright

Copyright © 2022 Elsevier Ltd and European Society for Clinical Nutrition and Metabolism. All rights reserved.

Déclaration de conflit d'intérêts

Conflict of interest The authors declare no conflict of interest.

Auteurs

A Scricciolo (A)

Center for Prevention and Diagnosis of Celiac Disease, Gastroenterology and Endoscopy Unit, Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan, Italy.

V Lombardo (V)

Center for Prevention and Diagnosis of Celiac Disease, Gastroenterology and Endoscopy Unit, Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan, Italy.

L Elli (L)

Center for Prevention and Diagnosis of Celiac Disease, Gastroenterology and Endoscopy Unit, Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan, Italy; Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy. Electronic address: luca.elli@policlinico.mi.it.

K A Bascuñán (KA)

Department of Nutrition, School of Medicine, University of Chile, Santiago, Chile.

L Doneda (L)

Department of Biomedical, Surgical and Dental Sciences, University of Milan, Milan, Italy.

F Rinaldi (F)

Human Advanced Microbiome Project-HMAP, Milan, Italy.

D Pinto (D)

Human Advanced Microbiome Project-HMAP, Milan, Italy.

M Araya (M)

Human Nutrition, Institute of Nutrition and Food Technology, INTA, University of Chile, Santiago, Chile.

A Costantino (A)

Center for Prevention and Diagnosis of Celiac Disease, Gastroenterology and Endoscopy Unit, Fondazione IRCCS Ca' Granda Ospedale Maggiore Policlinico, Milan, Italy.

M Vecchi (M)

Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy.

L Roncoroni (L)

Department of Biomedical, Surgical and Dental Sciences, University of Milan, Milan, Italy.

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