The effect of miR-372-5p regulation on CDX1 and CDX2 in the gastric cancer cell line.


Journal

Hormone molecular biology and clinical investigation
ISSN: 1868-1891
Titre abrégé: Horm Mol Biol Clin Investig
Pays: Germany
ID NLM: 101538885

Informations de publication

Date de publication:
01 Sep 2023
Historique:
received: 08 05 2022
accepted: 08 02 2023
medline: 2 10 2023
pubmed: 28 2 2023
entrez: 27 2 2023
Statut: epublish

Résumé

MicroRNA expression disruptions play an important function in the expansion of gastric cancer. Previous investigation has indicated that miR-372-5p doing as an oncogene in several malignancies. CDX1 and CDX2, as target genes of miR-372-5p, play the role of tumor suppressors and oncogenes in gastric cancer cells, respectively. The current investigation explored the effects of miR-372-5p regulation on CDX2 and CDX1 in AGS cell lines and studied their molecular mechanism. hsa-miR-372-5p miRCURY LNA miRNA Inhibitors and Mimic were transfected into AGS cell line. The cell viability and cell cycle calculation were defined by MTT assay and flow cytometry, respectively. The Expression levels of miR-372-5p, CDX1, CDX2 and transfection efficiency were measured using Real-time PCR. Statistical investigation p values <0.05 were considered to be meaningful. miR-372-5p particularly was upregulated in control cells and also after transfection by mimic. While its expression was reduced by the inhibitor. Upregulation of miR-372-5p remarkably increased cell growth and led to accumulation in the G2/M phase, although the inhibitor decreased cell growth and accumulation in the S phase. Accordingly, upregulation of miR-372-5p increased CDX2 and decreased CDX1 expression. By inhibition of miR-372-5p, expression of CDX2 was decreased and expression of CDX1 was increased. Up and down-regulation of miR-372-5P has a potential effect on the expression levels of its target genes, CDX1 and CDX22. Accordingly, the downregulation of miR-372-5p may be assumed as a possible therapeutic target in treating gastric cancer.

Identifiants

pubmed: 36848481
pii: hmbci-2022-0045
doi: 10.1515/hmbci-2022-0045
doi:

Substances chimiques

CDX1 protein, human 0
CDX2 protein, human 0
CDX2 Transcription Factor 0
Homeodomain Proteins 0
MicroRNAs 0
MIRN372 microRNA, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

271-276

Informations de copyright

© 2023 Walter de Gruyter GmbH, Berlin/Boston.

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Auteurs

Elaheh Asghari Gharakhyli (EA)

Department of Microbiology, Gorgan Branch, Islamic Azad University, Gorgan, Iran.

Agheel Tabar Molla Hassan (A)

Department of Cell and Molecular Biology, Babol Branch, Islamic Azad University, Babol, Iran.

Majid Alipour (M)

Department of Cell and Molecular Biology, Babol Branch, Islamic Azad University, Babol, Iran.

Sogand Vahidi (S)

Medical Biology Research Center, Kermanshah University of Medical Sciences, Kermanshah, Iran.

Ali Akbar Samadani (AA)

Guilan Road Trauma Research Center, Guilan University of Medical Sciences, Rasht, Iran.

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