The human liver lipidome is significantly related to the lipid composition and aggregation susceptibility of low-density lipoprotein (LDL) particles.

Atherosclerosis Cardiovascular disease Ceramides Cholesterol Lipidomics Phosphatidylcholines Triglycerides

Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 28 08 2022
revised: 03 11 2022
accepted: 22 11 2022
pubmed: 2 12 2022
medline: 15 12 2022
entrez: 1 12 2022
Statut: ppublish

Résumé

The susceptibility of low-density lipoprotein (LDL) to aggregation predicts atherosclerotic cardiovascular disease. However, causes of interindividual variation in LDL lipid composition and aggregation susceptibility remain unclear. We examined whether the lipid composition and aggregation susceptibility of LDL reflect the lipid composition of the human liver. Liver biopsies and blood samples for isolation of LDL particles were obtained from 40 obese subjects (BMI 45.9 ± 6.1 kg/m The composition (acyl carbon number and double bond count) of hepatic triglycerides, phosphatidylcholines, and sphingomyelins (SMs) was closely associated with that of LDL particles. Hepatic dihydroceramides and ceramides were positively correlated with concentrations of the corresponding SM species in LDL as well with LDL aggregation. These relationships remained statistically significant after adjustment for age, sex, and body mass index. Lipid composition of LDL reflects that of the human liver in obese patients. Changes in hepatic sphingolipid metabolism may contribute to interindividual variation of LDL lipid composition and susceptibility to aggregation.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
The susceptibility of low-density lipoprotein (LDL) to aggregation predicts atherosclerotic cardiovascular disease. However, causes of interindividual variation in LDL lipid composition and aggregation susceptibility remain unclear. We examined whether the lipid composition and aggregation susceptibility of LDL reflect the lipid composition of the human liver.
METHODS METHODS
Liver biopsies and blood samples for isolation of LDL particles were obtained from 40 obese subjects (BMI 45.9 ± 6.1 kg/m
RESULTS RESULTS
The composition (acyl carbon number and double bond count) of hepatic triglycerides, phosphatidylcholines, and sphingomyelins (SMs) was closely associated with that of LDL particles. Hepatic dihydroceramides and ceramides were positively correlated with concentrations of the corresponding SM species in LDL as well with LDL aggregation. These relationships remained statistically significant after adjustment for age, sex, and body mass index.
CONCLUSIONS CONCLUSIONS
Lipid composition of LDL reflects that of the human liver in obese patients. Changes in hepatic sphingolipid metabolism may contribute to interindividual variation of LDL lipid composition and susceptibility to aggregation.

Identifiants

pubmed: 36455305
pii: S0021-9150(22)01528-3
doi: 10.1016/j.atherosclerosis.2022.11.018
pii:
doi:

Substances chimiques

Lipoproteins, LDL 0
Triglycerides 0
Sphingomyelins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

22-29

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare the following financial interests/personal relationships which may be considered as potential competing interests: K.Ö. and M.R. have a patent pending on the LDL aggregation method.

Auteurs

Mari Lahelma (M)

Minerva Foundation Institute for Medical Research, Helsinki, Finland; Department of Medicine, University of Helsinki and Helsinki University Hospital, Helsinki, Finland. Electronic address: mari.lahelma@helsinki.fi.

Sami Qadri (S)

Minerva Foundation Institute for Medical Research, Helsinki, Finland; Department of Medicine, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Noora Ahlholm (N)

Minerva Foundation Institute for Medical Research, Helsinki, Finland; Department of Medicine, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Kimmo Porthan (K)

Minerva Foundation Institute for Medical Research, Helsinki, Finland; Department of Medicine, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Maija Ruuth (M)

Atherosclerosis Research Laboratory, Wihuri Research Institute, Helsinki, Finland.

Anne Juuti (A)

Department of Gastrointestinal Surgery, Abdominal Center, University of Helsinki and Helsinki University Hospital, Helsinki, Finland.

Matej Orešič (M)

School of Medical Sciences, Örebro University, Örebro, Sweden; Turku Bioscience Centre, University of Turku and Åbo Akademi University, Turku, Finland.

Tuulia Hyötyläinen (T)

Department of Chemistry, Örebro University, Örebro, Sweden.

Katariina Öörni (K)

Atherosclerosis Research Laboratory, Wihuri Research Institute, Helsinki, Finland.

Hannele Yki-Järvinen (H)

Minerva Foundation Institute for Medical Research, Helsinki, Finland; Department of Medicine, University of Helsinki and Helsinki University Hospital, Helsinki, Finland. Electronic address: hannele.yki-jarvinen@helsinki.fi.

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Classifications MeSH