Glycolytic System in Axons Supplement Decreased ATP Levels after Axotomy of the Peripheral Nerve.


Journal

eNeuro
ISSN: 2373-2822
Titre abrégé: eNeuro
Pays: United States
ID NLM: 101647362

Informations de publication

Date de publication:
03 2023
Historique:
received: 30 08 2022
revised: 04 02 2023
accepted: 25 02 2023
pubmed: 10 3 2023
medline: 23 3 2023
entrez: 9 3 2023
Statut: epublish

Résumé

Wallerian degeneration (WD) occurs in the early stages of numerous neurologic disorders, and clarifying WD pathology is crucial for the advancement of neurologic therapies. ATP is acknowledged as one of the key pathologic substances in WD. The ATP-related pathologic pathways that regulate WD have been defined. The elevation of ATP levels in axon contributes to delay WD and protects axons. However, ATP is necessary for the active processes to proceed WD, given that WD is stringently managed by auto-destruction programs. But little is known about the bioenergetics during WD. In this study, we made sciatic nerve transection models for

Identifiants

pubmed: 36894321
pii: ENEURO.0353-22.2023
doi: 10.1523/ENEURO.0353-22.2023
pmc: PMC10035771
pii:
doi:

Substances chimiques

Adenosine Triphosphate 8L70Q75FXE

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2023 Takenaka et al.

Déclaration de conflit d'intérêts

The authors declare no competing financial interests.

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Auteurs

Tomofumi Takenaka (T)

Department of neurosurgery, Graduate School of Medicine, Osaka University, Osaka, 565-0871, Japan.
Department of Research Promotion and Management, National Cerebral and Cardiovascular Center, Osaka, 564-8565, Japan.

Yuichiro Ohnishi (Y)

Department of Research Promotion and Management, National Cerebral and Cardiovascular Center, Osaka, 564-8565, Japan ohnishinsurg@gmail.com hkishima@nsurg.med.osaka-u.ac.jp.
Department of Neurosurgery, Osaka Gyoumeikan Hospital, Osaka, 554-0012, Japan.

Masamichi Yamamoto (M)

Department of Research Promotion and Management, National Cerebral and Cardiovascular Center, Osaka, 564-8565, Japan.

Daiki Setoyama (D)

Department of Clinical Chemistry and Laboratory Medicine, Graduate School of Medical Sciences, Kyushu University, Fukuoka, 812-8582, Japan.

Haruhiko Kishima (H)

Department of neurosurgery, Graduate School of Medicine, Osaka University, Osaka, 565-0871, Japan ohnishinsurg@gmail.com hkishima@nsurg.med.osaka-u.ac.jp.

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