Morphine upregulates Toll-like receptor 4 expression and promotes melanomas in mice.


Journal

Immunopharmacology and immunotoxicology
ISSN: 1532-2513
Titre abrégé: Immunopharmacol Immunotoxicol
Pays: England
ID NLM: 8800150

Informations de publication

Date de publication:
Jun 2023
Historique:
medline: 30 5 2023
pubmed: 17 11 2022
entrez: 16 11 2022
Statut: ppublish

Résumé

Morphine and other opioids are used to manage cancer-related pain; however, the role of these drugs in cancer progression remains controversial. Emerging evidence indicates that morphine can activate Toll-like receptor 4 (TLR4) and its signaling pathways, by the way the activation and expression of TLR4 can promote melanoma. In this study, we investigated the effects of morphine on the expression of TLR4 and promotion of melanoma in mice. Mice melanoma cells (B16F10) were cultured with morphine (0.1, 1 and 10 µM) for 24 h. In the other experiment, cells were treated with morphine with or without TLR4 agonist (LPS) or antagonist (TAK-242). In Chronic, acute, and escalating doses of morphine increased tumor. Morphine increased the expression of Morphine increases the progression of melanoma cancer and may be related to the increased expression of TLR4. Our results suggest that morphine should be used with caution in patients with melanoma.HighlightsMorphine increases the expression of TLR4 in melanoma.Morphine increases melanoma progression.These effects are mostly observed with chronic and escalating morphine administration.

Sections du résumé

BACKGROUND UNASSIGNED
Morphine and other opioids are used to manage cancer-related pain; however, the role of these drugs in cancer progression remains controversial. Emerging evidence indicates that morphine can activate Toll-like receptor 4 (TLR4) and its signaling pathways, by the way the activation and expression of TLR4 can promote melanoma. In this study, we investigated the effects of morphine on the expression of TLR4 and promotion of melanoma in mice.
METHODS UNASSIGNED
Mice melanoma cells (B16F10) were cultured with morphine (0.1, 1 and 10 µM) for 24 h. In the other experiment, cells were treated with morphine with or without TLR4 agonist (LPS) or antagonist (TAK-242). In
RESULTS UNASSIGNED
Chronic, acute, and escalating doses of morphine increased tumor. Morphine increased the expression of
CONCLUSION UNASSIGNED
Morphine increases the progression of melanoma cancer and may be related to the increased expression of TLR4. Our results suggest that morphine should be used with caution in patients with melanoma.HighlightsMorphine increases the expression of TLR4 in melanoma.Morphine increases melanoma progression.These effects are mostly observed with chronic and escalating morphine administration.

Identifiants

pubmed: 36382834
doi: 10.1080/08923973.2022.2145967
doi:

Substances chimiques

Lipopolysaccharides 0
Morphine 76I7G6D29C
NF-kappa B 0
Toll-Like Receptor 4 0
Tlr4 protein, mouse 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

347-354

Auteurs

Golnaz Vaseghi (G)

Isfahan Cardiovascular Research Center, Cardiovascular Research Institute, Isfahan University of Medical Sciences, Isfahan, Iran.

Ahmad Ghasemi (A)

Department of Physiology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.

Ismail Laher (I)

Department of Anesthesiology, Pharmacology and Therapeutics, University of British Columbia, Vancouver, British Columbia, Canada.

HojjatAllah Alaei (H)

Department of Physiology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.

Nasim Dana (N)

Applied Physiology Research Center, Cardiovascular Research Institute, Isfahan University of Medical Sciences, Isfahan, Iran.

Hajar Naji Esfahani (H)

Applied Physiology Research Center, Cardiovascular Research Institute, Isfahan University of Medical Sciences, Isfahan, Iran.

Shaghayegh Haghjooy Javanmard (SH)

Department of Physiology, School of Medicine, Isfahan University of Medical Sciences, Isfahan, Iran.
Applied Physiology Research Center, Cardiovascular Research Institute, Isfahan University of Medical Sciences, Isfahan, Iran.

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Classifications MeSH