Accelerated waning of immune responses to a third COVID-19 vaccination in patients with immune-mediated inflammatory diseases.


Journal

Journal of autoimmunity
ISSN: 1095-9157
Titre abrégé: J Autoimmun
Pays: England
ID NLM: 8812164

Informations de publication

Date de publication:
02 2023
Historique:
received: 24 11 2022
accepted: 11 12 2022
pubmed: 28 1 2023
medline: 3 3 2023
entrez: 27 1 2023
Statut: ppublish

Résumé

A 3 50 patients under immunosuppression and 42 healthy controls (HCs) received a 3 At week 12, reduced anti-RBD antibody levels were observed in IMID patients as compared to HCs (median antibody level 5345 BAU/ml [1781-10,208] versus 9650 BAU/ml [6633-16,050], p < 0.001). Reduction in relative antibody levels was significantly higher in IMID patients as compared to HCs at week 12 (p < 0.001). Lowest anti-RBD antibody levels were detected in IMID patients who received biological disease-modifying anti-rheumatic drugs (DMARDs) or a combination therapy with conventional synthetic and biological DMARDs. Number of SARS-CoV-2-specific T cells against wildtype and Omicron variants remained stable over 12 weeks in IMID patients. No serious adverse events were reported. Due to a fast decline in anti-RBD antibodies in IMID patients an early 4

Sections du résumé

BACKGROUND
A 3
METHODS
50 patients under immunosuppression and 42 healthy controls (HCs) received a 3
RESULTS
At week 12, reduced anti-RBD antibody levels were observed in IMID patients as compared to HCs (median antibody level 5345 BAU/ml [1781-10,208] versus 9650 BAU/ml [6633-16,050], p < 0.001). Reduction in relative antibody levels was significantly higher in IMID patients as compared to HCs at week 12 (p < 0.001). Lowest anti-RBD antibody levels were detected in IMID patients who received biological disease-modifying anti-rheumatic drugs (DMARDs) or a combination therapy with conventional synthetic and biological DMARDs. Number of SARS-CoV-2-specific T cells against wildtype and Omicron variants remained stable over 12 weeks in IMID patients. No serious adverse events were reported.
CONCLUSION
Due to a fast decline in anti-RBD antibodies in IMID patients an early 4

Identifiants

pubmed: 36706534
pii: S0896-8411(22)00189-5
doi: 10.1016/j.jaut.2022.102981
pmc: PMC9771756
pii:
doi:

Substances chimiques

COVID-19 Vaccines 0
Spike Glycoprotein, Coronavirus 0
Antibodies 0
Antirheumatic Agents 0
Antibodies, Viral 0
spike protein, SARS-CoV-2 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102981

Informations de copyright

Copyright © 2022 The Authors. Published by Elsevier Ltd.. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest DM reports support for meeting attendances from Pfizer and consultation fees from AstraZeneca; JS reports about grants, consulting and personal fees from AbbVie, Amgen, AstraZeneca, Astro, Bristol-Myers Squibb, Celltrion, Gilead-Galapagos, Janssen, Lilly, Pfizer, R-Pharma, Samsung, Sanofi, Chugai, Merck Sharp & Dohme, Novartis-Sandoz Roche, Samsung and UCB and grants from Abbvie, AstraZeneca, Lilly, Novartis, and Roche; HR received speaker fees from Gilead, Merck Sharp and Pfizer and travel support from Janssen; HH received grants from Glock Health, BlueSky Immunotherapies and Neutrolis; DA received grants, speaker fees, or consultancy fees from Abbvie, Amgen, Galapagos, Lilly, Janssen, Merck, Novartis, Pfizer, Sandoz, and Sanofi; RK reports consulting fees from AstraZeneca, Takeda Pharma, MEDahead and Janssen Cilag and speaker fees from Otsuka; ES received travel support from Pfizer, Bristol-Myers Squibb and Boehringer-Ingelheim and speaker fees from Lilly; MB reports about personal fees from Eli-Lilly. All other authors declare no competing interests.

Auteurs

Daniel Mrak (D)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Felix Kartnig (F)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Daniela Sieghart (D)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Elisabeth Simader (E)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Helga Radner (H)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Peter Mandl (P)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Lisa Göschl (L)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Philipp Hofer (P)

Department of Pathology, Medical University of Vienna, Vienna, Austria.

Thomas Deimel (T)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Irina Gessl (I)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Renate Kain (R)

Department of Pathology, Medical University of Vienna, Vienna, Austria.

Stefan Winkler (S)

Division of Infectious Diseases and Tropical Medicine, Department of Internal Medicine I, Medical University of Vienna, Austria.

Josef S Smolen (JS)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Thomas Perkmann (T)

Department of Laboratory Medicine, Medical University of Vienna, Austria.

Helmuth Haslacher (H)

Department of Laboratory Medicine, Medical University of Vienna, Austria.

Daniel Aletaha (D)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria.

Leonhard X Heinz (LX)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria. Electronic address: leonhard.heinz@meduniwien.ac.at.

Michael Bonelli (M)

Division of Rheumatology, Department of Internal Medicine III, Medical University of Vienna, Austria. Electronic address: michael.bonelli@meduniwien.ac.at.

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Classifications MeSH