The Role of the Interleukin-1 Family in Complications of Prematurity.
IL-1
IL-18
IL-1Ra
IL-33
IL-36
IL-36Ra
IL-37
IL-38
bronchopulmonary dysplasia
cerebral palsy
inflammation
necrotizing enterocolitis
prematurity
pulmonary hypertension
retinopathy of prematurity
sepsis
white matter injury
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
01 Feb 2023
01 Feb 2023
Historique:
received:
30
12
2022
revised:
20
01
2023
accepted:
22
01
2023
entrez:
11
2
2023
pubmed:
12
2
2023
medline:
15
2
2023
Statut:
epublish
Résumé
Preterm birth is a major contributor to neonatal morbidity and mortality. Complications of prematurity such as bronchopulmonary dysplasia (BPD, affecting the lung), pulmonary hypertension associated with BPD (BPD-PH, heart), white matter injury (WMI, brain), retinopathy of prematurity (ROP, eyes), necrotizing enterocolitis (NEC, gut) and sepsis are among the major causes of long-term morbidity in infants born prematurely. Though the origins are multifactorial, inflammation and in particular the imbalance of pro- and anti-inflammatory mediators is now recognized as a key driver of the pathophysiology underlying these illnesses. Here, we review the involvement of the interleukin (IL)-1 family in perinatal inflammation and its clinical implications, with a focus on the potential of these cytokines as therapeutic targets for the development of safe and effective treatments for early life inflammatory diseases.
Identifiants
pubmed: 36769133
pii: ijms24032795
doi: 10.3390/ijms24032795
pmc: PMC9918069
pii:
doi:
Substances chimiques
Interleukin-1
0
Anti-Inflammatory Agents
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Cerebral Palsy Alliance
ID : PHD01821
Organisme : Monash University
ID : Graduate scholarship
Organisme : National Health and Medical Research Council
ID : 1173584
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pubmed: 30995480