MRI spot sign in acute intracerebral hemorrhage: an independent biomarker of hematoma expansion and poor functional outcome.


Journal

Journal of neurology
ISSN: 1432-1459
Titre abrégé: J Neurol
Pays: Germany
ID NLM: 0423161

Informations de publication

Date de publication:
Mar 2023
Historique:
received: 25 10 2022
accepted: 18 11 2022
revised: 17 11 2022
pubmed: 27 11 2022
medline: 3 3 2023
entrez: 26 11 2022
Statut: ppublish

Résumé

In acute intracerebral hemorrhage (ICH), the prognostic value of the MRI spot sign on hematoma expansion (HE) and poor functional outcome is poorly known. We retrospectively included patients admitted over a 4-year period for an acute ICH in a single institution using MRI as the first-line imaging tool. The presence and number of MRI spot signs on contrast-enhanced T1-weighted imaging was evaluated by one neuroradiologist, blinded from outcomes. The primary outcome was HE, defined as > 6 mL or > 33% ICH volume growth from initial MRI to 24-48 h follow-up imaging; the secondary outcome was poor 3-month modified Rankin score (4-6). Overall, 147 patients were included, and 62% had a spot sign. Among the 130 patients with follow-up imaging, 24% experienced HE. HE occurred in 6%, 21% and 43% patients with 0, 1 and ≥ 2 spots, respectively (P < 0.001). The MRI spot sign was independently associated with HE (adjusted OR 6.15 [95% CI 1.60-23.65]; P = 0.008), with a dose-dependent effect. The negative and positive predictive values of the spot sign for HE were 0.94 and 0.35, respectively. Poor functional outcome occurred in 27%, 32% and 71% patients with 0, 1 and ≥ 2 spots, respectively (P < 0.001). In multivariable analysis, the presence of ≥ 2 spots was independently associated with poor functional outcome (adjusted OR 3.67 [95% CI 1.21-11.10]; P = 0.024). The MRI spot sign is an independent biomarker of HE, and the presence of ≥ 2 spots is independently associated with poor 3-month outcome. The lack of spot sign is highly predictive of a favorable evolution.

Sections du résumé

BACKGROUND BACKGROUND
In acute intracerebral hemorrhage (ICH), the prognostic value of the MRI spot sign on hematoma expansion (HE) and poor functional outcome is poorly known.
METHODS METHODS
We retrospectively included patients admitted over a 4-year period for an acute ICH in a single institution using MRI as the first-line imaging tool. The presence and number of MRI spot signs on contrast-enhanced T1-weighted imaging was evaluated by one neuroradiologist, blinded from outcomes. The primary outcome was HE, defined as > 6 mL or > 33% ICH volume growth from initial MRI to 24-48 h follow-up imaging; the secondary outcome was poor 3-month modified Rankin score (4-6).
RESULTS RESULTS
Overall, 147 patients were included, and 62% had a spot sign. Among the 130 patients with follow-up imaging, 24% experienced HE. HE occurred in 6%, 21% and 43% patients with 0, 1 and ≥ 2 spots, respectively (P < 0.001). The MRI spot sign was independently associated with HE (adjusted OR 6.15 [95% CI 1.60-23.65]; P = 0.008), with a dose-dependent effect. The negative and positive predictive values of the spot sign for HE were 0.94 and 0.35, respectively. Poor functional outcome occurred in 27%, 32% and 71% patients with 0, 1 and ≥ 2 spots, respectively (P < 0.001). In multivariable analysis, the presence of ≥ 2 spots was independently associated with poor functional outcome (adjusted OR 3.67 [95% CI 1.21-11.10]; P = 0.024).
CONCLUSION CONCLUSIONS
The MRI spot sign is an independent biomarker of HE, and the presence of ≥ 2 spots is independently associated with poor 3-month outcome. The lack of spot sign is highly predictive of a favorable evolution.

Identifiants

pubmed: 36434128
doi: 10.1007/s00415-022-11498-w
pii: 10.1007/s00415-022-11498-w
doi:

Substances chimiques

Biomarkers 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1531-1542

Informations de copyright

© 2022. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany.

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Auteurs

Nefeli Valyraki (N)

Neurology Department, A. de Rothschild Foundation Hospital, 29, Rue Manin, 75019, Paris, France.

Adrien Goujon (A)

Radiology Department, A. de Rothschild Foundation Hospital, Paris, France.

Marjorie Mateos (M)

Department of Neuroradiology, CHU de Lille, Lille, France.

Adrien Lecoeuvre (A)

Clinical Research Department, A. de Rothschild Foundation Hospital, Paris, France.

Augustin Lecler (A)

Radiology Department, A. de Rothschild Foundation Hospital, Paris, France.

Igor Raynouard (I)

Neurology Department, A. de Rothschild Foundation Hospital, 29, Rue Manin, 75019, Paris, France.

Candice Sabben (C)

Neurology Department, A. de Rothschild Foundation Hospital, 29, Rue Manin, 75019, Paris, France.

Michael Obadia (M)

Neurology Department, A. de Rothschild Foundation Hospital, 29, Rue Manin, 75019, Paris, France.

Julien Savatovsky (J)

Radiology Department, A. de Rothschild Foundation Hospital, Paris, France.

Pierre Seners (P)

Neurology Department, A. de Rothschild Foundation Hospital, 29, Rue Manin, 75019, Paris, France. pierre.seners@gmail.com.
Institut de Psychiatrie et Neurosciences de Paris (IPNP), UMR_S1266, INSERM, Université de Paris, Paris, France. pierre.seners@gmail.com.

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