Progesterone withdrawal and parturition.


Journal

The Journal of steroid biochemistry and molecular biology
ISSN: 1879-1220
Titre abrégé: J Steroid Biochem Mol Biol
Pays: England
ID NLM: 9015483

Informations de publication

Date de publication:
11 2022
Historique:
received: 23 06 2021
revised: 30 08 2022
accepted: 07 09 2022
medline: 23 10 2023
pubmed: 13 9 2022
entrez: 12 9 2022
Statut: ppublish

Résumé

The steroid hormone progesterone (P4), acting via the nuclear P4 receptors (PRs) in uterine cells, is essential for the establishment and maintenance of pregnancy. P4/PR signaling maintains pregnancy by promoting uterine quiescence and blocking the contractions of labor. Withdrawal of the P4/PR block to labor induces parturition. The success of pregnancy requires the timely birth of a mature neonate to a healthy mother, and to this end, the mechanism by which the P4/PR block is withdrawn, and how that process is physiologically controlled is critical. This review examines current understanding of cell and molecular biology of P4/PR withdrawal in the control of parturition.

Identifiants

pubmed: 36096351
pii: S0960-0760(22)00128-5
doi: 10.1016/j.jsbmb.2022.106177
pii:
doi:

Substances chimiques

Progesterone 4G7DS2Q64Y
Receptors, Progesterone 0

Types de publication

Review Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

106177

Informations de copyright

Copyright © 2022. Published by Elsevier Ltd.

Auteurs

Sam Mesiano (S)

William H Weir MD Professor of Reproductive Biology, Department of Reproductive Biology Case, Western Reserve University, USA; Department of Obstetrics and Gynecology, University Hospitals of Cleveland, 11100 Euclid Ave, Cleveland, OH 44106, USA. Electronic address: sam.mesiano@case.edu.

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Classifications MeSH