The c-di-AMP-binding protein CbpB modulates the level of ppGpp alarmone in Streptococcus agalactiae.


Journal

The FEBS journal
ISSN: 1742-4658
Titre abrégé: FEBS J
Pays: England
ID NLM: 101229646

Informations de publication

Date de publication:
06 2023
Historique:
revised: 07 12 2022
received: 17 08 2022
accepted: 09 01 2023
medline: 8 6 2023
pubmed: 12 1 2023
entrez: 11 1 2023
Statut: ppublish

Résumé

Cyclic di-AMP is an essential signalling molecule in Gram-positive bacteria. This second messenger regulates the osmotic pressure of the cell by interacting directly with the regulatory domains, either RCK_C or CBS domains, of several potassium and osmolyte uptake membrane protein systems. Cyclic di-AMP also targets stand-alone CBS domain proteins such as DarB in Bacillus subtilis and CbpB in Listeria monocytogenes. We show here that the CbpB protein of Group B Streptococcus binds c-di-AMP with a very high affinity. Crystal structures of CbpB reveal the determinants of binding specificity and significant conformational changes occurring upon c-di-AMP binding. Deletion of the cbpB gene alters bacterial growth in low potassium conditions most likely due to a decrease in the amount of ppGpp caused by a loss of interaction between CbpB and Rel, the GTP/GDP pyrophosphokinase.

Identifiants

pubmed: 36629470
doi: 10.1111/febs.16724
doi:

Substances chimiques

Carrier Proteins 0
cyclic diadenosine phosphate 0
Guanosine Pentaphosphate 38918-96-6
Guanosine Tetraphosphate 33503-72-9
Bacterial Proteins 0
Cyclic AMP E0399OZS9N
Dinucleoside Phosphates 0
Potassium RWP5GA015D

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2968-2992

Informations de copyright

© 2023 Federation of European Biochemical Societies.

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Auteurs

Giovanni Covaleda-Cortés (G)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Ariel Mechaly (A)

CNRS-UMR 3528, Crystallography Platform, Center for Technological Resources and Research, Institut Pasteur, Université Paris Cité, France.

Terry Brissac (T)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Heike Baehre (H)

Research Core Unit Metabolomics, Hannover Medical School, Germany.

Laura Devaux (L)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Patrick England (P)

CNRS UMR 3528, Molecular Biophysics Platform, Center for Technological Resources and Research, Institut Pasteur, Université Paris Cité, France.

Bertrand Raynal (B)

CNRS UMR 3528, Molecular Biophysics Platform, Center for Technological Resources and Research, Institut Pasteur, Université Paris Cité, France.

Sylviane Hoos (S)

CNRS UMR 3528, Molecular Biophysics Platform, Center for Technological Resources and Research, Institut Pasteur, Université Paris Cité, France.

Myriam Gominet (M)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Arnaud Firon (A)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Patrick Trieu-Cuot (P)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

Pierre Alexandre Kaminski (PA)

Unité Biologie des Bactéries Pathogènes à Gram-positif, CNRS UMR 6047, Institut Pasteur, Université Paris Cité, France.

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