Coronary Inflammation and Plaque Vulnerability: A Coronary Computed Tomography and Optical Coherence Tomography Study.


Journal

Circulation. Cardiovascular imaging
ISSN: 1942-0080
Titre abrégé: Circ Cardiovasc Imaging
Pays: United States
ID NLM: 101479935

Informations de publication

Date de publication:
03 2023
Historique:
pubmed: 4 3 2023
medline: 24 3 2023
entrez: 3 3 2023
Statut: ppublish

Résumé

Vascular inflammation plays a key role in atherogenesis and in the development of acute coronary syndromes. Coronary inflammation can be measured by peri-coronary adipose tissue (PCAT) attenuation on computed tomography angiography. We examined the relationships between the level of coronary artery inflammation assessed by PCAT attenuation and coronary plaque characteristics by optical coherence tomography. A total of 474 patients (198 acute coronary syndromes and 276 stable angina pectoris) who underwent preintervention coronary computed tomography angiography and optical coherence tomography were included. To compare the relationships between the level of coronary artery inflammation and detailed plaque characteristics, we divided the subjects into high (n=244) and low (n=230) PCAT attenuation groups using a threshold value of -70.1 Hounsfield units. The high PCAT attenuation group, compared with the low PCAT attenuation group, had more males (90.6% versus 69.6%; Optical coherence tomography features of plaque vulnerability were significantly more common in patients with high PCAT attenuation, compared with those with low PCAT attenuation. Vascular inflammation and plaque vulnerability are intimately related in patients with coronary artery disease. URL: https://www. gov; Unique identifier: NCT04523194.

Sections du résumé

BACKGROUND
Vascular inflammation plays a key role in atherogenesis and in the development of acute coronary syndromes. Coronary inflammation can be measured by peri-coronary adipose tissue (PCAT) attenuation on computed tomography angiography. We examined the relationships between the level of coronary artery inflammation assessed by PCAT attenuation and coronary plaque characteristics by optical coherence tomography.
METHODS
A total of 474 patients (198 acute coronary syndromes and 276 stable angina pectoris) who underwent preintervention coronary computed tomography angiography and optical coherence tomography were included. To compare the relationships between the level of coronary artery inflammation and detailed plaque characteristics, we divided the subjects into high (n=244) and low (n=230) PCAT attenuation groups using a threshold value of -70.1 Hounsfield units.
RESULTS
The high PCAT attenuation group, compared with the low PCAT attenuation group, had more males (90.6% versus 69.6%;
CONCLUSIONS
Optical coherence tomography features of plaque vulnerability were significantly more common in patients with high PCAT attenuation, compared with those with low PCAT attenuation. Vascular inflammation and plaque vulnerability are intimately related in patients with coronary artery disease.
REGISTRATION
URL: https://www.
CLINICALTRIALS
gov; Unique identifier: NCT04523194.

Identifiants

pubmed: 36866660
doi: 10.1161/CIRCIMAGING.122.014959
doi:

Banques de données

ClinicalTrials.gov
['NCT04523194']

Types de publication

Comparative Study Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e014959

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL151266
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL148787
Pays : United States

Auteurs

Haruhito Yuki (H)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.

Tomoyo Sugiyama (T)

Department of Cardiology, Tsuchiura Kyodo General Hospital, Ibaraki, Japan (T.S., T.K.).

Keishi Suzuki (K)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.

Daisuke Kinoshita (D)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.

Takayuki Niida (T)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.

Akihiro Nakajima (A)

Interventional Cardiology Unit, New Tokyo Hospital, Chiba, Japan (A.N., S.N.).

Makoto Araki (M)

Department of Interventional Cardiology, Tokyo Medical and Dental University, Japan (M.A.).

Damini Dey (D)

Biomedical Imaging Research Institute, Cedars-Sinai Medical Center, Los Angeles (D.D.).

Hang Lee (H)

Biostatistics Center (H.L.), Massachusetts General Hospital, Harvard Medical School, Boston.

Iris McNulty (I)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.

Sunao Nakamura (S)

Interventional Cardiology Unit, New Tokyo Hospital, Chiba, Japan (A.N., S.N.).

Tsunekazu Kakuta (T)

Department of Cardiology, Tsuchiura Kyodo General Hospital, Ibaraki, Japan (T.S., T.K.).

Ik-Kyung Jang (IK)

Cardiology Division (H.Y., K.S., D.K., T.N., I.M., I.-K.J.), Massachusetts General Hospital, Harvard Medical School, Boston.
Division of Cardiology, Kyung Hee University Hospital, Seoul, South Korea (I.-K.J.).

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Classifications MeSH