[Effect and mechanism of thymosin beta 4 on spinal cord-derived neural stem /progenitor cell injury induced by oxidative stress].
Animals
Apoptosis
Calcium
/ metabolism
Cell Survival
Hydrogen Peroxide
/ metabolism
Male
Myeloid Differentiation Factor 88
/ genetics
Oxidative Stress
Rats
Rats, Sprague-Dawley
Reactive Oxygen Species
/ metabolism
Spinal Cord Injuries
/ drug therapy
Stem Cells
Superoxide Dismutase
/ metabolism
Thymosin
/ metabolism
Toll-Like Receptor 4
/ genetics
Myeloid differentiation factor 88
Neural stem cells
Oxidative stress injury
Progenitor cells
Thymosin β4
Journal
Zhongguo gu shang = China journal of orthopaedics and traumatology
ISSN: 1003-0034
Titre abrégé: Zhongguo Gu Shang
Pays: China
ID NLM: 9815790
Informations de publication
Date de publication:
25 Aug 2022
25 Aug 2022
Historique:
entrez:
18
8
2022
pubmed:
19
8
2022
medline:
20
8
2022
Statut:
ppublish
Résumé
To investigate the role and mechanism of thymosin beta 4 (Tβ4) in oxidative stress injury of spinal cord-derived neural stem/progenitor cells (NSPCs) induced by hydrogen peroxide (H NSPCs were isolated from Sprague-Dawley (SD) adult male rats, and divided into control group (untreated NSPCs cells), H The expression of Tβ4 was decreased in H Tβ4 attenuates H
Identifiants
pubmed: 35979771
doi: 10.12200/j.issn.1003-0034.2022.08.012
doi:
Substances chimiques
Myeloid Differentiation Factor 88
0
Reactive Oxygen Species
0
Toll-Like Receptor 4
0
Thymosin
61512-21-8
Hydrogen Peroxide
BBX060AN9V
Superoxide Dismutase
EC 1.15.1.1
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
chi
Sous-ensembles de citation
IM