Microenvironmental elements singularity synergistically regulate the behavior and chemosensitivity of endometrioid carcinoma.


Journal

Human cell
ISSN: 1749-0774
Titre abrégé: Hum Cell
Pays: Japan
ID NLM: 8912329

Informations de publication

Date de publication:
May 2023
Historique:
received: 27 12 2022
accepted: 23 02 2023
medline: 19 4 2023
pubmed: 1 3 2023
entrez: 28 2 2023
Statut: ppublish

Résumé

The importance of the microenvironment is widely recognized as it regulates not only malignant cell behavior but also drug sensitivity. The cancer cell microenvironment is composed of biological, physical and chemical elements, and simultaneous reproduction of these three elements are important conditions investigated in cancer research. In the present study, we focused on the epidemiological and anatomical specificities of endometrioid carcinoma, obesity (biological), fluid flow (physical) and anticancer agents (chemical) to target the specific microenvironmental elements of endometrioid carcinoma. To elucidate the individual effects of these elements on endometrioid carcinoma and to investigate the relationships between these factors, we developed an adipose tissue fragments (ATFs)-embedded cell disc under a rotational culture method to generate carcinoma-stroma interactions and to create fluid flow. ATFs and fluid flow individually or synergistically influenced proliferative cellular behavior and the morphological changes underlying endometrioid carcinoma. ATFs and fluid flow also governed the expression of extracellular signal-regulated kinase and p38 signaling synergistically or individually, depending on the endometrioid carcinoma cell type. Adipose tissue induced chemoresistance to cis-diamminedichloro-platinum (CDDP) in endometrioid cancer, but the resistance effect was abolished by fluid flow. Thus, a simple reconstructed model was established to investigate three elements of the microenvironment of endometrioid carcinoma in vitro. This culture model unequivocally demonstrated the individual and synergistic effects of the three elements on endometrioid carcinoma. This new culture model is a promising tool for elucidating the mechanisms underlying endometrioid carcinoma and for developing further treatment strategies.

Identifiants

pubmed: 36853404
doi: 10.1007/s13577-023-00886-7
pii: 10.1007/s13577-023-00886-7
doi:

Substances chimiques

Antineoplastic Agents 0
Cisplatin Q20Q21Q62J

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1147-1159

Subventions

Organisme : JSPS KAKENHI
ID : 19K18468
Organisme : JSPS KAKENHI
ID : 21K16773

Informations de copyright

© 2023. The Author(s) under exclusive licence to Japan Human Cell Society.

