Nanobody-mediated complement activation to kill HIV-infected cells.


Journal

EMBO molecular medicine
ISSN: 1757-4684
Titre abrégé: EMBO Mol Med
Pays: England
ID NLM: 101487380

Informations de publication

Date de publication:
11 04 2023
Historique:
revised: 02 02 2023
received: 08 06 2022
accepted: 03 02 2023
medline: 12 4 2023
pubmed: 18 2 2023
entrez: 17 2 2023
Statut: ppublish

Résumé

The complement system which is part of the innate immune response against invading pathogens represents a powerful mechanism for killing of infected cells. Utilizing direct complement recruitment for complement-mediated elimination of HIV-1-infected cells is underexplored. We developed a novel therapeutic modality to direct complement activity to the surface of HIV-1-infected cells. This bispecific complement engager (BiCE) is comprised of a nanobody recruiting the complement-initiating protein C1q, and single-chain variable fragments of broadly neutralizing antibodies (bNAbs) targeting the HIV-1 envelope (Env) protein. Here, we show that two anti-HIV BiCEs targeting the V3 loop and the CD4 binding site, respectively, increase C3 deposition and mediate complement-dependent cytotoxicity (CDC) of HIV-1 Env-expressing Raji cells. Furthermore, anti-HIV BiCEs trigger complement activation on primary CD4 T cells infected with laboratory-adapted HIV-1 strain and facilitates elimination of HIV-1-infected cells over time. In summary, we present a novel approach to direct complement deposition to the surface of HIV-1-infected cells leading to complement-mediated killing of these cells.

Identifiants

pubmed: 36799046
doi: 10.15252/emmm.202216422
pmc: PMC10086584
doi:

Substances chimiques

Antibodies, Neutralizing 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e16422

Subventions

Organisme : Danmarks Frie Forskningsfond
ID : 9039-00039B
Organisme : Danmarks Frie Forskningsfond
ID : 1029-00004B
Organisme : Novo Nordisk Fonden
ID : NNF19OC0054577

Informations de copyright

© 2023 The Authors. Published under the terms of the CC BY 4.0 license.

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Auteurs

Maria Lange Pedersen (ML)

Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.

Dennis Vestergaard Pedersen (DV)

Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark.

Mikael Becher Lykkegaard Winkler (MBL)

Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark.

Heidi Gytz Olesen (HG)

Department of Molecular Biology and Genetics, Aarhus University, Aarhus, Denmark.

Ole Schmeltz Søgaard (OS)

Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

Lars Østergaard (L)

Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

Nick Stub Laursen (NS)

Department of Biomedicine, Aarhus University, Aarhus, Denmark.

Anna Halling Folkmar Rahimic (AHF)

Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.

Martin Tolstrup (M)

Department of Clinical Medicine, Aarhus University, Aarhus, Denmark.
Department of Infectious Diseases, Aarhus University Hospital, Aarhus, Denmark.

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