Epileptic encephalopathy as a new feature of the sudden infant death with dysgenesis of the testes syndrome caused by TSPYL1 variants.


Journal

American journal of medical genetics. Part A
ISSN: 1552-4833
Titre abrégé: Am J Med Genet A
Pays: United States
ID NLM: 101235741

Informations de publication

Date de publication:
12 2022
Historique:
revised: 13 05 2022
received: 18 02 2022
accepted: 25 07 2022
pubmed: 10 9 2022
medline: 15 11 2022
entrez: 9 9 2022
Statut: ppublish

Résumé

Sudden infant death with dysgenesis of the testes syndrome (SIDDT) is a rare autosomal recessive disorder associating developmental sex disorder (DSD) in patients with 46,XY karyotype and visceroautonomic dysfunction responsible for sudden infant death. First described in 2004, very few patients have since been reported. We describe here a new patient with SIDDT and epileptic encephalopathy (EE). We provide the phenotypic description and genetic results of a boy carrying biallelic TSPYL1 deleterious variants. We also reviewed the data of the 26 previously described patients with SIDDT. Our patient presented gonadal dysgenesis, cardio-respiratory dysfunction, and repeated seizures, leading in 1 month to severe intractable EE. He died at age 10 months of cardiorespiratory arrest. Four other reported patients from two families presented with progressive epilepsy, including one with severe EE. No similar phenotype was described in the 22 other patients and the recurrent variant p.Val242Glufs*52 appears to be more frequently associated with seizures. To note, our patient is the first case with compound heterozygous TSPYL1 variants. These findings expand the phenotypic spectrum of SIDDT by reporting progressive epilepsy and severe EE as a possible outcome. This information may help in managing patients with SIDDT.

Identifiants

pubmed: 36082874
doi: 10.1002/ajmg.a.62966
doi:

Substances chimiques

TSPYL1 protein, human 0
Nuclear Proteins 0

Types de publication

Case Reports Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

3540-3545

Informations de copyright

© 2022 Wiley Periodicals LLC.

Références

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Auteurs

Benoit Mazel (B)

Centre de Génétique et Centre de Référence Anomalies du Développement et Syndromes Malformatifs, FHUTRANSLAD - CHU Dijon Bourgogne, Dijon, France.
Fédération Hospitalo-Universitaire Médecine Translationnelle et Anomalies du Développement (FHUTRANSLAD), CHU Dijon Bourgogne et Université de Bourgogne-Franche Comté, Dijon, France.
Laboratoire de Génétique Chromosomique et Moléculaire, UF Innovation en Diagnostic Génomique des Maladies Rares, Centre Hospitalier Universitaire de Dijon, Dijon, France.

Delphine Mallet (D)

Hospices Civils de Lyon, Groupement Hospitalier Est, Laboratoire de Biochimie et Biologie Moléculaire Grand Est, UM Pathologies Endocriniennes et Mucoviscidose, Bron, France.
Centre de Référence du Développement Génital: du Fœtus à l'Adulte, Filière Maladies Rares Endocriniennes, Bron, France.

Florence Roucher-Boulez (F)

Hospices Civils de Lyon, Groupement Hospitalier Est, Laboratoire de Biochimie et Biologie Moléculaire Grand Est, UM Pathologies Endocriniennes et Mucoviscidose, Bron, France.
Centre de Référence du Développement Génital: du Fœtus à l'Adulte, Filière Maladies Rares Endocriniennes, Bron, France.
Université de Lyon, Université Claude-Bernard Lyon 1, Lyon, France.

Candace Ben Signor (CB)

Department of Endocrino-Pediatry, Dijon University Hospital, Dijon, France.

Marie Bournez (M)

Centre de Génétique et Centre de Référence Anomalies du Développement et Syndromes Malformatifs, FHUTRANSLAD - CHU Dijon Bourgogne, Dijon, France.

Véronique Darmency (V)

Service de Neurophysiologie Clinique, Hôpital d'Enfants, CHU Dijon Bourgogne, Dijon, France.

Valentin Bourgeois (V)

Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Charlotte Poe (C)

Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Fares El Khabbaz (F)

Service de Soins Intensifs Néonataux, Centre Hospitalier d'Auxerre, Auxerre, France.

Antonio Vitobello (A)

Laboratoire de Génétique Chromosomique et Moléculaire, UF Innovation en Diagnostic Génomique des Maladies Rares, Centre Hospitalier Universitaire de Dijon, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Christophe Philippe (C)

Laboratoire de Génétique Chromosomique et Moléculaire, UF Innovation en Diagnostic Génomique des Maladies Rares, Centre Hospitalier Universitaire de Dijon, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Yannis Duffourd (Y)

Laboratoire de Génétique Chromosomique et Moléculaire, UF Innovation en Diagnostic Génomique des Maladies Rares, Centre Hospitalier Universitaire de Dijon, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Christel Thauvin-Robinet (C)

Centre de Génétique et Centre de Référence Anomalies du Développement et Syndromes Malformatifs, FHUTRANSLAD - CHU Dijon Bourgogne, Dijon, France.
Fédération Hospitalo-Universitaire Médecine Translationnelle et Anomalies du Développement (FHUTRANSLAD), CHU Dijon Bourgogne et Université de Bourgogne-Franche Comté, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Laurence Faivre (L)

Centre de Génétique et Centre de Référence Anomalies du Développement et Syndromes Malformatifs, FHUTRANSLAD - CHU Dijon Bourgogne, Dijon, France.
Fédération Hospitalo-Universitaire Médecine Translationnelle et Anomalies du Développement (FHUTRANSLAD), CHU Dijon Bourgogne et Université de Bourgogne-Franche Comté, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

Sophie Nambot (S)

Centre de Génétique et Centre de Référence Anomalies du Développement et Syndromes Malformatifs, FHUTRANSLAD - CHU Dijon Bourgogne, Dijon, France.
Fédération Hospitalo-Universitaire Médecine Translationnelle et Anomalies du Développement (FHUTRANSLAD), CHU Dijon Bourgogne et Université de Bourgogne-Franche Comté, Dijon, France.
Inserm UMR1231 GAD, Génétique des Anomalies du Développement, Université de Bourgogne, Dijon, France.

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