Macroglossia in rapidly progressive inclusion body myositis.

Sjögren's syndrome inclusion body myositis macroglossia rapid progression

Journal

Neuropathology : official journal of the Japanese Society of Neuropathology
ISSN: 1440-1789
Titre abrégé: Neuropathology
Pays: Australia
ID NLM: 9606526

Informations de publication

Date de publication:
Jun 2023
Historique:
revised: 13 10 2022
received: 20 06 2022
accepted: 18 10 2022
medline: 5 6 2023
pubmed: 10 11 2022
entrez: 9 11 2022
Statut: ppublish

Résumé

Inclusion body myositis (IBM) is a refractory muscle disease characterized by inflammatory and degenerative features in myofibers. Macroglossia is common in systemic amyloid light chain amyloidosis; however, no reports have been published on patients with IBM. We encountered a female patient with clinicopathologically defined IBM who exhibited relatively rapid progression of dysphagia, gait disturbance, and macroglossia. Muscle biopsy demonstrated endomysial mononuclear inflammatory infiltrates, fiber necrosis and regeneration with rimmed vacuoles, and sarcoplasmic inclusions of p62. Tongue biopsy demonstrated fiber degeneration with fatty replacement and fibrosis, nonnecrotic fibers surrounded and invaded by mononuclear cells, and sarcoplasmic dotlike inclusions of p62. Based on the parotid gland, lip, and muscle biopsy, she was diagnosed as having IBM with Sjögren's syndrome. She was treated with steroid pulse and intravenous immunoglobulin therapy followed by oral administration of prednisolone, which resulted in temporary clinical improvement. Macroglossia might be an indicator of immunotherapy effectiveness.

Identifiants

pubmed: 36349419
doi: 10.1111/neup.12879
doi:

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

252-256

Subventions

Organisme : Japan Society for the Promotion of Science
ID : Grant-in-Aid for Scientific Research (16K09674)
Organisme : Ministry of Health, Labour and Welfare
ID : Grant-in-Aid for Research on Intractable Diseases
Organisme : Kumamoto University

Informations de copyright

© 2022 Japanese Society of Neuropathology.

Références

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Auteurs

Yoshimune Yamasaki (Y)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Akihiro Mukaino (A)

Department of Molecular Neurology and Therapeutics, Kumamoto University Hospital, Kumamoto, Japan.

Satoshi Yamashita (S)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Yousuke Takeuchi (Y)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Nozomu Tawara (N)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Ryoji Yoshida (R)

Department of Oral and Maxillofacial Surgery, Faculty of Sciences, Kumamoto University Hospital, Kumamoto, Japan.

Yumi Honda (Y)

Department of Diagnostic Pathology, Kumamoto University Hospital, Kumamoto, Japan.

Taro Yamashita (T)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Asako Kakimoto (A)

Department of Neurology, National Hospital Organization Kumamoto Saishun Medical Center, Koshi, Kumamoto, Japan.

Hidetsugu Ueyama (H)

Department of Neurology, National Hospital Organization Kumamoto Saishun Medical Center, Koshi, Kumamoto, Japan.

Yukio Ando (Y)

Department of Neurology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

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