High thrombus platelet content is associated with a lower rate of first pass effect in stroke treated by endovascular therapy.

Platelets biomarker endovascular therapy first pass effect thrombosis

Journal

European stroke journal
ISSN: 2396-9881
Titre abrégé: Eur Stroke J
Pays: England
ID NLM: 101688446

Informations de publication

Date de publication:
Dec 2022
Historique:
received: 07 02 2022
accepted: 02 06 2022
entrez: 8 12 2022
pubmed: 9 12 2022
medline: 9 12 2022
Statut: ppublish

Résumé

First pass effect (FPE), the occurrence of complete reperfusion after one pass with no rescue attempt during endovascular therapy (EVT), is associated with the best clinical outcome after an acute ischemic stroke (AIS). Previous studies evaluating FPE occurrence according to EVT technical strategies, occlusion locations, or thrombus composition have provided controversial results. Here, we performed a correlation analysis between FPE occurrence and AIS thrombus cellular composition, as assessed using quantitative biochemical assays. Homogenates of AIS thrombi from 250 patients were prepared by mechanical grinding. Platelet, red blood cell (RBC), and leukocyte contents of AIS thrombi were respectively estimated by quantification of GP (glycoprotein) VI, heme, and DNA in thrombus homogenates. FPE was defined as a modified Thrombolysis in Cerebral Infraction (mTICI) score of 2C or 3 after a single EVT device pass. AIS thrombi successfully removed after a single pass were poorer in GPVI (0.098 ± 0.023 vs 0.111 ± 0.024 ng/mg, Thrombus platelet content may hamper thrombus removal by EVT. This result suggests that adjunctive therapies or functionalization of retrieval devices targeting platelets may improve EVT efficacy.

Sections du résumé

Background and purpose UNASSIGNED
First pass effect (FPE), the occurrence of complete reperfusion after one pass with no rescue attempt during endovascular therapy (EVT), is associated with the best clinical outcome after an acute ischemic stroke (AIS). Previous studies evaluating FPE occurrence according to EVT technical strategies, occlusion locations, or thrombus composition have provided controversial results. Here, we performed a correlation analysis between FPE occurrence and AIS thrombus cellular composition, as assessed using quantitative biochemical assays.
Patients and methods UNASSIGNED
Homogenates of AIS thrombi from 250 patients were prepared by mechanical grinding. Platelet, red blood cell (RBC), and leukocyte contents of AIS thrombi were respectively estimated by quantification of GP (glycoprotein) VI, heme, and DNA in thrombus homogenates. FPE was defined as a modified Thrombolysis in Cerebral Infraction (mTICI) score of 2C or 3 after a single EVT device pass.
Results UNASSIGNED
AIS thrombi successfully removed after a single pass were poorer in GPVI (0.098 ± 0.023 vs 0.111 ± 0.024 ng/mg,
Discussion and conclusion UNASSIGNED
Thrombus platelet content may hamper thrombus removal by EVT. This result suggests that adjunctive therapies or functionalization of retrieval devices targeting platelets may improve EVT efficacy.

Identifiants

pubmed: 36478752
doi: 10.1177/23969873221108740
pii: 10.1177_23969873221108740
pmc: PMC9720861
doi:

Types de publication

Journal Article

Langues

eng

Pagination

376-383

Informations de copyright

© European Stroke Organisation 2022.

Déclaration de conflit d'intérêts

The author(s) declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article.

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Auteurs

François Delvoye (F)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
University of Liege, Liège, Belgium.

Lucas Di Meglio (L)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Arturo Consoli (A)

Interventional Neuroradiology Department, Hopital Foch, Suresnes, France.

Mialitiana Solo Nomenjanahary (MS)

Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Sébastien Dupont (S)

Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Julien Labreuche (J)

Department of Biostatistics, CHU Lille, Lille, France.

Benjamin Maier (B)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Michel Piotin (M)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Raphael Blanc (R)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Simon Escalard (S)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Perrine Boursin (P)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Mylène Hamdani (M)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Hocine Redjem (H)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Stanislas Smajda (S)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Solène Hébert (S)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.

Candice Sabben (C)

Department of Neurology, Rothschild Foundation Hospital, Paris, France.

Alain Maertens de Noordhout (AM)

University of Liege, Liège, Belgium.

Martine Jandrot-Perrus (M)

Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Bertrand Lapergue (B)

Department of Neurology, Hopital Foch, Suresnes, France.

Mikael Mazighi (M)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.
Université de Paris, Paris, France.
FHU Neurovasc, Department of Neurology, Hopital Lariboisère, APHP Nord, Paris, France.

Benoit Ho-Tin-Noé (B)

Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.

Jean-Philippe Desilles (JP)

Interventional Neuroradiology Department and Biological Resources Center, Rothschild Foundation Hospital, Paris, France.
Laboratory of Vascular Translational Science, U1148 INSERM, Université de Paris, Paris, France.
Université de Paris, Paris, France.

Classifications MeSH