Efficacy of Osimertinib in Patients with Lung Cancer Positive for Uncommon EGFR Exon 19 Deletion Mutations.


Journal

Clinical cancer research : an official journal of the American Association for Cancer Research
ISSN: 1557-3265
Titre abrégé: Clin Cancer Res
Pays: United States
ID NLM: 9502500

Informations de publication

Date de publication:
01 06 2023
Historique:
received: 15 11 2022
revised: 03 02 2023
accepted: 08 03 2023
medline: 2 6 2023
pubmed: 14 3 2023
entrez: 13 3 2023
Statut: ppublish

Résumé

The uncommon EGFR exon 19 deletion (ex19del), L747_A750>P, demonstrates reduced sensitivity to osimertinib compared with the common ex19del, E746_A750del in preclinical models. The clinical efficacy of osimertinib in patients with non-small cell lung cancer harboring L747_A750>P and other uncommon ex19dels is not known. The AACR GENIE database was interrogated to characterize the frequency of individual ex19dels relative to other variants, and a multicenter retrospective cohort was used to compare clinical outcomes for patients with tumors harboring E746_A750del, L747_A750>P, and other uncommon ex19dels who received osimertinib in the first line (1L) or in second or later lines of therapy and were T790M+ (≥2L). ex19dels comprised 45% of EGFR mutations, with 72 distinct variants ranging in frequency from 28.1% (E746_A750del) to 0.03%, with L747_A750>P representing 1.8% of the EGFR mutant cohort. In our multi-institutional cohort (N = 200), E746_A750del was associated with significantly prolonged progression-free survival (PFS) with 1L osimertinib versus L747_A750>P [median 21.3 months (95% confidence interval, 17.0-31.7) vs. 11.7 months (10.8-29.4); adjusted HR 0.52 (0.28-0.98); P = 0.043]. Osimertinib efficacy in patients with other uncommon ex19dels varied on the basis of the specific mutation present. The ex19del L747_A750>P is associated with inferior PFS compared with the common E746_A750del mutation in patients treated with 1L osimertinib. Understanding differences in osimertinib efficacy among EGFR ex19del subtypes could alter management of these patients in the future.

Identifiants

pubmed: 36913537
pii: 718746
doi: 10.1158/1078-0432.CCR-22-3497
pmc: PMC10493186
mid: NIHMS1883691
doi:

Substances chimiques

osimertinib 3C06JJ0Z2O
ErbB Receptors EC 2.7.10.1
Protein Kinase Inhibitors 0
Aniline Compounds 0
EGFR protein, human EC 2.7.10.1

Types de publication

Multicenter Study Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

2123-2130

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States
Organisme : NCI NIH HHS
ID : P50 CA196530
Pays : United States
Organisme : NCI NIH HHS
ID : T32 CA233414
Pays : United States

Informations de copyright

©2023 American Association for Cancer Research.

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Auteurs

Michael J Grant (MJ)

Department of Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, Connecticut.

Jacqueline V Aredo (JV)

Department of Medicine (Oncology), Stanford University, Stanford, California.
Department of Medicine, University of California San Francisco School of Medicine, San Francisco, California.

Jacqueline H Starrett (JH)

Department of Pathology, Yale School of Medicine, New Haven, Connecticut.

Paul Stockhammer (P)

Department of Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, Connecticut.

Iris K van Alderwerelt van Rosenburgh (IK)

Department of Pathology, Yale School of Medicine, New Haven, Connecticut.
Department of Pharmacology, Yale School of Medicine, New Haven, Connecticut.
Yale Cancer Biology Institute, West Haven, Connecticut.

Anna Wurtz (A)

Department of Pathology, Yale School of Medicine, New Haven, Connecticut.

Andrew J Piper-Valillo (AJ)

Department of Medicine (Hematology/Oncology), Massachusetts General Hospital, Boston, Massachusetts.

Zofia Piotrowska (Z)

Department of Medicine (Hematology/Oncology), Massachusetts General Hospital, Boston, Massachusetts.

Christina Falcon (C)

Department of Medicine (Thoracic Oncology), Memorial Sloan Kettering Cancer Center, New York, New York.

Helena A Yu (HA)

Department of Medicine (Thoracic Oncology), Memorial Sloan Kettering Cancer Center, New York, New York.

Charu Aggarwal (C)

Department of Medicine (Division of Hematology/Oncology), University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania.

Dylan Scholes (D)

Department of Medicine (Division of Hematology/Oncology), University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania.

Tejas Patil (T)

Department of Medicine (Division of Medical Oncology), University of Colorado School of Medicine, Aurora, Colorado.

Christina Nguyen (C)

Department of Medicine (Division of Medical Oncology), University of Colorado School of Medicine, Aurora, Colorado.

Manali Phadke (M)

Yale Center for Analytical Sciences, Yale School of Public Health, New Haven, Connecticut.

Fang-Yong Li (FY)

Yale Center for Analytical Sciences, Yale School of Public Health, New Haven, Connecticut.

Joel Neal (J)

Department of Medicine (Oncology), Stanford University, Stanford, California.

Mark A Lemmon (MA)

Department of Pharmacology, Yale School of Medicine, New Haven, Connecticut.
Yale Cancer Biology Institute, West Haven, Connecticut.

Zenta Walther (Z)

Department of Pathology, Yale School of Medicine, New Haven, Connecticut.

Katerina Politi (K)

Department of Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, Connecticut.
Department of Pathology, Yale School of Medicine, New Haven, Connecticut.

Sarah B Goldberg (SB)

Department of Medicine (Section of Medical Oncology), Yale School of Medicine, New Haven, Connecticut.

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