Prognostic Role of Biologically Active Volume of Disease in Patients With Metastatic Lung Adenocarcinoma.


Journal

Clinical lung cancer
ISSN: 1938-0690
Titre abrégé: Clin Lung Cancer
Pays: United States
ID NLM: 100893225

Informations de publication

Date de publication:
05 2023
Historique:
received: 22 11 2022
revised: 11 01 2023
accepted: 12 01 2023
medline: 1 5 2023
pubmed: 10 2 2023
entrez: 9 2 2023
Statut: ppublish

Résumé

Number of metastatic sites can identify patient populations with non-small cell lung cancer (NSCLC) that benefit from aggressive therapy. Total volume of disease is also relevant. We evaluated the prognostic impact of biologically active volume of disease (BaVD) on patients with metastatic lung adenocarcinoma. Positron emission tomography/computerized tomography (PET/CT) scans from patients with newly diagnosed lung adenocarcinoma prior to starting any therapy were identified. SUV thresholds of 3 and 4 were used to auto-contour all FDG avid areas. Kaplan-Meier analysis and Cox regression were performed to examine influence on OS. One hundred forty-eight patients were included in the analysis. The median BaVD when using an SUV threshold of 3 was 122.8 mL. The median BaVD when using an SUV threshold of 4 was 46.2 mL When stratified by median BaVD using an SUV of 3, median OS was higher for patients with <=122.8 mL (2.12 years) compared to patients with >122.8 mL (1.46 years) (log-rank P = .001). Similarly, when stratified by median BaVD using an SUV of 4, median OS was higher for patients with <=46.2 mL (1.91 years; 95% CI: 1.65-3.22 years) compared to patients with >46.2 mL (1.48 years; 95% CI: 1.07-1.80 years) (log-rank P = .007). On multivariable analysis, BaVD was significantly associated with OS when using an SUV threshold of 3 (HR: 20.169, P < .001) and 4 (HR: 4.117, P < .001). BaVD is an important prognostic factor in metastatic lung adenocarcinoma and may aid identification of patients with limited disease who may be candidates for more aggressive therapies.

Sections du résumé

BACKGROUND
Number of metastatic sites can identify patient populations with non-small cell lung cancer (NSCLC) that benefit from aggressive therapy. Total volume of disease is also relevant. We evaluated the prognostic impact of biologically active volume of disease (BaVD) on patients with metastatic lung adenocarcinoma.
MATERIALS AND METHODS
Positron emission tomography/computerized tomography (PET/CT) scans from patients with newly diagnosed lung adenocarcinoma prior to starting any therapy were identified. SUV thresholds of 3 and 4 were used to auto-contour all FDG avid areas. Kaplan-Meier analysis and Cox regression were performed to examine influence on OS.
RESULTS
One hundred forty-eight patients were included in the analysis. The median BaVD when using an SUV threshold of 3 was 122.8 mL. The median BaVD when using an SUV threshold of 4 was 46.2 mL When stratified by median BaVD using an SUV of 3, median OS was higher for patients with <=122.8 mL (2.12 years) compared to patients with >122.8 mL (1.46 years) (log-rank P = .001). Similarly, when stratified by median BaVD using an SUV of 4, median OS was higher for patients with <=46.2 mL (1.91 years; 95% CI: 1.65-3.22 years) compared to patients with >46.2 mL (1.48 years; 95% CI: 1.07-1.80 years) (log-rank P = .007). On multivariable analysis, BaVD was significantly associated with OS when using an SUV threshold of 3 (HR: 20.169, P < .001) and 4 (HR: 4.117, P < .001).
CONCLUSION
BaVD is an important prognostic factor in metastatic lung adenocarcinoma and may aid identification of patients with limited disease who may be candidates for more aggressive therapies.

Identifiants

pubmed: 36759265
pii: S1525-7304(23)00003-7
doi: 10.1016/j.cllc.2023.01.001
pii:
doi:

Substances chimiques

Fluorodeoxyglucose F18 0Z5B2CJX4D
Radiopharmaceuticals 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

244-251

Informations de copyright

Copyright © 2023 Elsevier Inc. All rights reserved.

Auteurs

Colton Ladbury (C)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Tariq Abuali (T)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Jason Liu (J)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

William Watkins (W)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Dongsu Du (D)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Erminia Massarelli (E)

Department of Medical Oncology and Therapeutics Research, City of Hope National Medical Center, Duarte, CA.

Victoria Villaflor (V)

Department of Medical Oncology and Therapeutics Research, City of Hope National Medical Center, Duarte, CA.

An Liu (A)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Ravi Salgia (R)

Department of Medical Oncology and Therapeutics Research, City of Hope National Medical Center, Duarte, CA.

Terence Williams (T)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Scott Glaser (S)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

Arya Amini (A)

Department of Radiation Oncology, City of Hope National Medical Center, Duarte, CA.

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