Exercise electrocardiogram for risk-based screening of severe residual coronary lesion in children after coronary surgery.


Journal

Archives of cardiovascular diseases
ISSN: 1875-2128
Titre abrégé: Arch Cardiovasc Dis
Pays: Netherlands
ID NLM: 101465655

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 03 04 2022
revised: 22 09 2022
accepted: 03 10 2022
pubmed: 14 11 2022
medline: 15 12 2022
entrez: 13 11 2022
Statut: ppublish

Résumé

Residual severe coronary artery (CA) lesion (SCL) in children after cardiac surgery involving the CA is a major concern. To evaluate the value of exercise electrocardiogram (eECG) for risk-based screening of SCL. We analysed 135 maximal eECG from 115 children (mean age 13.6±3.7 years) who underwent concomitant CA imaging. SCL was defined as a stenosis exceeding 50%. Underlying congenital heart diseases were transposition of the great arteries (TGA) (n = 116), CA pathway anomaly (n = 13) and left CA from the pulmonary artery (n = 6). Eleven SCLs were identified in 10 patients, of which 3 had a known untreated non-severe lesion and 4 had no lesions on previous imaging. In multivariable analysis, risks markers for SCL were effort chest pain (OR: 4.72, 95% CI: 1.23-18.17; P=0.024), intramural pathway (OR: 4.37, 95% CI: 1.14-16.81; P=0.032). Yacoubs C-type CA was added as a risk marker for patients with TGA (P=0.0009). All patients with SCL had a positive eECG (sensitivity: 100%, 95% CI: 72-100). Specificity was 81% (95% CI: 73-87). In the low-risk group (0 risk markers), 3/95 patients had SCL (3%), and the post-test probability of SCL with positive eECG (PPr+) was 15% (95% CI: 8-21). In the high-risk group (≥1 risk marker) comprising 8/40 SCLs (20%), PPr+ was 53% (95% CI: 35-67). Most SCL tended to develop gradually, years after surgery. Provided it is near maximal, a negative eECG appears sufficient to exclude SCL. In the high-risk group, PPr+ exceeded 50%.

Sections du résumé

BACKGROUND BACKGROUND
Residual severe coronary artery (CA) lesion (SCL) in children after cardiac surgery involving the CA is a major concern.
AIM OBJECTIVE
To evaluate the value of exercise electrocardiogram (eECG) for risk-based screening of SCL.
METHODS METHODS
We analysed 135 maximal eECG from 115 children (mean age 13.6±3.7 years) who underwent concomitant CA imaging. SCL was defined as a stenosis exceeding 50%.
RESULTS RESULTS
Underlying congenital heart diseases were transposition of the great arteries (TGA) (n = 116), CA pathway anomaly (n = 13) and left CA from the pulmonary artery (n = 6). Eleven SCLs were identified in 10 patients, of which 3 had a known untreated non-severe lesion and 4 had no lesions on previous imaging. In multivariable analysis, risks markers for SCL were effort chest pain (OR: 4.72, 95% CI: 1.23-18.17; P=0.024), intramural pathway (OR: 4.37, 95% CI: 1.14-16.81; P=0.032). Yacoubs C-type CA was added as a risk marker for patients with TGA (P=0.0009). All patients with SCL had a positive eECG (sensitivity: 100%, 95% CI: 72-100). Specificity was 81% (95% CI: 73-87). In the low-risk group (0 risk markers), 3/95 patients had SCL (3%), and the post-test probability of SCL with positive eECG (PPr+) was 15% (95% CI: 8-21). In the high-risk group (≥1 risk marker) comprising 8/40 SCLs (20%), PPr+ was 53% (95% CI: 35-67).
CONCLUSIONS CONCLUSIONS
Most SCL tended to develop gradually, years after surgery. Provided it is near maximal, a negative eECG appears sufficient to exclude SCL. In the high-risk group, PPr+ exceeded 50%.

Identifiants

pubmed: 36372663
pii: S1875-2136(22)00201-7
doi: 10.1016/j.acvd.2022.10.001
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

656-663

Informations de copyright

Copyright © 2022 Elsevier Masson SAS. All rights reserved.

Auteurs

Johanne Auriau (J)

M3C-Necker-Enfants malades, AP-HP Paris, 75015 Paris, France.

Zahra Belhadjer (Z)

M3C-Necker-Enfants malades, AP-HP Paris, 75015 Paris, France.

Elena Panaioli (E)

M3C-Necker-Enfants malades, Radiology Department, hôpital universitaire Necker enfants malades, AP-HP, 75015 Paris, France.

Neil Derridj (N)

M3C-Necker-Enfants malades, AP-HP Paris, 75015 Paris, France.

Jean-Philippe Jais (JP)

Biostatistics Unit, hôpital universitaire Necker-Enfants malades, AP-HP, Inserm U1163, Institut Imagine, Laboratory of Human Genetics of Infectious Diseases, Necker Branch, University of Paris, 75015 Paris, France.

Regis Gaudin (R)

M3C-Necker-Enfants malades, AP-HP Paris, 75015 Paris, France.

Francesca Raimondi (F)

M3C-Necker-Enfants malades, AP-HP, University of Paris, 75015 Paris, France.

Damien Bonnet (D)

M3C-Necker-Enfants malades, AP-HP, University of Paris, 75015 Paris, France.

Antoine Legendre (A)

M3C-Necker-Enfants malades, AP-HP Paris, 75015 Paris, France. Electronic address: antoine.legendre@aphp.fr.

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Classifications MeSH