Potential value of saline-induced Pd/Pa ratio in patients with coronary artery stenosis.

epicardial coronary artery fractional flow reserve physiological assessment resting full-cycle ratio saline-induced Pd/Pa ratio

Journal

Frontiers in cardiovascular medicine
ISSN: 2297-055X
Titre abrégé: Front Cardiovasc Med
Pays: Switzerland
ID NLM: 101653388

Informations de publication

Date de publication:
2022
Historique:
received: 24 07 2022
accepted: 12 12 2022
entrez: 23 1 2023
pubmed: 24 1 2023
medline: 24 1 2023
Statut: epublish

Résumé

Fractional flow reserve (FFR) is the current gold standard for identifying myocardial ischemia in individuals with coronary artery stenosis. However, FFR is not penetrated as much worldwide due to time consumption, costs associated with adenosine, FFR-related discomfort, and complications. Resting physiological indexes may be widely accepted alternatives to FFR, while the discrepancies with FFR were found in up to 20% of lesions. The saline-induced Pd/Pa ratio (SPR) is a new simplified option for evaluating coronary stenosis. However, the clinical implication of SPR remains unclear. In the present study, we aimed to compare the accuracies of SPR and resting full-cycle ratio (RFR) and to investigate the incremental value of SPR in clinical practice. In this multicenter prospective study, 112 coronary lesions (105 patients) were evaluated by SPR, RFR, and FFR. The overall median age was 71 years, and 84.8% were men. SPR was correlated more strongly with FFR than with RFR ( Saline-induced Pd/Pa ratio predicted FFR more accurately than RFR. SPR could be an alternative method for evaluating coronary artery stenosis and further investigation including elucidation of the mechanism of SPR is needed (225 words).

Sections du résumé

Background UNASSIGNED
Fractional flow reserve (FFR) is the current gold standard for identifying myocardial ischemia in individuals with coronary artery stenosis. However, FFR is not penetrated as much worldwide due to time consumption, costs associated with adenosine, FFR-related discomfort, and complications. Resting physiological indexes may be widely accepted alternatives to FFR, while the discrepancies with FFR were found in up to 20% of lesions. The saline-induced Pd/Pa ratio (SPR) is a new simplified option for evaluating coronary stenosis. However, the clinical implication of SPR remains unclear.
Objectives UNASSIGNED
In the present study, we aimed to compare the accuracies of SPR and resting full-cycle ratio (RFR) and to investigate the incremental value of SPR in clinical practice.
Methods UNASSIGNED
In this multicenter prospective study, 112 coronary lesions (105 patients) were evaluated by SPR, RFR, and FFR.
Results UNASSIGNED
The overall median age was 71 years, and 84.8% were men. SPR was correlated more strongly with FFR than with RFR (
Conclusion UNASSIGNED
Saline-induced Pd/Pa ratio predicted FFR more accurately than RFR. SPR could be an alternative method for evaluating coronary artery stenosis and further investigation including elucidation of the mechanism of SPR is needed (225 words).

Identifiants

pubmed: 36684556
doi: 10.3389/fcvm.2022.1001833
pmc: PMC9853169
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1001833

Informations de copyright

Copyright © 2023 Kiriyama, Kiyosue, Minatsuki, Kawahara, Katsushika, Kamon, Hirose, Shinohara, Miura, Saito, Kikuchi, Kodera, Hatano, Ando, Myojo, Itoh, Yamamoto, Ikenouchi, Takeda and Komuro.

Déclaration de conflit d'intérêts

MMi received a consultant fee and lecture fee from Abbott Vascular. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Hiroyuki Kiriyama (H)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Arihiro Kiyosue (A)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.
Department of Cardiology, Moriyama Memorial Hospital, Tokyo, Japan.

Shun Minatsuki (S)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Takuya Kawahara (T)

Biostatistics Division, Clinical Research Promotion Center, The University of Tokyo Hospital, Tokyo, Japan.

Susumu Katsushika (S)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Tatsuya Kamon (T)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Kazutoshi Hirose (K)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Hiroki Shinohara (H)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Mizuki Miura (M)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Akihito Saito (A)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Hironobu Kikuchi (H)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Satoshi Kodera (S)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Masaru Hatano (M)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Jiro Ando (J)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Masahiro Myojo (M)

Department of Cardiology, Kanto Central Hospital of the Mutual Aid Association of Public School Teachers, Tokyo, Japan.

Nobuhiko Itoh (N)

Department of Cardiology, Kanto Central Hospital of the Mutual Aid Association of Public School Teachers, Tokyo, Japan.

Keisuke Yamamoto (K)

Department of Cardiology, Japanese Red Cross Medical Center, Tokyo, Japan.

Hiroshi Ikenouchi (H)

Department of Cardiology, Japanese Red Cross Medical Center, Tokyo, Japan.

Norifumi Takeda (N)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Issei Komuro (I)

Department of Cardiovascular Medicine, The University of Tokyo Hospital, Tokyo, Japan.

Classifications MeSH