Association of blood-based biomarkers with radiologic markers and cognitive decline in atrial fibrillation patients.


Journal

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association
ISSN: 1532-8511
Titre abrégé: J Stroke Cerebrovasc Dis
Pays: United States
ID NLM: 9111633

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 16 08 2022
revised: 28 09 2022
accepted: 11 10 2022
pubmed: 30 10 2022
medline: 24 11 2022
entrez: 29 10 2022
Statut: ppublish

Résumé

Atrial fibrillation (AF) has been associated with an increased risk of silent brain infarcts (SBI) and cognitive impairment, even in patients with low embolic risk. We aimed to test the association between 11 blood-biomarkers representing different AF-related pathways, and SBI, white matter hyperintensities (WMH), and cognitive decline in patients with AF and low embolic risk. The present study followed a cross-sectional design. 70 patients with a history of AF and CHADS 45 patients presented SBI in the MRI, and 25 did not. Ang-2, FGF-23, and BMP-10 were increased in patients with SBI. Ang-2 was elevated only in patients with embolic infarcts, whereas FGF-23 and BMP-10 tended to be elevated in patients with both types of infarcts. Ang-2 (OR = 1.56 [0.94-2.59], p = 0.087), and BMP-10 (OR = 4.83 [0.99-23.60], p = 0.052) were the biomarkers that showed the highest association with SBI when entered in a multivariable logistic regression model corrected by age. No biomarker was found associated with WMH or mild cognitive impairment. BMP-10, and Ang-2 were increased in patients with SBI. Its usefulness to detect SBI in AF patients should be further explored.

Sections du résumé

BACKGROUND BACKGROUND
Atrial fibrillation (AF) has been associated with an increased risk of silent brain infarcts (SBI) and cognitive impairment, even in patients with low embolic risk. We aimed to test the association between 11 blood-biomarkers representing different AF-related pathways, and SBI, white matter hyperintensities (WMH), and cognitive decline in patients with AF and low embolic risk.
METHODS METHODS
The present study followed a cross-sectional design. 70 patients with a history of AF and CHADS
RESULTS RESULTS
45 patients presented SBI in the MRI, and 25 did not. Ang-2, FGF-23, and BMP-10 were increased in patients with SBI. Ang-2 was elevated only in patients with embolic infarcts, whereas FGF-23 and BMP-10 tended to be elevated in patients with both types of infarcts. Ang-2 (OR = 1.56 [0.94-2.59], p = 0.087), and BMP-10 (OR = 4.83 [0.99-23.60], p = 0.052) were the biomarkers that showed the highest association with SBI when entered in a multivariable logistic regression model corrected by age. No biomarker was found associated with WMH or mild cognitive impairment.
CONCLUSIONS CONCLUSIONS
BMP-10, and Ang-2 were increased in patients with SBI. Its usefulness to detect SBI in AF patients should be further explored.

Identifiants

pubmed: 36309005
pii: S1052-3057(22)00527-4
doi: 10.1016/j.jstrokecerebrovasdis.2022.106833
pii:
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

106833

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest Neurovascular research laboratory, Vall d'Hebron Institute of Research (VHIR) received institutional research support from Roche Diagnostics that funded part of the present study. JM and AB have participated in scientific advisory boards organized by ROCHE on the topic of “New biomarkers in Atrial Fibrillation”.

Auteurs

Elena Palà (E)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research (VHIR)-Universitat Autónoma de Barcelona, Barcelona, Spain.

Irene Escudero-Martínez (I)

Neurovascular Research Laboratory, Institute of Biomedicine of Seville-IBIS, Sevilla, Spain.

Anna Penalba (A)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research (VHIR)-Universitat Autónoma de Barcelona, Barcelona, Spain.

Alejandro Bustamante (A)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research (VHIR)-Universitat Autónoma de Barcelona, Barcelona, Spain; Stroke Unit, Hospital Universitari Germans Trias I Pujol, Badalona, Barcelona, Spain.

Marcel Lamana-Vallverdú (M)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research (VHIR)-Universitat Autónoma de Barcelona, Barcelona, Spain.

Fernando Mancha (F)

Neurovascular Research Laboratory, Institute of Biomedicine of Seville-IBIS, Sevilla, Spain.

Rafael F Ocete (RF)

Department of Radiology, Hospital Universitario Virgen del Rocío, Seville, Spain.

Pilar Piñero (P)

Department of Radiology, Hospital Universitario Virgen del Rocío, Seville, Spain.

Alejandro Galvao-Carmona (A)

Department of Psychology, Universidad Loyola Andalucía, Seville, Spain.

Marta Gómez-Herranz (M)

NEURORHB, Servicio de Neurorrehabilitación de Hospitales Vithas, Seville, Spain.

Soledad Pérez-Sánchez (S)

Neurovascular Research Laboratory, Institute of Biomedicine of Seville-IBIS, Sevilla, Spain; Stroke Unit, Hospital Universitario Virgen Macarena, Sevilla, Spain.

Francisco Moniche (F)

Stroke Unit, University Hospital Virgen del Rocio, Seville, Spain.

Alejandro González (A)

Department of Radiology, Hospital Universitario Virgen del Rocío, Seville, Spain.

Joan Montaner (J)

Neurovascular Research Laboratory, Vall d'Hebron Institute of Research (VHIR)-Universitat Autónoma de Barcelona, Barcelona, Spain; Neurovascular Research Laboratory, Institute of Biomedicine of Seville-IBIS, Sevilla, Spain. Electronic address: jmontaner-ibis@us.es.

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