Expression of the Calcium-Binding Protein CALB1 Is Induced and Controls Intracellular Ca
CALB1
Ca2+
NFAT
cellular senescence
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
19 Aug 2022
19 Aug 2022
Historique:
received:
01
07
2022
revised:
12
08
2022
accepted:
16
08
2022
entrez:
26
8
2022
pubmed:
27
8
2022
medline:
30
8
2022
Statut:
epublish
Résumé
In response to many stresses, such as oncogene activation or DNA damage, cells can enter cellular senescence, a state of proliferation arrest accompanied by a senescence-associated secretory phenotype (SASP). Cellular senescence plays a key role in many physiopathological contexts, including cancer, aging and aging-associated diseases, therefore, it is critical to understand how senescence is regulated. Calcium ions (Ca
Identifiants
pubmed: 36012633
pii: ijms23169376
doi: 10.3390/ijms23169376
pmc: PMC9409414
pii:
doi:
Substances chimiques
CALB1 protein, human
0
Calbindin 1
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : French National Cancer Institute
ID : 2018-144
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