Comparative effectiveness of a second-line biologic in patients with ulcerative colitis: vedolizumab followed by an anti-TNF versus anti-TNF followed by vedolizumab.

IBD clinical inflammatory bowel disease ulcerative colitis

Journal

Frontline gastroenterology
ISSN: 2041-4137
Titre abrégé: Frontline Gastroenterol
Pays: England
ID NLM: 101528589

Informations de publication

Date de publication:
2022
Historique:
received: 10 05 2021
accepted: 26 10 2021
entrez: 2 9 2022
pubmed: 3 9 2022
medline: 3 9 2022
Statut: epublish

Résumé

Sequential drug treatment with biological agents in ulcerative colitis (UC) is becoming increasingly complex. There are few studies comparing head-to-head outcomes in second-line treatments. The study assesses whether using anti-tumour necrosis factor (anti-TNF)-α therapy following the α4β7 integrin blocker vedolizumab (VDZ) or VDZ after an anti-TNF has more favourable clinical outcomes in UC in a real-world outpatient setting. Patients with UC who were exposed to first-line anti-TNF (adalimumab or infliximab) or VDZ who subsequently switched to the alternate class between May 2013 and August 2020 were identified by reviewing patient databases at 10 hospitals. Data were collected retrospectively using patient records. Baseline demographics, disease activity indices, biochemical markers, endoscopic Mayo score, colectomy rates, treatment persistence and urgent hospital utilisation composite endpoint (UHUC) rates were examined over a 52-week period. Second-line week 52 treatment persistence was higher in the VDZ group (71/81, 89%) versus the anti-TNF group (15/34, 44%; p=0.0001), as were week 52 colectomy-free survival (VDZ: 77/80, 96%, vs anti-TNF: 26/32, 81%; p=0.009), week 52 UHUC survival (VDZ: 68/84, 81%, vs anti-TNF: 20/34, 59%; p=0.002) and week 52 corticosteroid-free clinical remission (CFCR) rates (VDZ: 22/34, 65%, vs anti-TNF: 4/20, 20%; p=0.001). Compared with second-line anti TNF usage, the VDZ second-line cohort had significantly higher 52-week treatment persistence, UHUC survival, higher colectomy-free survival rates and higher week 52 CFCR. These data suggest that VDZ is an effective biologic in UC as a second-line therapy after anti-TNF exposure. It highlights the effect of biological order on clinically important outcomes.

Sections du résumé

Background UNASSIGNED
Sequential drug treatment with biological agents in ulcerative colitis (UC) is becoming increasingly complex. There are few studies comparing head-to-head outcomes in second-line treatments. The study assesses whether using anti-tumour necrosis factor (anti-TNF)-α therapy following the α4β7 integrin blocker vedolizumab (VDZ) or VDZ after an anti-TNF has more favourable clinical outcomes in UC in a real-world outpatient setting.
Methods UNASSIGNED
Patients with UC who were exposed to first-line anti-TNF (adalimumab or infliximab) or VDZ who subsequently switched to the alternate class between May 2013 and August 2020 were identified by reviewing patient databases at 10 hospitals. Data were collected retrospectively using patient records. Baseline demographics, disease activity indices, biochemical markers, endoscopic Mayo score, colectomy rates, treatment persistence and urgent hospital utilisation composite endpoint (UHUC) rates were examined over a 52-week period.
Results UNASSIGNED
Second-line week 52 treatment persistence was higher in the VDZ group (71/81, 89%) versus the anti-TNF group (15/34, 44%; p=0.0001), as were week 52 colectomy-free survival (VDZ: 77/80, 96%, vs anti-TNF: 26/32, 81%; p=0.009), week 52 UHUC survival (VDZ: 68/84, 81%, vs anti-TNF: 20/34, 59%; p=0.002) and week 52 corticosteroid-free clinical remission (CFCR) rates (VDZ: 22/34, 65%, vs anti-TNF: 4/20, 20%; p=0.001).
Conclusion UNASSIGNED
Compared with second-line anti TNF usage, the VDZ second-line cohort had significantly higher 52-week treatment persistence, UHUC survival, higher colectomy-free survival rates and higher week 52 CFCR. These data suggest that VDZ is an effective biologic in UC as a second-line therapy after anti-TNF exposure. It highlights the effect of biological order on clinically important outcomes.