Références

World Health Organization. Female genital tumours 5th edn. Lyon, France. International Agency for Research on Cancer. Geneva, Switzerland: Print copies are distributed by WHO Press; 2020.
Clement PB, Young RH. Endometrioid carcinoma of the uterine corpus: a review of its pathology with emphasis on recent advances and problematic aspects. Adv Anat Pathol. 2002;9:145–84.
doi: 10.1097/00125480-200205000-00001 pubmed: 11981113
Nevadunsky NS, Van Arsdale A, Strickler HD, et al. Obesity and age at diagnosis of endometrial cancer. Obstet Gynecol. 2014;124:300–6.
doi: 10.1097/AOG.0000000000000381 pubmed: 25004350
Feng Y-H. The association between obesity and gynecological cancer. Gynecol Minim Invasive Ther. 2015;4:102–5.
doi: 10.1016/j.gmit.2015.03.003
Unamuno X, Gómez-Ambrosi J, Rodríguez A, Becerril S, Frühbeck G, Catalán V. Adipokine dysregulation and adipose tissue inflammation in human obesity. Eur J Clin Invest. 2018;48: e12997.
doi: 10.1111/eci.12997 pubmed: 29995306
Matsuzawa Y. Adiponectin: a key player in obesity related disorders. Curr Pharm Des. 2010;16:1896–901.
doi: 10.2174/138161210791208893 pubmed: 20370675
Frühbeck G, Catalán V, Rodríguez A, Gómez-Ambrosi J. Adiponectin-leptin ratio: a promising index to estimate adipose tissue dysfunction. Relation with obesity-associated cardiometabolic risk. Adipocyte. 2018;7:57–62.
doi: 10.1080/21623945.2017.1402151 pubmed: 29205099
Prieto-Hontoria PL, Pérez-Matute P, Fernández-Galilea M, Bustos M, Martínez JA, Moreno-Aliaga MJ. Role of obesity-associated dysfunctional adipose tissue in cancer: a molecular nutrition approach. Biochim Biophys Acta (BBA) Bioenerg. 2011;1807:664–78.
doi: 10.1016/j.bbabio.2010.11.004
Balkwill FR, Capasso M, Hagemann T. The tumor microenvironment at a glance. J Cell Sci. 2012;125:5591–6.
doi: 10.1242/jcs.116392 pubmed: 23420197
Hinshaw DC, Shevde LA. The tumor microenvironment innately modulates cancer progression. Cancer Res. 2019;79:4557–66.
doi: 10.1158/0008-5472.CAN-18-3962 pubmed: 31350295 pmcid: 6744958
Wu T, Dai Y. Tumor microenvironment and therapeutic response. Cancer Lett. 2017;387:61–8.
doi: 10.1016/j.canlet.2016.01.043 pubmed: 26845449
Senthebane DA, Rowe A, Thomford NE, et al. The role of tumor microenvironment in chemoresistance: to survive, keep your enemies closer. Int J Mol Sci. 2017;18:1586.
doi: 10.3390/ijms18071586 pubmed: 28754000 pmcid: 5536073
Maia J, Caja S, Strano Moraes MC, Couto N, Costa-Silva B. Exosome-based cell-cell communication in the tumor microenvironment. Front Cell Dev Biol. 2018;6:18.
doi: 10.3389/fcell.2018.00018 pubmed: 29515996 pmcid: 5826063
Komohara Y, Takeya M. CAFs and TAMs: maestros of the tumour microenvironment. J Pathol. 2017;241:313–5.
doi: 10.1002/path.4824 pubmed: 27753093
Mitchell MJ, King MR. Computational and experimental models of cancer cell response to fluid shear stress. Front Oncol. 2013;3:44.
doi: 10.3389/fonc.2013.00044 pubmed: 23467856 pmcid: 3587800
Repasky EA, Evans SS, Dewhirst MW. Temperature matters! And why it should matter to tumor immunologists. Cancer Immunol Res. 2013;1:210–6.
doi: 10.1158/2326-6066.CIR-13-0118 pubmed: 24490177 pmcid: 3904378
Trédan O, Galmarini CM, Patel K, Tannock IF. Drug resistance and the solid tumor microenvironment. J Natl Cancer Inst. 2007;99:1441–54.
doi: 10.1093/jnci/djm135 pubmed: 17895480
Landskron G, De la Fuente M, Thuwajit P, Thuwajit C, Hermoso MA. Chronic inflammation and cytokines in the tumor microenvironment. J Immunol Res. 2014;2014:149185.
doi: 10.1155/2014/149185 pubmed: 24901008 pmcid: 4036716
Matsumoto M, Yamaguchi Y, Seino Y, et al. Estrogen signaling ability in human endometrial cancer through the cancer–stromal interaction. Endocr Relat Cancer. 2008;15:451–63.
doi: 10.1677/ERC-07-0227 pubmed: 18508998
Arnold JT, Lessey BA, Seppälä M, Kaufman DG. Effect of normal endometrial stroma on growth and differentiation in Ishikawa endometrial adenocarcinoma cells. Can Res. 2002;62:79–88.
Mescher AL. Junqueira’s basic histology: text and atlas, Sixteenth edition, 50th anniversary edition. New York: McGraw-Hill; 2021.
Kim SW, Ehrman J, Ahn MR, et al. Shear stress induces noncanonical autophagy in intestinal epithelial monolayers. Mol Biol Cell. 2017;28:3043–56.
doi: 10.1091/mbc.e17-01-0021 pubmed: 28855375 pmcid: 5662261
Lai SK, Wang Y-Y, Wirtz D, Hanes J. Micro-and macrorheology of mucus. Adv Drug Deliv Rev. 2009;61:86–100.
doi: 10.1016/j.addr.2008.09.012 pubmed: 19166889 pmcid: 2736374
Van Wijk F, Van der Burg M, Burger CW, Vergote I, van Doorn HC. Management of recurrent endometrioid endometrial carcinoma: an overview. Int J Gynecol Cancer. 2009;19:314–20.
doi: 10.1111/IGC.0b013e3181a7f71e pubmed: 19407552
Nagase K, Akutagawa T, Rikitake-Yamamoto M, et al. Cellular and physical microenvironments regulate the aggressiveness and sunitinib chemosensitivity of clear cell renal cell carcinoma. J Pathol. 2021;254:46–56.
pubmed: 33512712 pmcid: 8248239
Aoki S, Toda S, Ando T, Sugihara H. Bone marrow stromal cells, preadipocytes, and dermal fibroblasts promote epidermal regeneration in their distinctive fashions. Mol Biol Cell. 2004;15:4647–57.
doi: 10.1091/mbc.e04-01-0038 pubmed: 15292451 pmcid: 519156
Aoki S, Makino J, Nagashima A, et al. Fluid flow stress affects peritoneal cell kinetics: possible pathogenesis of peritoneal fibrosis. Perit Dial Int. 2011;31:466–76.
doi: 10.3747/pdi.2010.00157 pubmed: 21532005
Donohoe F, Wilkinson M, Baxter E, Brennan DJ. Mitogen-activated protein kinase (MAPK) and obesity-related cancer. Int J Mol Sci. 2020;21:1241.
doi: 10.3390/ijms21041241 pubmed: 32069845 pmcid: 7072904
Wilson J, Balkwill F. The role of cytokines in the epithelial cancer microenvironment. In: Seminars in cancer biology. Elsevier; 2002. p. 113–20.
Kawata K, Aoki S, Futamata M, et al. Mesenchymal cells and fluid flow stimulation synergistically regulate the kinetics of corneal epithelial cells at the air–liquid interface. Graefes Arch Clin Exp Ophthalmol. 2019;257:1915–24.
doi: 10.1007/s00417-019-04422-y pubmed: 31321523
Makker A, Goel MM, Das V, Agarwal A. PI3K-Akt-mTOR and MAPK signaling pathways in polycystic ovarian syndrome, uterine leiomyomas and endometriosis: an update. Gynecol Endocrinol. 2012;28:175–81.
doi: 10.3109/09513590.2011.583955 pubmed: 21916800
Chen J. Multiple signal pathways in obesity-associated cancer. Obes Rev. 2011;12:1063–70.
doi: 10.1111/j.1467-789X.2011.00917.x pubmed: 22093240
Liu F, Yang X, Geng M, Huang M. Targeting ERK, an Achilles’ Heel of the MAPK pathway, in cancer therapy. Acta Pharm Sin B. 2018;8:552–62.
doi: 10.1016/j.apsb.2018.01.008 pubmed: 30109180 pmcid: 6089851
Housman G, Byler S, Heerboth S, et al. Drug resistance in cancer: an overview. Cancers. 2014;6:1769–92.
doi: 10.3390/cancers6031769 pubmed: 25198391 pmcid: 4190567
Tsuruo T, Naito M, Tomida A, et al. Molecular targeting therapy of cancer: drug resistance, apoptosis and survival signal. Cancer Sci. 2003;94:15–21.
doi: 10.1111/j.1349-7006.2003.tb01345.x pubmed: 12708468
McCubrey JA, Steelman LS, Kempf CR, et al. Therapeutic resistance resulting from mutations in Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR signaling pathways. J Cell Physiol. 2011;226:2762–81.
doi: 10.1002/jcp.22647 pubmed: 21302297
Ramakrishnan VM, Boyd NL. The adipose stromal vascular fraction as a complex cellular source for tissue engineering applications. Tissue Eng Part B Rev. 2018;24:289–99.
doi: 10.1089/ten.teb.2017.0061 pubmed: 28316259 pmcid: 6080106
Wang T, Sharma AK, Wolfrum C. Novel insights into adipose tissue heterogeneity. Rev Endocr Metab Disord. 2022;23:5–12.
doi: 10.1007/s11154-021-09703-8 pubmed: 34935088
Lihn A, Pedersen SB, Richelsen B. Adiponectin: action, regulation and association to insulin sensitivity. Obes Rev. 2005;6:13–21.
doi: 10.1111/j.1467-789X.2005.00159.x pubmed: 15655035
Gregoire FM, Smas CM, Sul HS. Understanding adipocyte differentiation. Physiol Rev. 1998;78:783–809.
doi: 10.1152/physrev.1998.78.3.783 pubmed: 9674695
Aoki S, Toda S, Sakemi T, Sugihara H. Coculture of endothelial cells and mature adipocytes actively promotes immature preadipocyte development in vitro. Cell Struct Funct. 2003;28:55–60.
doi: 10.1247/csf.28.55 pubmed: 12655151

