Functional assessment of DMRT1 variants and their pathogenicity for isolated male infertility.

DMRT1 gonadal development luciferase assay male infertility nonobstructive azoospermia

Journal

Fertility and sterility
ISSN: 1556-5653
Titre abrégé: Fertil Steril
Pays: United States
ID NLM: 0372772

Informations de publication

Date de publication:
02 2023
Historique:
received: 28 06 2022
revised: 18 10 2022
accepted: 19 10 2022
pubmed: 27 12 2022
medline: 8 2 2023
entrez: 26 12 2022
Statut: ppublish

Résumé

To study the impact of Doublesex and mab-3-related transcription factor 1 (DMRT1) gene variants on the encoded protein's function and the variants' pathogenic relevance for isolated male infertility caused by azoospermia. This study established a novel luciferase assay for DMRT1 missense variants using 2 different target promotors and validated the assay by analyzing previously published variants associated with differences in sex development. University genetics research institute and tertiary referral center for couples' infertility. Eleven infertile men with severely impaired spermatogenesis resulting in crypto- or azoospermia and carrying rare heterozygous missense variants in DMRT1 were identified within the Male Reproductive Genomics study. Luciferase assays with human DMRT1 variants to test functional effects on the CYP19A1 and Stra8 target promoters. We first developed and refined luciferase assays to reliably test the functional impact of DMRT1 missense variants. Next, the assay was validated by analyzing 2 DMRT1 variants associated with differences in sex development, of which c.240G>C p.(Arg80Ser) displayed highly significant effects on both target promoters compared with the wild-type protein (-40% and +100%, respectively) and c.331A>G p.(Arg111Gly) had a significant effect on the Stra8 promoter (-76%). We then systematically characterized 11 DMRT1 variants identified in infertile men. The de novo variant c.344T>A p.(Met115Lys) showed a pronounced loss of function in both DMRT1 target promoters (-100% and -86%, respectively). Variants c.308A>G p.(Lys103Arg) and c.991G>C p.(Asp331His) showed a significant gain of function exclusively for the CYP19A1 promoter (+15% and +19%, respectively). Based on these results, 3 variants were reclassified according to clinical guidelines. The present study highlights the importance of functionally characterizing DMRT1 variants of uncertain clinical significance. Using luciferase assays for diagnostic purposes enables an improved causal diagnosis for isolated male infertility.

Identifiants

pubmed: 36572623
pii: S0015-0282(22)01982-3
doi: 10.1016/j.fertnstert.2022.10.032
pii:
doi:

Substances chimiques

Transcription Factors 0
DMRT1 protein 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

219-228

Informations de copyright

Copyright © 2022 American Society for Reproductive Medicine. Published by Elsevier Inc. All rights reserved.

Auteurs

Jana Emich (J)

Institute of Reproductive Genetics, University of Münster, Münster, Germany.

Avinash S Gaikwad (AS)

Institute of Reproductive Genetics, University of Münster, Münster, Germany.

Birgit Stallmeyer (B)

Institute of Reproductive Genetics, University of Münster, Münster, Germany.

Daniela Fietz (D)

Institute for Veterinary Anatomy, Histology and Embryology, Justus Liebig University Gießen, Gießen, Germany; Hessian Centre of Reproductive Medicine, Justus Liebig University Gießen, Gießen, Germany.

Hans-Christian Schuppe (HC)

Hessian Centre of Reproductive Medicine, Justus Liebig University Gießen, Gießen, Germany; Department of Urology, Paediatric Urology and Andrology, Justus Liebig University Gießen, Gießen, Germany.

Manon S Oud (MS)

Department of Human Genetics, Radboud University Medical Centre, Nijmegen, the Netherlands.

Sabine Kliesch (S)

Centre of Reproductive Medicine and Andrology, University Hospital Münster, Münster, Germany.

Jörg Gromoll (J)

Centre of Reproductive Medicine and Andrology, University Hospital Münster, Münster, Germany.

Corinna Friedrich (C)

Institute of Reproductive Genetics, University of Münster, Münster, Germany. Electronic address: corinna.friedrich@ukmuenster.de.

Frank Tüttelmann (F)

Institute of Reproductive Genetics, University of Münster, Münster, Germany.

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Classifications MeSH