Exploring new therapeutic potentials of curcumin against post-surgical adhesion bands.

Integrative medicine Peritendinous adhesion Peritoneal fibrosis Phytosomal curcumin Post-surgical adhesion bands

Journal

BMC complementary medicine and therapies
ISSN: 2662-7671
Titre abrégé: BMC Complement Med Ther
Pays: England
ID NLM: 101761232

Informations de publication

Date de publication:
31 Jan 2023
Historique:
received: 25 08 2022
accepted: 23 11 2022
entrez: 1 2 2023
pubmed: 2 2 2023
medline: 3 2 2023
Statut: epublish

Résumé

Adhesion band formation is a common cause of morbidity for patients undergoing surgeries. Anti-inflammatory and anti-fibrotic properties of curcumin, a pharmacologically active component of Curcuma longa, have been investigated in several studies. The aim of this study is to explore the therapeutic potential of curcumin in attenuating post-operative adhesion band (PSAB) formation in both peritoneal and peritendinous surgeries in animal models. Bio-mechanical, histological and quantitative evaluation of inflammation, and total fibrosis scores were graded and measured in the presence and absence of phytosomal curcumin. Results showed that phytosomal curcumin significantly decreased severity, length, density and tolerance of mobility of peritendinous adhesions as well as incidence and severity of abdominal fibrotic bands post-surgery. Curcumin may decrease inflammation by attenuating recruitment of inflammatory cells and regulating oxidant/anti-oxidant balance in post-operative tissue samples. Moreover, markedly lower fibrosis scores were obtained in the adhesive tissues of phytosomal curcumin-treated groups which correlated with a significant decrease in quantity, quality and grading of fibers, and collagen deposition in animal models. These results suggest that protective effects of phytosomal curcumin against PSAB formation is partially mediated by decreasing inflammation and fibrosis at site of surgery. Further studies are needed to investigate the therapeutic potential of this molecule in preventing PSAB.

Sections du résumé

BACKGROUND BACKGROUND
Adhesion band formation is a common cause of morbidity for patients undergoing surgeries. Anti-inflammatory and anti-fibrotic properties of curcumin, a pharmacologically active component of Curcuma longa, have been investigated in several studies. The aim of this study is to explore the therapeutic potential of curcumin in attenuating post-operative adhesion band (PSAB) formation in both peritoneal and peritendinous surgeries in animal models.
METHODS METHODS
Bio-mechanical, histological and quantitative evaluation of inflammation, and total fibrosis scores were graded and measured in the presence and absence of phytosomal curcumin.
RESULTS RESULTS
Results showed that phytosomal curcumin significantly decreased severity, length, density and tolerance of mobility of peritendinous adhesions as well as incidence and severity of abdominal fibrotic bands post-surgery. Curcumin may decrease inflammation by attenuating recruitment of inflammatory cells and regulating oxidant/anti-oxidant balance in post-operative tissue samples. Moreover, markedly lower fibrosis scores were obtained in the adhesive tissues of phytosomal curcumin-treated groups which correlated with a significant decrease in quantity, quality and grading of fibers, and collagen deposition in animal models.
CONCLUSION CONCLUSIONS
These results suggest that protective effects of phytosomal curcumin against PSAB formation is partially mediated by decreasing inflammation and fibrosis at site of surgery. Further studies are needed to investigate the therapeutic potential of this molecule in preventing PSAB.

Identifiants

pubmed: 36721147
doi: 10.1186/s12906-022-03808-6
pii: 10.1186/s12906-022-03808-6
pmc: PMC9887929
doi:

Substances chimiques

Curcumin IT942ZTH98

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

27

Informations de copyright

© 2023. The Author(s).

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Auteurs

Mohammad-Mostafa Askarnia-Faal (MM)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Sayyed-Hadi Sayyed-Hosseinian (SH)

Orthopedic Research Center, Shahid Kamyab Hospital, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyedeh Elnaz Nazari (SE)

Department of Medical Physiology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Fereshteh Asgharzadeh (F)

Department of Medical Physiology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Ehsan Vahedi (E)

Orthopedic Research Center, Shahid Kamyab Hospital, Mashhad University of Medical Sciences, Mashhad, Iran.

Moein Eskandari (M)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Haniyeh Ghasemi (H)

Department of Medical Physiology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Amir Avan (A)

Metabolic syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Human Genetics, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Maryam Alaei (M)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Hamideh Naimi (H)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Maryam Daghiani (M)

Department of Physiotherapy, School of Paramedical Sciences, Mashhad University of Medical Sciences, Mashhad, Iran.

Atena Soleimani (A)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Abbas Alalikhan (A)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Reza Mohammadzadeh (R)

Department of Biology, Faculty of Basic Sciences, University of Maragheh, Maragheh, Iran.

Gordon Ferns (G)

Division of Medical Education, Brighton & Sussex Medical School, Falmer, Brighton, Sussex, BN1 9PH, UK.

Mikhail Ryzhikov (M)

Saint Louis University, School of Medicine, Saint Louis, MO, USA.

Majid Khazaei (M)

Department of Medical Physiology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran. KhazaeiM@mums.ac.ir.
Metabolic syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. KhazaeiM@mums.ac.ir.

Seyed Mahdi Hassanian (SM)

Department of Clinical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran. hasanianmehrm@mums.ac.ir.
Metabolic syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran. hasanianmehrm@mums.ac.ir.

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Classifications MeSH