Responses of complement C1q/tumor necrosis factor-related proteins to acute aerobic exercise.


Journal

Cytokine
ISSN: 1096-0023
Titre abrégé: Cytokine
Pays: England
ID NLM: 9005353

Informations de publication

Date de publication:
01 2023
Historique:
received: 23 03 2022
revised: 30 08 2022
accepted: 25 10 2022
pubmed: 11 11 2022
medline: 29 11 2022
entrez: 10 11 2022
Statut: ppublish

Résumé

Aerobic exercise is an effective therapeutic strategy to manage metabolic disorders. However, the mechanisms of aerobic exercise-induced improvements in metabolic diseases are not completely understood. Complement C1q/tumor necrosis factor-related protein (CTRP) 1, CTRP3, CTRP5, and CTRP9 have important roles in improving metabolic disorders via the adenosine monophosphate-activated protein kinase signaling pathway. In this study, we investigated the effects of acute aerobic exercise on circulating CTRP1, CTRP3, CTRP5, and CTRP9 levels in human participants. Eight healthy males with an age of 20.4 ± 0.2 years, height 173.1 ± 1.7 cm, body mass 68.0 ± 1.8 kg, body mass index 22.7 ± 0.7 kg/m

Identifiants

pubmed: 36356496
pii: S1043-4666(22)00292-7
doi: 10.1016/j.cyto.2022.156083
pii:
doi:

Substances chimiques

Adiponectin 0
Complement C1q 80295-33-6
Insulin 0
Tumor Necrosis Factor-alpha 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

156083

Informations de copyright

Copyright © 2022. Published by Elsevier Ltd.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Michihiro Kon (M)

Department of Health Care and Sports, Faculty of Human Life Design, Toyo University, 1-7-11, Akabanedai, Kita-ku, Tokyo 115-0053, Japan. Electronic address: kon@toyo.jp.

Yuko Tanimura (Y)

Department of Sports Research, Japan Institute of Sports Sciences, 3-15-1 Nishigaoka, Kita-ku, Tokyo 115-0056, Japan.

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Classifications MeSH