Relationships between the expression of adipose genes and profiles of hospitalized dogs.


Journal

Veterinary research communications
ISSN: 1573-7446
Titre abrégé: Vet Res Commun
Pays: Switzerland
ID NLM: 8100520

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 16 06 2022
accepted: 23 08 2022
pubmed: 2 9 2022
medline: 26 11 2022
entrez: 1 9 2022
Statut: ppublish

Résumé

Obesity is one of the risk factors for the onset of various metabolic diseases in dogs. Energy expenditure in brown/beige adipocytes, which is partially regulated by the bone morphogenetic protein (BMP) pathway, is a key factor determining systemic energy balance. Here, we examined gene expression in the fat depots of 129 hospitalized dogs, and the relationship between the relative levels of gene expression and profiles of dogs. We evaluated the expression levels of 23 genes such as regulatory genes of adipocyte differentiation and function, adipokines, genes related to brown adipogenesis and uncoupling protein (Ucp), and genes involved in BMP signaling. A reliable equation of multiple regression was not obtained to explain the body condition score (BCS), which is an index of adiposity. Positive relationships were detected between the expression levels of many genes, except for Ucp1 or Ucp3. BCS was found to increase with age. BCS was negatively correlated to the expression levels of Pparγ and Fasn, and positively correlated to Leptin and Opn3 expression. Aging decreased the expression levels of genes related to adipocyte differentiation and function (Pparγ, Fabp4, Fasn, Hsl, and Insr) and Adipoq. In addition, age was negatively correlated with the expression of genes involved in brown adipogenesis and BMP signaling components (Prdm16, Bmp4, Alk3, Actr2a, and Actr2b). In contrast, the expression levels of Leptin and Ucp2 were found to increase with age. The present study clarifies BCS- and age-related gene expressions in the adipose tissue, which potentially contribute to elucidating the etiology of canine obesity.

Identifiants

pubmed: 36048336
doi: 10.1007/s11259-022-09989-2
pii: 10.1007/s11259-022-09989-2
doi:

Substances chimiques

Leptin 0
PPAR gamma 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1239-1244

Subventions

Organisme : MEXT-Supported Program for the Private University Research Branding Project
ID : 2016-2020
Organisme : Japan Society for the Promotion of Science
ID : 21K05963

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer Nature B.V.

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Auteurs

Yukina Sugiyama (Y)

Laboratory of Molecular Biology, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan.
Sugiyama Animal Hospital, Shizuoka, 424-0068, Japan.

Fumie Shimokawa (F)

Laboratory of Molecular Biology, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan.

Kazutoshi Sugiyama (K)

Laboratory of Molecular Biology, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan.
Sugiyama Animal Hospital, Shizuoka, 424-0068, Japan.

Takashi Kobayashi (T)

Kobayashi Animal Hospital, Nagano, 380-0816, Japan.

Yusuke Yamashita (Y)

Aoi Animal Hospital, Shizuoka, 420-0076, Japan.

Kei Kazama (K)

Laboratory of Farm Animal Internal Medicine, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan.

Ken Onda (K)

Laboratory of Farm Animal Internal Medicine, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan.

Masayuki Funaba (M)

Division of Applied Biosciences, Kyoto University Graduate School of Agriculture, Kyoto, 606-8502, Japan. funaba.masayuki.8w@kyoto-u.ac.jp.

Masaru Murakami (M)

Laboratory of Molecular Biology, Azabu University School of Veterinary Medicine, Sagamihara, 252-5201, Japan. murakami@azabu-u.ac.jp.

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