Liver fibrosis is closely related to metabolic factors in metabolic associated fatty liver disease with hepatitis B virus infection.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
25 01 2023
Historique:
received: 31 10 2022
accepted: 17 01 2023
entrez: 25 1 2023
pubmed: 26 1 2023
medline: 28 1 2023
Statut: epublish

Résumé

This case-control study aimed to identify the clinical characteristics and explore the risk factors for liver fibrosis in metabolic associated fatty liver disease (MAFLD) patients with hepatitis B virus (HBV) infection. The patients were grouped into MAFLD + HBV and MAFLD (without HBV infection). Propensity score matching (PSM) was used to match baseline features between the groups. We included 401 patients with biopsy-proven MAFLD, 179 of whom had HBV infection. A total of 83 pairs were successfully matched via PSM, and steatosis scores and ballooning in the MAFLD + HBV group were lower than those in the MAFLD group, while the inflammation scores and liver fibrosis stages were higher. After adjusted for confounding factors, HBV infection was associated with a higher risk of significant liver fibrosis in patients with MAFLD [odds ratio (OR): 3.140, P = 0.003]. Overall, 43.58% (78/179) of patients in the MAFLD + HBV group had significant liver fibrosis. Further multivariate regression analysis, hypertension (OR: 2.640; P = 0.031), type 2 diabetes (OR: 4.939; P = 0.035), and elevated glutamyl-transferase levels (OR: 3.980; P = 0.001) were risk factors for liver fibrosis in the MAFLD + HBV group. This suggests metabolic rather than viral factors are more closely associated with liver fibrosis in MAFLD patients with HBV infection.

Identifiants

pubmed: 36697471
doi: 10.1038/s41598-023-28351-3
pii: 10.1038/s41598-023-28351-3
pmc: PMC9877001
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1388

Informations de copyright

© 2023. The Author(s).

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Auteurs

Haifeng Lv (H)

Department of Intensive Care Unit, The First Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310000, Zhejiang, China.

Yanming Jiang (Y)

Department of Hepatology, The Affiliated Hospital of Hangzhou Normal University, No. 126 Wenzhou Road, Gongshu District, Hangzhou, 310015, Zhejiang, China.

Geli Zhu (G)

Department of Hepatology, The Affiliated Hospital of Hangzhou Normal University, No. 126 Wenzhou Road, Gongshu District, Hangzhou, 310015, Zhejiang, China.

Shiyi Liu (S)

Zhejiang Chinese Medical University, Hangzhou, 310035, Zhejiang, China.

Dian Wang (D)

School of Clinical Medicine, Hangzhou Normal University, Hangzhou, 310015, Zhejiang, China.

Jie Wang (J)

Department of Hepatology, The Affiliated Hospital of Hangzhou Normal University, No. 126 Wenzhou Road, Gongshu District, Hangzhou, 310015, Zhejiang, China.

Ke Zhao (K)

School of Basic Medical Sciences, Hangzhou Normal University, Hangzhou, 311121, Zhejiang, China.

Jing Liu (J)

Department of Hepatology, The Affiliated Hospital of Hangzhou Normal University, No. 126 Wenzhou Road, Gongshu District, Hangzhou, 310015, Zhejiang, China. 20181580@hznu.edu.cn.

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