Identifiants

pubmed: 36051959
doi: 10.1136/flgastro-2021-101906
pii: flgastro-2021-101906
pmc: PMC9380760
doi:

Types de publication

Journal Article

Langues

eng

Pagination

392-401

Informations de copyright

© Author(s) (or their employer(s)) 2022. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: SB has acted as an advisor for Takeda, Johnson & Johnson, Sublimity Therapeutics, Tillotts and Janssen. MAS served as a speaker, a consultant and/or advisory board member for Janssen, Sandoz, Takeda, MSD, Falk, Samsung Bioepis, Galapagos and Bristol-Myers Squibb.

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Auteurs

Charles Miller (C)

Department of Gastroenterology, University College Hospital, London, UK.

Hanson Kwok (H)

Department of Gastroenterology, University College Hospital, London, UK.

Paul Harrow (P)

Department of Gastroenterology, University College Hospital, London, UK.

Roser Vega (R)

Department of Gastroenterology, University College Hospital, London, UK.

Edward Seward (E)

Department of Gastroenterology, University College Hospital, London, UK.

Shameer Mehta (S)

Department of Gastroenterology, University College Hospital, London, UK.

Farooq Rahman (F)

Department of Gastroenterology, University College Hospital, London, UK.

Sara McCartney (S)

Department of Gastroenterology, University College Hospital, London, UK.

Ioanna Parisi (I)

Department of Gastroenterology, University College Hospital, London, UK.

Samuel Hsiang Lim (SH)

Department of Gastroenterology, Guy's and Thomas' Hospital, London, UK.

Esha Sharma (E)

Department of Gastroenterology, Guy's and Thomas' Hospital, London, UK.

Mark A Samaan (MA)

Department of Gastroenterology, Guy's and Thomas' Hospital, London, UK.

Aaron Bancil (A)

Department of Gastroenterology, Royal London Hospital, London, UK.

Klaartje Bel Kok (KB)

Department of Gastroenterology, Royal London Hospital, London, UK.

Ahmed Shalabi (A)

Department of Gastroenterology, West Middlesex Hospital, London, UK.

Emma L Johnston (EL)

Department of Gastroenterology, West Middlesex Hospital, London, UK.

Dev Katarey (D)

Department of Gastroenterology, Royal Free Hospital London, London, UK.

Nina Taherzadeh (N)

Department of Gastroenterology, Royal Free Hospital London, London, UK.

Charles Murray (C)

Department of Gastroenterology, Royal Free Hospital London, London, UK.

Mohammed Tauseef Sharip (MT)

Department of Gastroenterology, Lister Hospital, Stevenage, UK.

Martyn J Carter (MJ)

Department of Gastroenterology, Lister Hospital, Stevenage, UK.

Shiva T Radhakrishnan (ST)

Departments of Gastroenterology and Hepatology, St Mary's Hospital, Imperial College Healthcare NHS Trust, London, UK.

Simon Peake (S)

Departments of Gastroenterology and Hepatology, St Mary's Hospital, Imperial College Healthcare NHS Trust, London, UK.

Imran Khakoo (I)

Departments of Gastroenterology, Chelsea and Westminster Hospital, London, UK.

Mahmood Wahed (M)

Departments of Gastroenterology, Chelsea and Westminster Hospital, London, UK.

Sebastian Povlsen (S)

Department of Gastroenterology, King's College Hospital, London, UK.

Mehul Patel (M)

Department of Gastroenterology, King's College Hospital, London, UK.

Patrick DuBois (P)

Department of Gastroenterology, King's College Hospital, London, UK.

Jemima Finkel (J)

Department of Gastroenterology, Whittington Hospital, London, UK.

Clive Onnie (C)

Department of Gastroenterology, Whittington Hospital, London, UK.

Stuart Bloom (S)

Department of Gastroenterology, University College Hospital, London, UK.

Classifications MeSH