Improving obesity and lipid metabolism using conjugated linoleic acid.


Journal

Veterinary medicine and science
ISSN: 2053-1095
Titre abrégé: Vet Med Sci
Pays: England
ID NLM: 101678837

Informations de publication

Date de publication:
11 2022
Historique:
pubmed: 16 9 2022
medline: 23 11 2022
entrez: 15 9 2022
Statut: ppublish

Résumé

Conjugated linoleic acid (CLA) can prevent fatty acid accumulation induced by a high-fructose diet and improve lipid metabolism disorders in patients. We aimed to investigate the effect of CLA on obesity and lipid metabolism and its possible mechanism. Eight-month-old male BKS.Cg-Dock7 CLA treatment notably reduced the dietary and water intake of db/db mice, effectively reduced body weight, and decreased serum TG and TC levels (p < 0.05). Increased expression of PPARα (p < 0.05) and decreased expression of CD36 (p < 0.001) were observed in the liver of mice that were fed CLA. CLA increased PPARα expression (p < 0.001) and decreased PPARγ (p < 0.001) and CD36 expression (p < 0.01) in HepG2 cells. Our results showed that CLA can improve lipid metabolism in obese mice through upregulation of PPARα expression and downregulation of CD36 expression.

Sections du résumé

BACKGROUND
Conjugated linoleic acid (CLA) can prevent fatty acid accumulation induced by a high-fructose diet and improve lipid metabolism disorders in patients.
OBJECTIVES
We aimed to investigate the effect of CLA on obesity and lipid metabolism and its possible mechanism.
METHODS
Eight-month-old male BKS.Cg-Dock7
RESULTS
CLA treatment notably reduced the dietary and water intake of db/db mice, effectively reduced body weight, and decreased serum TG and TC levels (p < 0.05). Increased expression of PPARα (p < 0.05) and decreased expression of CD36 (p < 0.001) were observed in the liver of mice that were fed CLA. CLA increased PPARα expression (p < 0.001) and decreased PPARγ (p < 0.001) and CD36 expression (p < 0.01) in HepG2 cells.
CONCLUSIONS
Our results showed that CLA can improve lipid metabolism in obese mice through upregulation of PPARα expression and downregulation of CD36 expression.

Identifiants

pubmed: 36104831
doi: 10.1002/vms3.921
pmc: PMC9677407
doi:

Substances chimiques

Linoleic Acids, Conjugated 0
PPAR alpha 0
PPAR gamma 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2538-2544

Informations de copyright

© 2022 The Authors. Veterinary Medicine and Science published by John Wiley & Sons Ltd.

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Auteurs

Ye Sun (Y)

Department of General Practice, Zhujiang Hospital, Southern Medical University, Guangzhou, China.

Xufeng Hou (X)

Department of Cell Biology, School of Basic Medicine, Southern Medical University, Guangzhou, China.

Lingjie Li (L)

Department of Cell Biology, School of Basic Medicine, Southern Medical University, Guangzhou, China.

Yanqing Tang (Y)

Department of Cell Biology, School of Basic Medicine, Southern Medical University, Guangzhou, China.

Mingyue Zheng (M)

Department of Cell Biology, School of Basic Medicine, Southern Medical University, Guangzhou, China.

Weisen Zeng (W)

Department of Cell Biology, School of Basic Medicine, Southern Medical University, Guangzhou, China.

XiaoLong Lei (X)

Department of Nutrition, Nanfang Hospital, Southern Medical University, Guangzhou, China.

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Classifications MeSH