Borderzone infarction and recurrent stroke in intracranial atherosclerosis.


Journal

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association
ISSN: 1532-8511
Titre abrégé: J Stroke Cerebrovasc Dis
Pays: United States
ID NLM: 9111633

Informations de publication

Date de publication:
Jan 2023
Historique:
received: 15 08 2022
revised: 10 10 2022
accepted: 10 11 2022
pubmed: 20 11 2022
medline: 11 1 2023
entrez: 19 11 2022
Statut: ppublish

Résumé

Intracranial stenosis (ICAS) is a common cause of stroke worldwide and patients with symptomatic ICAS exhibit a high rate of recurrence, particularly in the early period after the initial event. In this study, we aimed to study the association between borderzone infarct and recurrent ischemic stroke in patients hospitalized with symptomatic ICAS. This is a retrospective single center study that included patients hospitalized with acute ischemic stroke in the setting of intracranial stenosis (50% or more and an acute ischemic stroke in the territory supplied by the stenosed artery) over a 32-month period. We excluded patients who did not receive a brain MRI or did not have an infarct on brain imaging. The primary predictor is infarct pattern (any borderzone vs. no borderzone infarct) and the primary outcome was recurrent cerebrovascular events (RCVE) within 90 days. We used unadjusted, and age and sex adjusted logistic regression models to determine associations between infarct pattern and RCVE at 90-days. Among 99 patients who met the inclusion criteria (4 tandem), the mean age was 70.1 ± 11.2 years and 41.4% were women; 43 had borderzone infarcts and 19 had RCVE. In adjusted binary logistic regression analysis, borderzone infarct was associated with increased risk of RCVE (adjusted OR 4.00 95% CI 1.33-11.99, p=0.013). The association between borderzone infarction and RCVE was not different among anterior circulation ICAD (adjusted HR 2.85 95% CI 0.64-12.76, p=0.172) vs. posterior circulation ICAD (adjusted HR 6.69 95% CI 1.06-42.11, p=0.043), p-value for interaction = 0.592. In real world post-SAMMPRIS medically treated patients with ICAD, the borderzone infarct pattern was associated with 90-day RCVE. Borderzone infarcts are likely a surrogate marker of impaired distal blood flow, highlighting the importance of targeting stroke mechanisms and developing alternative treatment strategies for high-risk cohorts.

Sections du résumé

BACKGROUND BACKGROUND
Intracranial stenosis (ICAS) is a common cause of stroke worldwide and patients with symptomatic ICAS exhibit a high rate of recurrence, particularly in the early period after the initial event. In this study, we aimed to study the association between borderzone infarct and recurrent ischemic stroke in patients hospitalized with symptomatic ICAS.
METHODS METHODS
This is a retrospective single center study that included patients hospitalized with acute ischemic stroke in the setting of intracranial stenosis (50% or more and an acute ischemic stroke in the territory supplied by the stenosed artery) over a 32-month period. We excluded patients who did not receive a brain MRI or did not have an infarct on brain imaging. The primary predictor is infarct pattern (any borderzone vs. no borderzone infarct) and the primary outcome was recurrent cerebrovascular events (RCVE) within 90 days. We used unadjusted, and age and sex adjusted logistic regression models to determine associations between infarct pattern and RCVE at 90-days.
RESULTS RESULTS
Among 99 patients who met the inclusion criteria (4 tandem), the mean age was 70.1 ± 11.2 years and 41.4% were women; 43 had borderzone infarcts and 19 had RCVE. In adjusted binary logistic regression analysis, borderzone infarct was associated with increased risk of RCVE (adjusted OR 4.00 95% CI 1.33-11.99, p=0.013). The association between borderzone infarction and RCVE was not different among anterior circulation ICAD (adjusted HR 2.85 95% CI 0.64-12.76, p=0.172) vs. posterior circulation ICAD (adjusted HR 6.69 95% CI 1.06-42.11, p=0.043), p-value for interaction = 0.592.
CONCLUSION CONCLUSIONS
In real world post-SAMMPRIS medically treated patients with ICAD, the borderzone infarct pattern was associated with 90-day RCVE. Borderzone infarcts are likely a surrogate marker of impaired distal blood flow, highlighting the importance of targeting stroke mechanisms and developing alternative treatment strategies for high-risk cohorts.

Identifiants

pubmed: 36402094
pii: S1052-3057(22)00589-4
doi: 10.1016/j.jstrokecerebrovasdis.2022.106897
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

106897

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Auteurs

Alexandra Kvernland (A)

Department of Neurology, New York Langone Health, New York, NY, USA. Electronic address: Alexandra.Kvernland@nyulangone.org.

Jose Torres (J)

Department of Neurology, New York Langone Health, New York, NY, USA. Electronic address: Jose.Torres2@nyulangone.org.

Eytan Raz (E)

Department of Radiology, New York Langone Health, New York, NY, USA. Electronic address: Eytan.Raz@nyulangone.org.

Erez Nossek (E)

Department of Neurosurgery, New York Langone Health, New York, NY, USA. Electronic address: Erez.Nossek@nyulangone.org.

Adam de Havenon (A)

Department of Neurology, Yale University, New Haven, CT, USA. Electronic address: adam.dehavenon@yale.edu.

Mulugeta Gebregziabher (M)

Department of Public Health Sciences, Medical University of South Carolina, Charleston, SC, USA. Electronic address: gebregz@musc.edu.

Pooja Khatri (P)

Department of Neurology, University of Cincinnati, Cincinnati, OH, USA. Electronic address: khatrip@UCMAIL.UC.EDU.

Shyam Prabhakaran (S)

Department of Neurology, University of Chicago, Chicago, IL, USA.

David S Liebeskind (DS)

Department of Neurology, University of California at Los Angeles, Los Angeles, CA, USA. Electronic address: davidliebeskind@yahoo.com.

Shadi Yaghi (S)

Department of Neurology, Brown University, Providence, RI, USA. Electronic address: shadiyaghi@yahoo.com.

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Classifications MeSH