Dominant-negative signal transducer and activator of transcription (STAT)3 variants in adult patients: A single center experience.


Journal

Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960

Informations de publication

Date de publication:
2022
Historique:
received: 15 09 2022
accepted: 07 12 2022
entrez: 6 1 2023
pubmed: 7 1 2023
medline: 10 1 2023
Statut: epublish

Résumé

Autosomal dominant hyper-IgE syndrome (AD-HIES) caused by dominant negative (DN) variants in the signal transducer and activator of transcription 3 gene ( To describe our experience with the diagnosis and treatment of adult patients with AD-HIES induced by DN- The medical records of adult patients (>18 years) treated at the Allergy and Clinical Immunology Clinic of Hadassah Medical Center, Jerusalem, Israel, were retrospectively analyzed. Immune and genetic workups were used to confirm diagnosis. Three adult patients (2 males; age 29-41 years) were diagnosed with DN- Dupilumab can be used safely as a biotherapy for atopic dermatitis in these patients as it can effectively alleviate eczema-related symptoms. Immunologists and dermatologists treating AD-HIES adult patients should be aware of demodicosis as a possible manifestation. DN-

Sections du résumé

Background
Autosomal dominant hyper-IgE syndrome (AD-HIES) caused by dominant negative (DN) variants in the signal transducer and activator of transcription 3 gene (
Objectives
To describe our experience with the diagnosis and treatment of adult patients with AD-HIES induced by DN-
Methods
The medical records of adult patients (>18 years) treated at the Allergy and Clinical Immunology Clinic of Hadassah Medical Center, Jerusalem, Israel, were retrospectively analyzed. Immune and genetic workups were used to confirm diagnosis.
Results
Three adult patients (2 males; age 29-41 years) were diagnosed with DN-
Discussion
Dupilumab can be used safely as a biotherapy for atopic dermatitis in these patients as it can effectively alleviate eczema-related symptoms. Immunologists and dermatologists treating AD-HIES adult patients should be aware of demodicosis as a possible manifestation. DN-

Identifiants

pubmed: 36605204
doi: 10.3389/fimmu.2022.1044933
pmc: PMC9807906
doi:

Substances chimiques

STAT3 Transcription Factor 0
STAT3 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1044933

Informations de copyright

Copyright © 2022 Shamriz, Rubin, Simon, Lev, Barel, Somech, Korem, Matza Porges, Freund, Hagin, Garty, Nahum, Molho Pessach and Tal.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Oded Shamriz (O)

Allergy and Clinical Immunology Unit, Department of Medicine, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.
The Lautenberg Center for Immunology and Cancer Research, Institute of Medical Research Israel-Canada, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

Limor Rubin (L)

Allergy and Clinical Immunology Unit, Department of Medicine, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

Amos J Simon (AJ)

Sheba Cancer Research Center and Institute of Hematology, Sheba Medical Center, Ramat Gan, Israel.
Pediatric Department A and the Immunology Service, Jeffrey Modell Foundation Center, Edmond and Lily Safra Children's Hospital, Tel-Hashomer Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Atar Lev (A)

Pediatric Department A and the Immunology Service, Jeffrey Modell Foundation Center, Edmond and Lily Safra Children's Hospital, Tel-Hashomer Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Ortal Barel (O)

The Genomic Unit, Sheba Cancer Research Center, Sheba Medical Center, Ramat Gan, Israel.
Sheba Medical Center, Wohl Institute of Translational Medicine, Ramat Gan, Israel.

Raz Somech (R)

Pediatric Department A and the Immunology Service, Jeffrey Modell Foundation Center, Edmond and Lily Safra Children's Hospital, Tel-Hashomer Medical Center, Affiliated to the Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Maya Korem (M)

Department of Clinical Microbiology and Infectious Diseases, Hadassah Medical Center, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

Sigal Matza Porges (S)

Department of Human Genetics, Institute for Medical Research, the Hebrew University of Jerusalem, Jerusalem, Israel.
Department of Biotechnology, Hadassah Academic College, Jerusalem, Israel.

Tal Freund (T)

Allergy and Clinical Immunology Unit, Department of Medicine, Tel-Aviv Sourasky Medical Center and Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

David Hagin (D)

Allergy and Clinical Immunology Unit, Department of Medicine, Tel-Aviv Sourasky Medical Center and Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel.

Ben Zion Garty (BZ)

Sackler School of Medicine, Tel Aviv University, Tel-Aviv, Israel.
Felsenstein Medical Research Center, Rabin Medical Center, Petach-Tikva, Israel.
Allergy and Clinical Immunology Unit, Schneider Children's Medical Center, Petach-Tikva, Israel.

Amit Nahum (A)

Pediatrics Department A, Soroka University Medical Center and Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva, Israel.

Vered Molho Pessach (V)

Pediatric Dermatology Service, Department of Dermatology, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

Yuval Tal (Y)

Allergy and Clinical Immunology Unit, Department of Medicine, Hadassah Medical Organization, Faculty of Medicine, Hebrew University of Jerusalem, Jerusalem, Israel.

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Classifications MeSH