Baseline Serum Biomarkers of Inflammation and Subsequent Visit-to-Visit Blood Pressure Variability: A Post Hoc Analysis of MESA.

6 C Ethnic Study of Atherosclerosis II Multi angiotensin antiplasmin complex blood pressure blood pressure variability hypertension interleukin plasmin reactive protein residual standard deviation

Journal

American journal of hypertension
ISSN: 1941-7225
Titre abrégé: Am J Hypertens
Pays: United States
ID NLM: 8803676

Informations de publication

Date de publication:
24 02 2023
Historique:
received: 05 08 2022
accepted: 28 10 2022
pubmed: 1 11 2022
medline: 3 3 2023
entrez: 31 10 2022
Statut: ppublish

Résumé

Higher blood pressure variability (BPV) is associated with the development of major vascular diseases, independent of mean blood pressure. However, despite data indicating that serum inflammatory markers are linked to hypertension, the association between serum inflammatory markers and BPV has not been studied in humans. This is a post hoc analysis of the Multi-Ethnic Study of Atherosclerosis (MESA) study. The study exposure was tertiles of serum level of interleukin-6 (IL-6), C-reactive protein (CRP), d-dimer, plasmin-antiplasmin complex (PAP), fibrinogen antigen, and calibrated Factor VIII (%) at the baseline study visit. The primary outcome was visit-to-visit BPV measured as the residual standard deviation (rSD) of at least 4 study visits (2000-2018). Two logistic regression models were fit to the top tertile of rSD during follow-up: in Model 1, we adjusted for age, sex, and hypertension, and in Model 2, for patient age categories, sex, race/ethnicity, education, hypertension, diabetes, smoking, drinking, body mass index, lipid-lowering medication, and mean systolic blood pressure. Our analysis included 5,483 patients, with a mean (SD) age of 61.4 (10.0) years, 52.9% female, and 40.7% White. In unadjusted analyses, all markers of inflammation were associated with higher BPV, but after adjustment, only IL-6 retained significance (P < 0.001). The odds ratio for the highest tertile of BPV and IL-6 was 1.49 (95% confidence interval [CI] 1.28-1.74, P < 0.001). Baseline serum IL-6 was associated with increased subsequent BPV in a large multiracial cohort. Further investigation is needed to better understand the relationship between chronic inflammation and BPV.

Sections du résumé

BACKGROUND
Higher blood pressure variability (BPV) is associated with the development of major vascular diseases, independent of mean blood pressure. However, despite data indicating that serum inflammatory markers are linked to hypertension, the association between serum inflammatory markers and BPV has not been studied in humans.
METHODS
This is a post hoc analysis of the Multi-Ethnic Study of Atherosclerosis (MESA) study. The study exposure was tertiles of serum level of interleukin-6 (IL-6), C-reactive protein (CRP), d-dimer, plasmin-antiplasmin complex (PAP), fibrinogen antigen, and calibrated Factor VIII (%) at the baseline study visit. The primary outcome was visit-to-visit BPV measured as the residual standard deviation (rSD) of at least 4 study visits (2000-2018). Two logistic regression models were fit to the top tertile of rSD during follow-up: in Model 1, we adjusted for age, sex, and hypertension, and in Model 2, for patient age categories, sex, race/ethnicity, education, hypertension, diabetes, smoking, drinking, body mass index, lipid-lowering medication, and mean systolic blood pressure.
RESULTS
Our analysis included 5,483 patients, with a mean (SD) age of 61.4 (10.0) years, 52.9% female, and 40.7% White. In unadjusted analyses, all markers of inflammation were associated with higher BPV, but after adjustment, only IL-6 retained significance (P < 0.001). The odds ratio for the highest tertile of BPV and IL-6 was 1.49 (95% confidence interval [CI] 1.28-1.74, P < 0.001).
CONCLUSIONS
Baseline serum IL-6 was associated with increased subsequent BPV in a large multiracial cohort. Further investigation is needed to better understand the relationship between chronic inflammation and BPV.

Identifiants

pubmed: 36315490
pii: 6783088
doi: 10.1093/ajh/hpac122
doi:

Substances chimiques

Interleukin-6 0
Biomarkers 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

144-147

Informations de copyright

© The Author(s) 2022. Published by Oxford University Press on behalf of American Journal of Hypertension, Ltd. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Auteurs

Ka-Ho Wong (KH)

Department of Neurology, University of Utah, Salt Lake City, Utah, USA.

Varsha Muddasani (V)

Department of Neurology, Einstein Healthcare Network, Philadelphia, Pennsylvania, USA.

Cecilia Peterson (C)

Department of Neurology, University of Utah, Salt Lake City, Utah, USA.

Nazanin Sheibani (N)

Department of Neurology, Tufts Medical Center, Boston, Massachusetts, USA.

Cameron Arkin (C)

Department of Neurology, University of Utah, Salt Lake City, Utah, USA.

Irene Cheong (I)

Department of Internal Medicine, University of Utah, Salt Lake City, Utah, USA.

Jennifer J Majersik (JJ)

Department of Neurology, University of Utah, Salt Lake City, Utah, USA.

Alessandro Biffi (A)

Department of Neurology, Massachusetts General Hospital, Boston, Massachusetts, USA.

Nils Petersen (N)

Department of Neurology, Yale University, New Haven, Connecticut, USA.

Guido J Falcone (GJ)

Department of Neurology, Yale University, New Haven, Connecticut, USA.

Lauren H Sansing (LH)

Department of Neurology, Yale University, New Haven, Connecticut, USA.

Adam H de Havenon (AH)

Department of Neurology, Yale University, New Haven, Connecticut, USA.

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