Evaluation of miR-371a-3p to predict viable germ cell tumor in patients with pure seminoma receiving retroperitoneal lymph node dissection.


Journal

Andrology
ISSN: 2047-2927
Titre abrégé: Andrology
Pays: England
ID NLM: 101585129

Informations de publication

Date de publication:
05 2023
Historique:
revised: 01 09 2022
received: 09 06 2022
accepted: 30 09 2022
medline: 4 4 2023
pubmed: 19 10 2022
entrez: 18 10 2022
Statut: ppublish

Résumé

Conventional serum tumor markers (STMs) for testicular germ cell tumors (GCTs) offer limited performance with particularly poor sensitivity in cases of minimal residual disease and pure seminoma. While growing evidence has indicated miR-371a-3p to be a superior biomarker, its utility in detecting pure seminoma at recurrence has not been extensively explored. This study's objective was to explore miR-371a-3p's utility in detecting metastatic pure seminoma at retroperitoneal lymph node dissection (RPLND). RNA was isolated from patient serum samples collected pre-RPLND. Fifteen patients were assigned to our 'benign' (n = 6) or 'seminoma' (n = 9) group based on pathological confirmation of viable seminoma. Five of the patients received chemotherapy before RPLND (PC-RPLND), and 10 were chemotherapy naïve. MiR-371a-3p expression was quantified via RT-quantitative polymerase chain reaction. The Cq values were statistically evaluated to obtain performance measurements. Median relative expression of miR-371a-3p was higher in the Seminoma group than the Benign, but this difference was not statistically significant (Rq = 3705 and 241, respectively, p = 0.2844). Of the 10 chemotherapy naïve patients, nine had viable seminoma at RPLND, and seven had elevated miR-371a-3p expression. Among the five postchemotherapy (PC) patients, zero had viable GCT at RPLND, and two had elevated miR-371a-3p expression. The primary RPLND group presented 78% sensitivity and 100% specificity. Specificity in the PC-RPLND group was 60%. An optimal Rq threshold of 28.62 was determined by Youden's J statistic, yielding 78% sensitivity and 67% specificity. Receiver operating characteristic analysis provided an AUC of 0.704 (95% CI: 0.43-0.98, p = 0.1949). Despite modest performance, miR-371a-3p exhibited improved sensitivity and specificity compared with conventional STMs. MiR-371a-3p outperformed STMs in the primary RPLND settings. However, miR-371a-3p was not a robust predictor of pathology in the PC setting. These results suggest that pure seminoma at RPLND is a clinical context, wherein the miRNA assay may require further refinement.

Identifiants

pubmed: 36254623
doi: 10.1111/andr.13317
doi:

Substances chimiques

MicroRNAs 0
Biomarkers, Tumor 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

634-640

Informations de copyright

© 2022 American Society of Andrology and European Academy of Andrology.

Références

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Auteurs

Bendu Konneh (B)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Pediatrics, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

John T Lafin (JT)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Jeffrey Howard (J)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Thomas Gerald (T)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Armon Amini (A)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Anna Savelyeva (A)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Solomon L Woldu (SL)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Cheryl M Lewis (CM)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Liwei Jia (L)

Department of Pathology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.

Vitaly Margulis (V)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Urology, I.M. Sechenov First Moscow State University, Moscow, Russia.

Nicholas Coleman (N)

Department of Pathology, University of Cambridge, Cambridge, UK.
Department of Histopathology, Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

Cinzia Scarpini (C)

Department of Pathology, University of Cambridge, Cambridge, UK.

A Lindsay Frazier (AL)

Department of Pediatric Hematology and Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA.

Matthew J Murray (MJ)

Department of Pathology, University of Cambridge, Cambridge, UK.
Department of Paediatric Hematology and Oncology, Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

James F Amatruda (JF)

Cancer and Blood Disease Institute, Children's Hospital Los Angeles, Los Angeles, California, USA.

Aditya Bagrodia (A)

Department of Urology, University of Texas Southwestern Medical Center, Dallas, Texas, USA.
Department of Urology, University of California San Diego, San Diego, California, USA.

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