Atypical cardiac amyloidosis phenotypes identified at transplant: a case series.

Cardiac amyloidosis Case series Dilated cardiomyopathy Heart transplant

Journal

European heart journal. Case reports
ISSN: 2514-2119
Titre abrégé: Eur Heart J Case Rep
Pays: England
ID NLM: 101730741

Informations de publication

Date de publication:
Mar 2023
Historique:
received: 03 10 2022
revised: 22 11 2022
accepted: 23 02 2023
entrez: 16 3 2023
pubmed: 17 3 2023
medline: 17 3 2023
Statut: epublish

Résumé

Transthyretin amyloidosis (TTR) is increasingly implicated as an aetiology of advanced cardiomyopathy. Typically, both genetic variant (TTRv) and wild-type (TTRwt) amyloidosis present with a restrictive phenotype. We present a series of three patients who were found to have cardiac amyloidosis on explant following heart transplant (HT) who had atypical, non-restrictive phenotypes. All three patients were men, three were Black, and only one had an alternative pre-HT explanation for their advanced, dilated cardiomyopathy. Pre-HT transthoracic echocardiograms were notable for left ventricular (LV) dilation (>95th percentile for height and gender), low EF, and normal LV wall thickness. Explants showed varying amounts of amyloid deposition, ranging from diffuse biventricular patterns to perivascular involvement. Mass spectrometry confirmed the presence of TTRv (two cases) and TTRwt (one case). Patients with dilated cardiomyopathy may harbour cardiac amyloidosis. Uncertainty remains regarding the contribution of amyloidosis to the development of a dilated phenotype. The pathogenic Val142Ile variant seen in two of these patients, a variant common in Black patients, suggests a need for further investigation into the potential relationship between TTRv amyloidosis and dilated cardiomyopathy.

Sections du résumé

Background UNASSIGNED
Transthyretin amyloidosis (TTR) is increasingly implicated as an aetiology of advanced cardiomyopathy. Typically, both genetic variant (TTRv) and wild-type (TTRwt) amyloidosis present with a restrictive phenotype. We present a series of three patients who were found to have cardiac amyloidosis on explant following heart transplant (HT) who had atypical, non-restrictive phenotypes.
Case Summary UNASSIGNED
All three patients were men, three were Black, and only one had an alternative pre-HT explanation for their advanced, dilated cardiomyopathy. Pre-HT transthoracic echocardiograms were notable for left ventricular (LV) dilation (>95th percentile for height and gender), low EF, and normal LV wall thickness. Explants showed varying amounts of amyloid deposition, ranging from diffuse biventricular patterns to perivascular involvement. Mass spectrometry confirmed the presence of TTRv (two cases) and TTRwt (one case).
Discussion UNASSIGNED
Patients with dilated cardiomyopathy may harbour cardiac amyloidosis. Uncertainty remains regarding the contribution of amyloidosis to the development of a dilated phenotype. The pathogenic Val142Ile variant seen in two of these patients, a variant common in Black patients, suggests a need for further investigation into the potential relationship between TTRv amyloidosis and dilated cardiomyopathy.

Identifiants

pubmed: 36923113
doi: 10.1093/ehjcr/ytad105
pii: ytad105
pmc: PMC10010473
doi:

Types de publication

Case Reports

Langues

eng

Pagination

ytad105

Informations de copyright

© The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology.

Déclaration de conflit d'intérêts

Conflict of interest: None declared.

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Auteurs

Joshua A Rushakoff (JA)

Smidt Heart Institute, Cedars-Sinai Medical Center, 127 S. San Vicente Blvd, A3107, Los Angeles, CA 90048, USA.

Evan P Kransdorf (EP)

Smidt Heart Institute, Cedars-Sinai Medical Center, 127 S. San Vicente Blvd, A3107, Los Angeles, CA 90048, USA.

Michelle M Kittleson (MM)

Smidt Heart Institute, Cedars-Sinai Medical Center, 127 S. San Vicente Blvd, A3107, Los Angeles, CA 90048, USA.

Jonathan R Neyer (JR)

Division of Cardiology, Kaiser Permanente, 1526 N Edgemont St., Fl 2, Los Angeles, CA 90027, USA.

Daniel Luthringer (D)

Department of Pathology and Laboratory Medicine, Cedars-Sinai Medical Center, 8700 Beverly Blvd., Los Angeles, CA 90048, USA.

Jignesh K Patel (JK)

Smidt Heart Institute, Cedars-Sinai Medical Center, 127 S. San Vicente Blvd, A3107, Los Angeles, CA 90048, USA.

Classifications MeSH