Persistent virus-specific and clonally expanded antibody-secreting cells respond to induced self-antigen in the CNS.

Antibody-secreting cells Autoimmunity CNS tolerance Multiple sclerosis Viral infection

Journal

Acta neuropathologica
ISSN: 1432-0533
Titre abrégé: Acta Neuropathol
Pays: Germany
ID NLM: 0412041

Informations de publication

Date de publication:
03 2023
Historique:
received: 06 09 2022
accepted: 02 01 2023
revised: 20 12 2022
pubmed: 26 1 2023
medline: 16 2 2023
entrez: 25 1 2023
Statut: ppublish

Résumé

B cells contribute to the pathogenesis of both cellular- and humoral-mediated central nervous system (CNS) inflammatory diseases through a variety of mechanisms. In such conditions, B cells may enter the CNS parenchyma and contribute to local tissue destruction. It remains unexplored, however, how infection and autoimmunity drive transcriptional phenotypes, repertoire features, and antibody functionality. Here, we profiled B cells from the CNS of murine models of intracranial (i.c.) viral infections and autoimmunity. We identified a population of clonally expanded, antibody-secreting cells (ASCs) that had undergone class-switch recombination and extensive somatic hypermutation following i.c. infection with attenuated lymphocytic choriomeningitis virus (rLCMV). Recombinant expression and characterisation of these antibodies revealed specificity to viral antigens (LCMV glycoprotein GP), correlating with ASC persistence in the brain weeks after resolved infection. Furthermore, these virus-specific ASCs upregulated proliferation and expansion programs in response to the conditional and transient induction of the LCMV GP as a neo-self antigen by astrocytes. This class-switched, clonally expanded, and mutated population persisted and was even more pronounced when peripheral B cells were depleted prior to autoantigen induction in the CNS. In contrast, the most expanded B cell clones in mice with persistent expression of LCMV GP in the CNS did not exhibit neo-self antigen specificity, potentially a consequence of local tolerance induction. Finally, a comparable population of clonally expanded, class-switched, and proliferating ASCs was detected in the cerebrospinal fluid of relapsing multiple sclerosis (RMS) patients. Taken together, our findings support the existence of B cells that populate the CNS and are capable of responding to locally encountered autoantigens.

Identifiants

pubmed: 36695896
doi: 10.1007/s00401-023-02537-5
pii: 10.1007/s00401-023-02537-5
pmc: PMC9925600
doi:

Substances chimiques

Autoantigens 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

335-355

Informations de copyright

© 2023. The Author(s).

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Auteurs

Andreas Agrafiotis (A)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.
Institute of Microbiology, ETH Zurich, Zurich, Switzerland.

Raphael Dizerens (R)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Ilena Vincenti (I)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Ingrid Wagner (I)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Raphael Kuhn (R)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Danielle Shlesinger (D)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Marcos Manero-Carranza (M)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Tudor-Stefan Cotet (TS)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Kai-Lin Hong (KL)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Nicolas Page (N)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Nicolas Fonta (N)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Ghazal Shammas (G)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Alexandre Mariotte (A)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Margot Piccinno (M)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.

Mario Kreutzfeldt (M)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland.
Division of Clinical Pathology, Geneva University Hospital, Geneva, Switzerland.

Benedikt Gruntz (B)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Roy Ehling (R)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Alessandro Genovese (A)

Institute of Microbiology, ETH Zurich, Zurich, Switzerland.

Alessandro Pedrioli (A)

Institute of Microbiology, ETH Zurich, Zurich, Switzerland.

Andreas Dounas (A)

Institute for Biomedical Engineering, University and ETH Zurich, Zurich, Switzerland.

Sören Franzenburg (S)

Institute of Clinical Molecular Biology, Kiel University and University Medical Center Schleswig-Holstein, Kiel, Germany.

Hayrettin Tumani (H)

Department of Neurology, University Hospital Ulm, Ulm, Germany.

Tania Kümpfel (T)

Institute of Clinical Neuroimmunology, Faculty of Medicine, University Hospital and Biomedical Center (BMC), LMU Munich, Munich, Germany.
Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Martinsried, Germany.
Munich Cluster of Systems Neurology (SyNergy), Munich, Germany.

Vladyslav Kavaka (V)

Institute of Clinical Neuroimmunology, Faculty of Medicine, University Hospital and Biomedical Center (BMC), LMU Munich, Munich, Germany.
Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Martinsried, Germany.

Lisa Ann Gerdes (LA)

Institute of Clinical Neuroimmunology, Faculty of Medicine, University Hospital and Biomedical Center (BMC), LMU Munich, Munich, Germany.
Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Martinsried, Germany.
Munich Cluster of Systems Neurology (SyNergy), Munich, Germany.

Klaus Dornmair (K)

Institute of Clinical Neuroimmunology, Faculty of Medicine, University Hospital and Biomedical Center (BMC), LMU Munich, Munich, Germany.
Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Martinsried, Germany.
Munich Cluster of Systems Neurology (SyNergy), Munich, Germany.

Eduardo Beltrán (E)

Institute of Clinical Neuroimmunology, Faculty of Medicine, University Hospital and Biomedical Center (BMC), LMU Munich, Munich, Germany.
Biomedical Center (BMC), Faculty of Medicine, LMU Munich, Martinsried, Germany.
Munich Cluster of Systems Neurology (SyNergy), Munich, Germany.

Annette Oxenius (A)

Institute of Microbiology, ETH Zurich, Zurich, Switzerland.

Sai T Reddy (ST)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland.

Doron Merkler (D)

Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland. Doron.Merkler@unige.ch.
Division of Clinical Pathology, Geneva University Hospital, Geneva, Switzerland. Doron.Merkler@unige.ch.

Alexander Yermanos (A)

Department of Biosystems Science and Engineering, ETH Zurich, Basel, Switzerland. ayermanos@gmail.com.
Institute of Microbiology, ETH Zurich, Zurich, Switzerland. ayermanos@gmail.com.
Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland. ayermanos@gmail.com.
Center for Translational Immunology, University Medical Center Utrecht, Utrecht, Netherlands. ayermanos@gmail.com.

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