Auteurs

Sayuri Morito (S)

Division of Pathology, Department of Pathology and Microbiology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, Saga, 849-8501, Japan.

Maki Kawasaki (M)

Department of Urology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, 849-8501, Japan.

Megumi Nishiyama (M)

Division of Pathology, Department of Pathology and Microbiology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, Saga, 849-8501, Japan.

Takehisa Sakumoto (T)

Division of Pathology, Department of Pathology and Microbiology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, Saga, 849-8501, Japan.

Mariko Hashiguchi (M)

Division of Pathology, Department of Pathology and Microbiology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, Saga, 849-8501, Japan.

Takayuki Narita (T)

Department of Chemistry and Applied Chemistry, Faculty of Science and Engineering, Saga University, 1 Honjo, Saga, 840-8502, Japan.

Atsushi Kawaguchi (A)

Education and Research Center for Community Medicine, Faculty of Medicine, Saga University, Saga, 849-8501, Japan.

Shuji Toda (S)

Department of Pathology, Takagi Hospital, Okawa, Fukuoka, 831-8501, Japan.

Shigehisa Aoki (S)

Division of Pathology, Department of Pathology and Microbiology, Faculty of Medicine, Saga University, 5-1-1 Nabeshima, Saga, Saga, 849-8501, Japan. aokis@cc.saga-u.ac.jp.

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