Prenatal exposure to persistent organic pollutants and changes in infant growth and childhood growth trajectories.


Journal

Chemosphere
ISSN: 1879-1298
Titre abrégé: Chemosphere
Pays: England
ID NLM: 0320657

Informations de publication

Date de publication:
Feb 2023
Historique:
received: 30 08 2022
revised: 25 12 2022
accepted: 28 12 2022
pubmed: 2 1 2023
medline: 17 1 2023
entrez: 1 1 2023
Statut: ppublish

Résumé

Children are born with a burden of persistent organic pollutants (POPs) which may have endocrine disrupting properties and have been postulated to contribute to the rise in childhood obesity. The current evidence is equivocal, which may partly because many studies investigate the effects at one time point during childhood. We assessed associations between prenatal exposure to POPs and growth during infancy and childhood. We used data from two Belgian cohorts with cord blood measurements of five organochlorines [(dichlorodiphenyldichloroethylene (p,p'-DDE), hexachlorobenzene (HCB), polychlorinated biphenyls (PCB-138, -150, -180)] (N = 1418) and two perfluoroalkyl substances [perfluorooctanoic acid (PFOA) and perfluorooctanesulfonic acid (PFOS)] (N = 346). We assessed infant growth, defined as body mass index (BMI) z-score change between birth and 2 years, and childhood growth, characterized as BMI trajectory from birth to 8 years. To evaluate associations between POP exposures and infant growth, we applied a multi-pollutant approach, using penalized elastic net regression with stability selection, controlling for covariates. To evaluate associations with childhood growth, we used single-pollutant linear mixed models with random effects for child individual, parametrized using a natural cubic spline formulation. PCB-153 was associated with increased and p,p'-DDE with decreased infant growth, although these results were imprecise. No clear association between any of the exposures and longer-term childhood growth trajectories was observed. We did not find evidence of effect modification by child sex. Our results suggest that prenatal exposure to PCB-153 and p,p'-DDE may affect infant growth in the first two years, with no evidence of more persistent effects.

Sections du résumé

BACKGROUND BACKGROUND
Children are born with a burden of persistent organic pollutants (POPs) which may have endocrine disrupting properties and have been postulated to contribute to the rise in childhood obesity. The current evidence is equivocal, which may partly because many studies investigate the effects at one time point during childhood. We assessed associations between prenatal exposure to POPs and growth during infancy and childhood.
METHODS METHODS
We used data from two Belgian cohorts with cord blood measurements of five organochlorines [(dichlorodiphenyldichloroethylene (p,p'-DDE), hexachlorobenzene (HCB), polychlorinated biphenyls (PCB-138, -150, -180)] (N = 1418) and two perfluoroalkyl substances [perfluorooctanoic acid (PFOA) and perfluorooctanesulfonic acid (PFOS)] (N = 346). We assessed infant growth, defined as body mass index (BMI) z-score change between birth and 2 years, and childhood growth, characterized as BMI trajectory from birth to 8 years. To evaluate associations between POP exposures and infant growth, we applied a multi-pollutant approach, using penalized elastic net regression with stability selection, controlling for covariates. To evaluate associations with childhood growth, we used single-pollutant linear mixed models with random effects for child individual, parametrized using a natural cubic spline formulation.
RESULTS RESULTS
PCB-153 was associated with increased and p,p'-DDE with decreased infant growth, although these results were imprecise. No clear association between any of the exposures and longer-term childhood growth trajectories was observed. We did not find evidence of effect modification by child sex.
CONCLUSION CONCLUSIONS
Our results suggest that prenatal exposure to PCB-153 and p,p'-DDE may affect infant growth in the first two years, with no evidence of more persistent effects.

Identifiants

pubmed: 36587911
pii: S0045-6535(22)04188-1
doi: 10.1016/j.chemosphere.2022.137695
pii:
doi:

Substances chimiques

2,4,5,2',4',5'-hexachlorobiphenyl ZRU0C9E32O
Persistent Organic Pollutants 0
Dichlorodiphenyl Dichloroethylene 4M7FS82U08
Polychlorinated Biphenyls DFC2HB4I0K
Environmental Pollutants 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

137695

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Anran Cai (A)

Institute for Risk Assessment Sciences, Department of Population Health Sciences, Utrecht University, Utrecht, the Netherlands; VITO Health, Flemish Institute for Technological Research (VITO), Mol, Belgium. Electronic address: a.cai@uu.nl.

Lützen Portengen (L)

Institute for Risk Assessment Sciences, Department of Population Health Sciences, Utrecht University, Utrecht, the Netherlands.

Eva Govarts (E)

VITO Health, Flemish Institute for Technological Research (VITO), Mol, Belgium.

Laura Rodriguez Martin (LR)

VITO Health, Flemish Institute for Technological Research (VITO), Mol, Belgium.

Greet Schoeters (G)

Department of Biomedical Sciences, University of Antwerp, Antwerp, Belgium.

Juliette Legler (J)

Institute for Risk Assessment Sciences, Department of Population Health Sciences, Utrecht University, Utrecht, the Netherlands.

Roel Vermeulen (R)

Institute for Risk Assessment Sciences, Department of Population Health Sciences, Utrecht University, Utrecht, the Netherlands; Julius Center for Health Sciences and Primary Care, University Medical Center Utrecht, Utrecht, the Netherlands.

Virissa Lenters (V)

Julius Center for Health Sciences and Primary Care, University Medical Center Utrecht, Utrecht, the Netherlands.

Sylvie Remy (S)

VITO Health, Flemish Institute for Technological Research (VITO), Mol, Belgium.

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Classifications MeSH