Management of infusion-related reactions (IRRs) in patients receiving amivantamab in the CHRYSALIS study.


Journal

Lung cancer (Amsterdam, Netherlands)
ISSN: 1872-8332
Titre abrégé: Lung Cancer
Pays: Ireland
ID NLM: 8800805

Informations de publication

Date de publication:
04 2023
Historique:
received: 21 11 2022
revised: 08 02 2023
accepted: 13 02 2023
medline: 31 3 2023
pubmed: 4 3 2023
entrez: 3 3 2023
Statut: ppublish

Résumé

Amivantamab, a fully humanized EGFR-MET bispecific antibody, has antitumor activity in diverse EGFR- and MET-driven non-small cell lung cancer (NSCLC) and a safety profile consistent with associated on-target activities. Infusion-related reaction(s) (IRR[s]) are reported commonly with amivantamab. We review IRR and subsequent management in amivantamab-treated patients. Patients treated with the approved dose of intravenous amivantamab (1050 mg, <80 kg; 1400 mg, ≥80 kg) in CHRYSALIS-an ongoing, phase 1 study in advanced EGFR-mutated NSCLC-were included in this analysis. IRR mitigations included split first dose (350 mg, day 1 [D1]; remainder, D2), reduced initial infusion rates with proactive infusion interruption, and steroid premedication before initial dose. For all doses, pre-infusion antihistamines and antipyretics were required. Steroids were optional after the initial dose. As of 3/30/2021, 380 patients received amivantamab. IRRs were reported in 256 (67%) patients. Signs/symptoms of IRR included chills, dyspnea, flushing, nausea, chest discomfort, and vomiting. Most of the 279 IRRs were grade 1 or 2; grade 3 and 4 IRR occurred in 7 and 1 patients, respectively. Most (90%) IRRs occurred on cycle 1, D1 (C1D1); median time-to-first-IRR onset during C1D1 was 60 min; and first-infusion IRRs did not compromise subsequent infusions. Per protocol, IRR was mitigated on C1D1 with holding of infusion (56% [214/380]), reinitiating at reduced rate (53% [202/380]), and aborting infusion (14% [53/380]). C1D2 infusions were completed in 85% (45/53) of patients who had C1D1 infusions aborted. Four patients (1% [4/380]) discontinued treatment due to IRR. In studies aimed at elucidating the underlying mechanism(s) of IRR, no pattern was observed between patients with versus without IRR. IRRs with amivantamab were predominantly low grade and limited to first infusion, and rarely occurred with subsequent dosing. Close monitoring for IRR with the initial amivantamab dose and early intervention at first IRR signs/symptoms should be part of routine amivantamab administration.

Sections du résumé

BACKGROUND
Amivantamab, a fully humanized EGFR-MET bispecific antibody, has antitumor activity in diverse EGFR- and MET-driven non-small cell lung cancer (NSCLC) and a safety profile consistent with associated on-target activities. Infusion-related reaction(s) (IRR[s]) are reported commonly with amivantamab. We review IRR and subsequent management in amivantamab-treated patients.
METHODS
Patients treated with the approved dose of intravenous amivantamab (1050 mg, <80 kg; 1400 mg, ≥80 kg) in CHRYSALIS-an ongoing, phase 1 study in advanced EGFR-mutated NSCLC-were included in this analysis. IRR mitigations included split first dose (350 mg, day 1 [D1]; remainder, D2), reduced initial infusion rates with proactive infusion interruption, and steroid premedication before initial dose. For all doses, pre-infusion antihistamines and antipyretics were required. Steroids were optional after the initial dose.
RESULTS
As of 3/30/2021, 380 patients received amivantamab. IRRs were reported in 256 (67%) patients. Signs/symptoms of IRR included chills, dyspnea, flushing, nausea, chest discomfort, and vomiting. Most of the 279 IRRs were grade 1 or 2; grade 3 and 4 IRR occurred in 7 and 1 patients, respectively. Most (90%) IRRs occurred on cycle 1, D1 (C1D1); median time-to-first-IRR onset during C1D1 was 60 min; and first-infusion IRRs did not compromise subsequent infusions. Per protocol, IRR was mitigated on C1D1 with holding of infusion (56% [214/380]), reinitiating at reduced rate (53% [202/380]), and aborting infusion (14% [53/380]). C1D2 infusions were completed in 85% (45/53) of patients who had C1D1 infusions aborted. Four patients (1% [4/380]) discontinued treatment due to IRR. In studies aimed at elucidating the underlying mechanism(s) of IRR, no pattern was observed between patients with versus without IRR.
CONCLUSION
IRRs with amivantamab were predominantly low grade and limited to first infusion, and rarely occurred with subsequent dosing. Close monitoring for IRR with the initial amivantamab dose and early intervention at first IRR signs/symptoms should be part of routine amivantamab administration.

Identifiants

pubmed: 36868177
pii: S0169-5002(23)00069-7
doi: 10.1016/j.lungcan.2023.02.008
pii:
doi:

Substances chimiques

amivantamab-vmjw 0
Antibodies, Bispecific 0
ErbB Receptors EC 2.7.10.1

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

166-171

Informations de copyright

Copyright © 2023 Janssen Research and Development LLC. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Keunchil Park (K)

Dept of Thoracic/Head and Neck Medical Oncology, UT M.D. Anderson Cancer Center, Texas.

Joshua K Sabari (JK)

NYU School of Medicine, New York, NY, USA.

Eric B Haura (EB)

H. Lee Moffitt Cancer Center & Research Institute, Tampa, FL, USA.

Catherine A Shu (CA)

Columbia University Medical Center, New York, NY, USA.

Alexander Spira (A)

Virginia Cancer Specialists Research Institute, US Oncology Research, Fairfax, VA, USA.

Ravi Salgia (R)

City of Hope, Duarte, CA, USA.

Karen L Reckamp (KL)

Cedars-Sinai Medical Center, Los Angeles, CA, USA.

Rachel E Sanborn (RE)

Earle A. Chiles Research Institute, Providence Cancer Institute, Portland, OR, USA.

Ramaswamy Govindan (R)

Washington University School of Medicine, St. Louis, MO, USA.

Joshua M Bauml (JM)

Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA, USA.

Joshua C Curtin (JC)

Janssen R&D, Spring House, PA, USA.

John Xie (J)

Janssen R&D, Spring House, PA, USA.

Amy Roshak (A)

Janssen R&D, Spring House, PA, USA.

Patricia Lorenzini (P)

Janssen R&D, Spring House, PA, USA.

Dawn Millington (D)

Janssen R&D, Spring House, PA, USA.

Meena Thayu (M)

Janssen R&D, Spring House, PA, USA.

Roland E Knoblauch (RE)

Janssen R&D, Spring House, PA, USA.

Byoung Chul Cho (BC)

Yonsei Cancer Center, Yonsei University College of Medicine, Seoul, Republic of Korea. Electronic address: cbc1971@yuhs.ac.

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Classifications MeSH