Maturation of circulating Ly6C
EAE
MDSC
PD-L1, Cre/loxP mouse
demyelination
immunotherapy
monocyte maturation
multiple sclerosis
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2022
2022
Historique:
received:
17
06
2022
accepted:
03
08
2022
entrez:
26
9
2022
pubmed:
27
9
2022
medline:
28
9
2022
Statut:
epublish
Résumé
Autoimmune diseases affecting the CNS not only overcome immune privilege mechanisms that protect neural tissues but also peripheral immune tolerance mechanisms towards self. Together with antigen-specific T cells, myeloid cells are main effector cells in CNS autoimmune diseases such as multiple sclerosis, but the relative contributions of blood-derived monocytes and the tissue resident macrophages to pathology and repair is incompletely understood. Through the study of oxidized mannan-conjugated myelin oligodendrocyte glycoprotein 35-55 (OM-MOG), we show that peripheral maturation of Ly6C
Identifiants
pubmed: 36159850
doi: 10.3389/fimmu.2022.972003
pmc: PMC9501702
doi:
Substances chimiques
Antigens, Ly
0
Ccr2 protein, mouse
0
Ly-6C antigen, mouse
0
Mannans
0
Mog protein, mouse
0
Myelin-Oligodendrocyte Glycoprotein
0
Peptides
0
Receptors, CCR2
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
972003Informations de copyright
Copyright © 2022 Dagkonaki, Papalambrou, Avloniti, Gkika, Evangelidou, Androutsou, Tselios and Probert.
Déclaration de conflit d'intérêts
M-EA was employed by the company Vianex S.A. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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