Constructing a novel competing Endogenous RNAs network based on NR3C1 and X-linked inhibitor of apoptosis protein genes reveals potential prognostic biomarkers in colorectal cancer.

Colorectal cancer KCNQ1OT1 NR3C1 X-linked inhibitor of apoptosis protein competing endogenous RNA miR-421

Journal

Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences
ISSN: 1735-1995
Titre abrégé: J Res Med Sci
Pays: India
ID NLM: 101235599

Informations de publication

Date de publication:
2022
Historique:
received: 06 10 2021
revised: 18 04 2022
accepted: 09 05 2022
entrez: 10 11 2022
pubmed: 11 11 2022
medline: 11 11 2022
Statut: epublish

Résumé

Long noncoding RNAs (lncRNAs) have been recognized as the main modulatory molecules in various cancers and perform as competing endogenous RNAs (ceRNAs). The nuclear hormone receptor superfamily of ligand-activated transcription factors (NR3C1) regulates numerous proliferative and metabolic processes such as tumorigenesis and metabolic diseases. Furthermore, X-linked inhibitor of apoptosis protein (XIAP) belongs to a family of the inhibitors of apoptosis proteins, is located downstream of the glucocorticoid receptor (GR or NR3C1) pathway, and cooperates with GR to suppress apoptosis. However, the underlying mechanisms of NR3C1 and XIAP in colorectal cancer (CRC) remain mainly unclear. This research aims to clarify the potential RNA biomarkers and to construct a novel ceRNA network in CRC. Multistep bioinformatics methods such as Lnc2cancer and miRDB databases were applied to identify candidate lncRNAs and miRNAs. The interaction energy between lncRNAs, NR3C1, and XIAP genes was analyzed by the LncRRIsearch database. Plus, microRNAs and lncRNA were evaluated via the Diana tools database to select microRNAs with the most binding scores. Quantitative reverse transcription-polymerase chain reaction (QRT-PCR) was applied to verify RNA molecules' expression levels and their association with the clinicopathological factors in 30 CRC tissues compared to 30 adjacent tissues. QRT-PCR showed upregulation of KCNQ1OT1, NR3C1, and XIAP and downregulation of miR-421. The ceRNA network was constructed with 17 lncRNAs, 2 mRNAs, and 42 miRNAs. Thus, we explained the potential interactions between KCNQ1OT1 and miR-421 with NR3C1 and XIAP genes. Our study represents potential prognostic biomarkers and a new ceRNA network for further study in CRC.

Sections du résumé

Background UNASSIGNED
Long noncoding RNAs (lncRNAs) have been recognized as the main modulatory molecules in various cancers and perform as competing endogenous RNAs (ceRNAs). The nuclear hormone receptor superfamily of ligand-activated transcription factors (NR3C1) regulates numerous proliferative and metabolic processes such as tumorigenesis and metabolic diseases. Furthermore, X-linked inhibitor of apoptosis protein (XIAP) belongs to a family of the inhibitors of apoptosis proteins, is located downstream of the glucocorticoid receptor (GR or NR3C1) pathway, and cooperates with GR to suppress apoptosis. However, the underlying mechanisms of NR3C1 and XIAP in colorectal cancer (CRC) remain mainly unclear. This research aims to clarify the potential RNA biomarkers and to construct a novel ceRNA network in CRC.
Materials and Methods UNASSIGNED
Multistep bioinformatics methods such as Lnc2cancer and miRDB databases were applied to identify candidate lncRNAs and miRNAs. The interaction energy between lncRNAs, NR3C1, and XIAP genes was analyzed by the LncRRIsearch database. Plus, microRNAs and lncRNA were evaluated via the Diana tools database to select microRNAs with the most binding scores. Quantitative reverse transcription-polymerase chain reaction (QRT-PCR) was applied to verify RNA molecules' expression levels and their association with the clinicopathological factors in 30 CRC tissues compared to 30 adjacent tissues.
Results UNASSIGNED
QRT-PCR showed upregulation of KCNQ1OT1, NR3C1, and XIAP and downregulation of miR-421. The ceRNA network was constructed with 17 lncRNAs, 2 mRNAs, and 42 miRNAs. Thus, we explained the potential interactions between KCNQ1OT1 and miR-421 with NR3C1 and XIAP genes.
Conclusion UNASSIGNED
Our study represents potential prognostic biomarkers and a new ceRNA network for further study in CRC.

Identifiants

pubmed: 36353350
doi: 10.4103/jrms.jrms_889_21
pii: JRMS-27-71
pmc: PMC9639721
doi:

Types de publication

Journal Article

Langues

eng

Pagination

71

Informations de copyright

Copyright: © 2022 Journal of Research in Medical Sciences.

Déclaration de conflit d'intérêts

There are no conflicts of interest.

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Auteurs

Mohammad Abdolvand (M)

Molecular and Genetics Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.
Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Science, Isfahan, Iran.

Minoosh Sadeghi (M)

Department of Genetics, Faculty of Sciences, Shahrekord University, Shahrekord, Iran.

Mohammad Hassan Emami (MH)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Alireza Fahim (A)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Hojjatolah Rahimi (H)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Elham Amjadi (E)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Abdolmehdi Baghaei (A)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Shirin Abdolvand (S)

Department of Genetics, Faculty of Sciences, Islamic Azad University, Shahrekord Branch, Shahrekord, Iran.

Fatemeh Maghool (F)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Sara Feizbakhshan (S)

Molecular and Genetics Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.

Sharareh Salmanizadeh (S)

Molecular and Genetics Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.

Ehsan Heidari (E)

Young Researchers and Elite Club, Shahrekord Branch, Islamic Azad University, Shahrekord, Iran.
Blood Transfusion Research Center, High Institute for Research and Education in Transfusion Medicine, Tehran, Iran.

Mohammad Chehelgerdi (M)

Young Researchers and Elite Club, Shahrekord Branch, Islamic Azad University, Shahrekord, Iran.

Mahsa Khodadoostan (M)

Department of Gastroenterology and Hepatology, AlZahra Hospital, Isfahan University of Medical Sciences, Isfahan, Iran.

Maryam Ebrahim (M)

Department of Gastroenterology and Hepatology, AlZahra Hospital, Isfahan University of Medical Sciences, Isfahan, Iran.

Faeze Ahmadi Beni (FA)

Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.
Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Science, Isfahan, Iran.

Mohammad Kazemi (M)

Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Science, Isfahan, Iran.

Simin Hemati (S)

Department of Radiooncology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.

Farinaz Khosravian (F)

Molecular and Genetics Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.
Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Science, Isfahan, Iran.

Hassan Rahimi (H)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Alireza Samadian (A)

Poursina Hakim Digestive Diseases Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Mansoor Salehi (M)

Molecular and Genetics Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.
Medical Genetics Research Center of Genome, Isfahan University of Medical Sciences, Isfahan, Iran.
Department of Genetics and Molecular Biology, School of Medicine, Isfahan University of Medical Science, Isfahan, Iran.

Classifications MeSH