The Prognostic Value of Lung Injury and Fibrosis Markers, KL-6, TGF-β1, FGF-2 in COVID-19 Patients.

Coronavirus disease 2019 Krebs von den Lungen-6 biomarker computed tomography fibroblast growth factor-2 transforming growth factor-beta1

Journal

Biomarker insights
ISSN: 1177-2719
Titre abrégé: Biomark Insights
Pays: United States
ID NLM: 101288638

Informations de publication

Date de publication:
2022
Historique:
received: 07 07 2022
accepted: 11 10 2022
entrez: 16 11 2022
pubmed: 17 11 2022
medline: 17 11 2022
Statut: epublish

Résumé

Biomarkers of lung injury and interstitial fibrosis give insight about the extent of involvement and prognosis in well-known interstitial lung diseases (ILD). Serum Krebs von den Lungen-6 (KL-6) reflects direct alveolar injury and, transforming growth factor-beta1 (TGF-β1) and fibroblast growth factor-2 (FGF-2) are principal mediators of fibrosis in ILD and in almost all fibrotic diseases. In this sense, we aimed to assess associations of these biomarkers with traditional inflammatory markers and clinical course of COVID-19. Patients with COVID-19 who had confirmed diagnosis with SARS-CoV-2 nucleic acid RT-PCR were enrolled and followed up prospectively with a standardized approach one month after diagnosis. Patients were divided into severe and non-severe groups according to National Institutes of Health criteria. Outcome was assessed for the requirement of intensive care unit (ICU) admission, long term respiratory support and death. Blood samples were collected at enrollment and serum levels of KL-6, TGF-β1, FGF-2 were determined by ELISA. Association between these markers with other prognostic markers and prognosis were analyzed. Overall 31 severe and 28 non-severe COVID-19 patients were enrolled and were compared with healthy control subjects (n = 30). Serum KL-6 levels in COVID-19 patients were significantly higher (median [IQR]; 11.54 [4.86] vs 8.54 [3.98] ng/mL, Serum KL-6 levels were significantly elevated at the diagnosis of COVID-19 and correlated well with the other traditional prognostic inflammatory markers. Serum levels of principal fibrosis mediators, TGF-β1, FGF-2, were not elevated at diagnosis of COVID-19, therefore did not help to anticipate long term prognosis.

Sections du résumé

Background UNASSIGNED
Biomarkers of lung injury and interstitial fibrosis give insight about the extent of involvement and prognosis in well-known interstitial lung diseases (ILD). Serum Krebs von den Lungen-6 (KL-6) reflects direct alveolar injury and, transforming growth factor-beta1 (TGF-β1) and fibroblast growth factor-2 (FGF-2) are principal mediators of fibrosis in ILD and in almost all fibrotic diseases. In this sense, we aimed to assess associations of these biomarkers with traditional inflammatory markers and clinical course of COVID-19.
Methods UNASSIGNED
Patients with COVID-19 who had confirmed diagnosis with SARS-CoV-2 nucleic acid RT-PCR were enrolled and followed up prospectively with a standardized approach one month after diagnosis. Patients were divided into severe and non-severe groups according to National Institutes of Health criteria. Outcome was assessed for the requirement of intensive care unit (ICU) admission, long term respiratory support and death. Blood samples were collected at enrollment and serum levels of KL-6, TGF-β1, FGF-2 were determined by ELISA. Association between these markers with other prognostic markers and prognosis were analyzed.
Results UNASSIGNED
Overall 31 severe and 28 non-severe COVID-19 patients were enrolled and were compared with healthy control subjects (n = 30). Serum KL-6 levels in COVID-19 patients were significantly higher (median [IQR]; 11.54 [4.86] vs 8.54 [3.98] ng/mL,
Conclusions UNASSIGNED
Serum KL-6 levels were significantly elevated at the diagnosis of COVID-19 and correlated well with the other traditional prognostic inflammatory markers. Serum levels of principal fibrosis mediators, TGF-β1, FGF-2, were not elevated at diagnosis of COVID-19, therefore did not help to anticipate long term prognosis.

Identifiants

pubmed: 36380740
doi: 10.1177/11772719221135443
pii: 10.1177_11772719221135443
pmc: PMC9643117
doi:

Types de publication

Journal Article

Langues

eng

Pagination

11772719221135443

Informations de copyright

© The Author(s) 2022.

Déclaration de conflit d'intérêts

The author(s) declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article.

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Auteurs

Hazan Karadeniz (H)

Division of Rheumatology, Department of Internal Medicine, Gazi University Faculty of Medicine, Ankara, Turkey.

Aslıhan Avanoğlu Güler (A)

Division of Rheumatology, Department of Internal Medicine, Gazi University Faculty of Medicine, Ankara, Turkey.

Hasan Selçuk Özger (HS)

Department of Infectious Disease, Gazi University Faculty of Medicine, Ankara, Turkey.

Pınar Aysert Yıldız (PA)

Department of Infectious Disease, Gazi University Faculty of Medicine, Ankara, Turkey.

Gonca Erbaş (G)

Department of Radiology, Gazi University Faculty of Medicine, Ankara, Turkey.

Gülendam Bozdayı (G)

Department of Medical Microbiology, Gazi University Faculty of Medicine, Ankara, Turkey.

Tuba Deveci Bulut (T)

Department of Biochemistry, Gazi University Faculty of Medicine, Ankara, Turkey.

Özlem Gülbahar (Ö)

Department of Biochemistry, Gazi University Faculty of Medicine, Ankara, Turkey.

Dilek Yapar (D)

Department of Public Health and Biostatistics Faculty of Medicine, Gazi University, Ankara, Turkey.

Hamit Küçük (H)

Division of Rheumatology, Department of Internal Medicine, Gazi University Faculty of Medicine, Ankara, Turkey.

Mehmet Akif Öztürk (MA)

Division of Rheumatology, Department of Internal Medicine, Gazi University Faculty of Medicine, Ankara, Turkey.

Abdurrahman Tufan (A)

Division of Rheumatology, Department of Internal Medicine, Gazi University Faculty of Medicine, Ankara, Turkey.

Classifications MeSH