Regulation of glutamate homeostasis in the nucleus accumbens by astrocytic CB1 receptors and its role in cocaine-motivated behaviors.
Anandamide
Astrocyte
Cannabinoid type I receptors
Cocaine
Glutamate homeostasis
Reinstatement
Journal
Addiction neuroscience
ISSN: 2772-3925
Titre abrégé: Addict Neurosci
Pays: Netherlands
ID NLM: 9918367980706676
Informations de publication
Date de publication:
Sep 2022
Sep 2022
Historique:
entrez:
24
11
2022
pubmed:
25
11
2022
medline:
25
11
2022
Statut:
ppublish
Résumé
Cannabinoid type 1 receptors (CB1Rs) orchestrate brain reward circuitry and are prevalent neurobiological targets for endocannabinoids and cannabis in the mammalian brain. Decades of histological and electrophysiological studies have established CB1R as presynaptic G-protein coupled receptors (GPCRs) that inhibit neurotransmitter release through retrograde signaling mechanisms. Recent seminal work demonstrates CB1R expression on astrocytes and the pivotal function of glial cells in endocannabinoid-mediated modulation of neuron-astrocyte signaling. Here, we review key facets of CB1R-mediated astroglia regulation of synaptic glutamate transmission in the nucleus accumbens with a specific emphasis on cocaine-directed behaviors.
Identifiants
pubmed: 36419922
doi: 10.1016/j.addicn.2022.100022
pmc: PMC9681119
mid: NIHMS1829265
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : NIDA NIH HHS
ID : R01 DA022340
Pays : United States
Déclaration de conflit d'intérêts
Declaration of Competing Interest The authors declare no competing interests.
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